| Phenotype | Fish | Conditions | Figures | 
    
    
        
            | notochord truncated, abnormal | AB + MO1-chrd | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | whole organism ventral region ved expression increased distribution, abnormal | AB + MO1-chrd | standard conditions | Fig 6  from Ye et al., 2019 
 | 
    
        
            | whole organism wholly ventralized, abnormal | AB + MO1-chrd | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | pancreatic B cell increased amount, abnormal | AB + MO1-chrd | standard conditions | Fig. 2
                    
                    from Song et al., 2007 
 | 
    
        
            | notochord decreased width, abnormal | AB + MO1-chrd | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | whole organism ventral region szl expression increased distribution, abnormal | AB + MO1-chrd | standard conditions | Fig 6  from Ye et al., 2019 
 | 
    
        
            | whole organism ventralized, abnormal | AB + MO1-chrd | standard conditions | Fig 6  from Ye et al., 2019 
 | 
    
        
            | head decreased size, abnormal | AB + MO1-chrd | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | intermediate cell mass of mesoderm increased size, abnormal | TL + MO1-chrd | standard conditions | Fig. 2
                    
                    from Kamachi et al., 2008 
 | 
    
        
            | post-vent region increased size, abnormal | TL + MO1-chrd | standard conditions | Fig. 2
                    
                    from Kamachi et al., 2008 
 | 
    
        
            | brain anterior region decreased size, abnormal | TL + MO1-chrd | standard conditions | Fig. 2
                    
                    from Kamachi et al., 2008 
 | 
    
        
            | post-vent region increased size, abnormal | TL + MO1-chrd + MO2-chrd | standard conditions | Fig. 2
                    
                    from Kamachi et al., 2008 
 | 
    
        
            | brain anterior region decreased size, abnormal | TL + MO1-chrd + MO2-chrd | standard conditions | Fig. 2
                    
                    from Kamachi et al., 2008 
 | 
    
        
            | intermediate cell mass of mesoderm increased size, abnormal | TL + MO1-chrd + MO2-chrd | standard conditions | Fig. 2
                    
                    from Kamachi et al., 2008 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd | standard conditions | Fig. 7  from Branam et al., 2010 Fig. 8
  from Dal-Pra et al., 2006 
 | 
    
        
            | primitive heart tube inverted, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Aamar et al., 2010 
 | 
    
        
            | Kupffer's vesicle inverted, abnormal | WT + MO1-chrd | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | determination of left/right symmetry disrupted, abnormal | WT + MO1-chrd | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | Kupffer's vesicle bilateral, abnormal | WT + MO1-chrd | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | post-vent region increased size, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Xie et al., 2011 
 | 
    
        
            | somite U-shaped, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Griepenburg et al., 2015 Fig. 5
  from Xie et al., 2011 
 | 
    
        
            | neural plate decreased length, abnormal | WT + MO1-chrd | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | median fin fold increased amount, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Xie et al., 2011 
 | 
    
        
            | Kupffer's vesicle decreased size, abnormal | WT + MO1-chrd | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | post-vent region apoptotic, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Robu et al., 2007 
 | 
    
        
            | primitive heart tube bilateral, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Aamar et al., 2010 
 | 
    
        
            | whole organism ventralized, abnormal | WT + MO1-chrd | control | Figure 4  from Cheng et al., 2019 
 | 
    
        
            | determination of heart left/right asymmetry disrupted, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Aamar et al., 2010 
 | 
    
        
            | forerunner cell group decreased size, abnormal | WT + MO1-chrd | standard conditions | Fig. 7  from Aamar et al., 2010 
 | 
    
        
            | heart looping disrupted, abnormal | WT + MO1-chrd | standard conditions | Fig. S4  from Aamar et al., 2010 
 | 
    
        
            | blood island size, abnormal | WT + MO1-chrd | standard conditions | Fig. 5  from Griepenburg et al., 2015 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd | standard conditions | Fig. 4  from Kapp et al., 2013 Fig. 8
                    
                    from Zhong et al., 2011
 Fig. 7
  from Branam et al., 2010 Fig. 3
  , 
                    
                        Fig. 4  from Zhang et al., 2010 Fig. 3
  , 
                    
                        Fig. 7  , 
                    
                        Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | post-vent region wholly ventralized, abnormal | WT + MO1-chrd | standard conditions | Fig. 2  from Esterberg et al., 2009 
 | 
    
        
            | neural plate decreased thickness, abnormal | WT + MO1-chrd | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | determination of left/right asymmetry in lateral mesoderm disrupted, abnormal | WT + MO1-chrd | standard conditions | Fig. 4  from Aamar et al., 2010 
 | 
    
        
            | otic vesicle decreased size, abnormal | WT + MO1-chrd | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd + MO4-tp53 | standard conditions | Fig. 8  from Branam et al., 2010 
 | 
    
        
            | post-vent region bent, abnormal | WT + MO1-chrd + MO4-tp53 | standard conditions | Fig. 8  from Branam et al., 2010 
 | 
    
        
            | post-vent region apoptotic, abnormal | WT + MO1-chrd + MO4-tp53 | standard conditions | Fig. 5  from Robu et al., 2007 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO4-tp53 | standard conditions | Fig. S6  from Moreno-Ayala et al., 2015 Fig. 8
  from Branam et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | t32231Tg + MO1-chrd | standard conditions | Fig. 3  from Chen et al., 2012 
 | 
    
        
            | pancreatic B cell decreased amount, abnormal | hnf1bahi2169Tg/hi2169Tg + MO1-chrd | standard conditions | Fig. 2
                    
                    from Song et al., 2007 
 | 
    
        
            | whole organism ventralized, exacerbated | marcksbihb199/ihb199 + MO1-chrd | standard conditions | Fig 6  from Ye et al., 2019 
 | 
    
        
            | whole organism ventral region ved expression increased distribution, abnormal | marcksbihb199/ihb199 + MO1-chrd | standard conditions | Fig 6  from Ye et al., 2019 
 | 
    
        
            | whole organism ventral region szl expression increased distribution, abnormal | marcksbihb199/ihb199 + MO1-chrd | standard conditions | Fig 6  from Ye et al., 2019 
 | 
    
        
            | notochord decreased width, abnormal | AB + MO1-chrd + MO1-gsc | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | head decreased size, abnormal | AB + MO1-chrd + MO1-gsc | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | notochord truncated, abnormal | AB + MO1-chrd + MO1-gsc | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | whole organism wholly ventralized, abnormal | AB + MO1-chrd + MO1-gsc | standard conditions | Fig. 7  from Dixon Fox et al., 2009 
 | 
    
        
            | dorsal convergence disrupted, abnormal | WT + MO1-cdh2 + MO1-chrd | standard conditions | Fig. 6  from von der Hardt et al., 2007 
 | 
    
        
            | lateral mesoderm mislocalised, abnormal | WT + MO1-cdh2 + MO1-chrd | standard conditions | Fig. 6  from von der Hardt et al., 2007 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd + MO1-fstl1b | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO1-fstl1b | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd + MO1-fstl1b | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | head anterior region agenesis, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 3  from Dal-Pra et al., 2006 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | neural plate decreased length, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | head anterior region truncated, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 3  from Dal-Pra et al., 2006 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 3  from Zhang et al., 2010 Fig. 3
  , 
                    
                        Fig. 7  , 
                    
                        Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | neural plate decreased thickness, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | otic vesicle mislocalised medially, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | otic vesicle decreased size, abnormal | WT + MO1-chrd + MO1-nog1 | standard conditions | Fig. 4  from Dal-Pra et al., 2006 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO1-zmiz2 + MO4-tp53 | standard conditions | Fig. S6  from Moreno-Ayala et al., 2015 
 | 
    
        
            | post-vent region increased thickness, abnormal | WT + MO1-chrd + MO2-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | blood circulation disrupted, abnormal | WT + MO1-chrd + MO2-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO2-chrdl2 | standard conditions | Fig. 7  , 
                    
                        Fig. 8  from Branam et al., 2010 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd + MO2-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | head morphology, abnormal | WT + MO1-chrd + MO2-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd + MO2-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd + MO2-chrdl2 + MO4-tp53 | standard conditions | Fig. 8  from Branam et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO2-chrdl2 + MO4-tp53 | standard conditions | Fig. 8  from Branam et al., 2010 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd + MO2-igfbp3 | standard conditions | Fig. 8
                    
                    from Zhong et al., 2011 
 | 
    
        
            | hematopoietic system increased size, abnormal | WT + MO1-chrd + MO2-igfbp3 | standard conditions | Fig. 8
                    
                    from Zhong et al., 2011 
 | 
    
        
            | post-vent region increased size, abnormal | WT + MO1-chrd + MO2-igfbp3 | standard conditions | Fig. 8
                    
                    from Zhong et al., 2011 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO2-igfbp3 | standard conditions | Fig. 8
                    
                    from Zhong et al., 2011 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO2-nog1 | standard conditions | text only
                    
                    from Dal-Pra et al., 2006 
 | 
    
        
            | Kupffer's vesicle bilateral, abnormal | WT + MO1-chrd + MO2-sox17 | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | Kupffer's vesicle decreased size, abnormal | WT + MO1-chrd + MO2-sox17 | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | determination of left/right symmetry disrupted, abnormal | WT + MO1-chrd + MO2-sox17 | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | Kupffer's vesicle inverted, abnormal | WT + MO1-chrd + MO2-sox17 | standard conditions | Fig. 6  from Aamar et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO3-bmper | standard conditions | Fig. 3  from Zhang et al., 2010 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd + MO3-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | post-vent region increased thickness, abnormal | WT + MO1-chrd + MO3-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | head morphology, abnormal | WT + MO1-chrd + MO3-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | blood circulation disrupted, abnormal | WT + MO1-chrd + MO3-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO3-chrdl2 | standard conditions | Fig. 7  from Branam et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO3-wnt8a + MO5-wnt8a | standard conditions | Fig. 4  from Kapp et al., 2013 
 | 
    
        
            | otic vesicle formation process quality, abnormal | WT + MO1-chrd + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic placode formation process quality, abnormal | WT + MO1-chrd + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic vesicle decreased size, abnormal | WT + MO1-chrd + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic vesicle formation process quality, abnormal | fgf8ati282a/ti282a + MO1-chrd + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic placode formation process quality, abnormal | fgf8ati282a/ti282a + MO1-chrd + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | whole organism cytolysis increased occurrence, abnormal | TL + MO1-chrd + MO1-fstl1b + MO1-nog1 | standard conditions | Fig. 4  from Langdon et al., 2016 
 | 
    
        
            | whole organism wholly ventralized, abnormal | TL + MO1-chrd + MO1-fstl1b + MO1-nog1 | standard conditions | Fig. 4  from Langdon et al., 2016 
 | 
    
        
            | whole organism wholly dorsalized, abnormal | WT + MO1-bmp1a + MO1-chrd + MO1-tll1 | standard conditions | Fig. 4  from Zhang et al., 2010 
 | 
    
        
            | whole organism wholly ventralized, abnormal | WT + MO1-chrd + MO1-fstl1b + MO1-nog1 | standard conditions | Fig. 2  from Kapp et al., 2013 Fig. 8
  from Dal-Pra et al., 2006 
 | 
    
        
            | dorsal/ventral pattern formation disrupted, abnormal | WT + MO1-chrd + MO1-fstl1b + MO1-nog1 | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | head decreased size, abnormal | WT + MO1-chrd + MO1-fstl1b + MO1-nog1 | standard conditions | Fig. 8  from Dal-Pra et al., 2006 
 | 
    
        
            | trigeminal placode decreased size, abnormal | WT + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 3  from Kwon et al., 2009 
 | 
    
        
            | otic placode formation process quality, abnormal | WT + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic vesicle decreased size, abnormal | WT + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic vesicle malformed, abnormal | WT + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic vesicle formation process quality, abnormal | WT + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | inner ear lacks all parts of type otolith, abnormal | WT + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | post-vent region wholly ventralized, abnormal | WT + MO1-chrd + MO5-dlx3b + MO5-dlx4b | standard conditions | Fig. 2  from Esterberg et al., 2009 
 | 
    
        
            | intermediate cell mass of mesoderm increased size, abnormal | WT + MO1-chrd + MO5-dlx3b + MO5-dlx4b | standard conditions | Fig. 2  from Esterberg et al., 2009 
 | 
    
        
            | whole organism cytolysis increased occurrence, abnormal | ctsbap24bdth/+ + MO1-chrd + MO1-fstl1b + MO1-nog1 (TL) | standard conditions | Fig. 4  from Langdon et al., 2016 
 | 
    
        
            | whole organism wholly dorsalized, ameliorated | ctsbap24bdth/+ + MO1-chrd + MO1-fstl1b + MO1-nog1 (TL) | standard conditions | Fig. 4  from Langdon et al., 2016 
 | 
    
        
            | whole organism wholly ventralized, abnormal | ctsbap24bdth/+ + MO1-chrd + MO1-fstl1b + MO1-nog1 (TL) | standard conditions | Fig. 4  from Langdon et al., 2016 
 | 
    
        
            | otic vesicle formation process quality, abnormal | fgf8ati282a/ti282a + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic placode formation process quality, abnormal | fgf8ati282a/ti282a + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | otic vesicle aplastic, abnormal | fgf8ati282a/ti282a + MO1-chrd + MO2-bmper + MO6-fsta | standard conditions | Fig. 2  from Kwon et al., 2009 
 | 
    
        
            | whole organism wholly ventralized, abnormal | Df(Chr25:szl)m60/m60 + MO1-chrd (HK) | standard conditions | Fig. 1  from Dal-Pra et al., 2006 
 | 
    
        
            | whole organism wholly ventralized, abnormal | ints6p18ahub + MO1-chrd (AB/TU) | standard conditions | Fig. 4  from Kapp et al., 2013 
 | 
    
        
            | whole organism wholly dorsalized, abnormal | ints6p18ahub + MO1-chrd + MO3-wnt8a + MO5-wnt8a (AB/TU) | standard conditions | Fig. 4  from Kapp et al., 2013 
 | 
    
        
            | whole organism wholly ventralized, abnormal | ints6p18ahub + MO1-chrd + MO1-fstl1b + MO1-nog1 (AB/TU) | standard conditions | Fig. 2  from Kapp et al., 2013 
 | 
    
        
            | whole organism wholly ventralized, abnormal | brsb2; ctnnb2p1/p1 + MO1-chrd | standard conditions | Fig. 3  from Varga et al., 2007 
 | 
    
        
            | post-vent region morphology, abnormal | brsb2; ctnnb2p1/p1 + MO1-chrd | standard conditions | Fig. 3  from Varga et al., 2007 
 | 
    
        
            | dorsal/ventral axis specification decreased process quality, ameliorated | pou5f3m793/+; sox19bm1434/+ + MO1-chrd | standard conditions | Fig. 2  from Gao et al., 2022 
 | 
    
        
            | whole organism wholly ventralized, abnormal | brsb2; ctnnb2p1/p1 + MO1-chrd + MO1-ctnnb2 + MO2-ctnnb1 | standard conditions | Fig. 3  from Varga et al., 2007 
 | 
    
        
            | post-vent region morphology, abnormal | brsb2; ctnnb2p1/p1 + MO1-chrd + MO1-ctnnb2 + MO2-ctnnb1 | standard conditions | Fig. 3  from Varga et al., 2007 
 | 
    
        
            | dorsal/ventral axis specification decreased process quality, ameliorated | pou5f3m793/+; sox19bm1434/+ + MO1-chrd + MO1-nog1 | standard conditions | Fig. 2  from Gao et al., 2022 
 |