Publication	Data		Fish	Anatomy
Prendergast et al., 2022	Figure 4		AB + CRISPR5-mib1 + CRISPR6-mib1	brain hemorrhagic, abnormal dorsal aorta hemorrhagic, abnormal vasculature broken, abnormal whole organism increased curvature, abnormal
Saraswathy et al., 2022	Figure 1		mib1tfi91/tfi91 ,   mib1ta52b/ta52b ,   mib1nce2a/nce2a ,   mib1tfi91/+; mib1nce2a/+ ,   AB/TU + MO1-mib1	convergent extension involved in gastrulation decreased process quality, abnormal convergent extension involved in gastrulation normal process quality, abnormal neural plate dlx3b expression spatial pattern, abnormal neural plate increased width, abnormal notochord increased width, abnormal somite pcdh8 expression spatial pattern, abnormal somite increased width, abnormal whole organism mib1 expression decreased amount, abnormal
	Figure 2		AB/TU + MO1-mib1	convergent extension involved in gastrulation decreased process quality, abnormal
	Figure 3		mib1tfi91/tfi91	gastrula cell early endosome decreased amount, abnormal gastrula cell early endosome disorganized, abnormal
	Figure 5		AB/TU + MO1-mib1	convergent extension involved in gastrulation decreased process quality, abnormal
Horstick et al., 2020	Fig. 6		mib1ta52b/+ (TL)	motor behavior process quality, abnormal
Xie et al., 2020	Fig 6		mib1ta52b/ta52b	pronephric duct e2f5 expression increased distribution, abnormal
Yang et al., 2020	Fig. 3		mib1ta52b/ta52b (AB)	floor plate chl1a expression spatial pattern, abnormal hypochord chl1a expression absent, abnormal
Gou et al., 2018	Fig. 9		mib1ta52b/ta52b	otic vesicle atoh1a expression increased amount, abnormal otic vesicle atoh1a expression increased distribution, abnormal otic vesicle atoh1a expression spatial pattern, abnormal otic vesicle neurog1 expression increased amount, abnormal otic vesicle neurog1 expression increased distribution, abnormal otic vesicle neurog1 expression spatial pattern, abnormal
Kawahara et al., 2018	Fig. S3		mib1ta52b/ta52b	hypochord ly97.3 expression absent, abnormal
Kuwabara et al., 2018	Fig. 1		mib1ta52b/ta52b	brain hk1 expression increased amount, abnormal brain ldha expression increased amount, abnormal brain pkma expression increased amount, abnormal brain slc2a3a expression increased amount, abnormal spinal cord hk1 expression increased amount, abnormal spinal cord ldha expression increased amount, abnormal spinal cord pkma expression increased amount, abnormal spinal cord slc2a3a expression increased amount, abnormal
	Fig. 2		mib1ta52b/ta52b	whole organism elavl3 expression increased amount, abnormal whole organism hk1 expression increased amount, abnormal whole organism hk2 expression increased amount, abnormal whole organism ldha expression increased amount, abnormal whole organism pkma expression increased amount, abnormal whole organism slc2a3a expression increased amount, abnormal
	Fig. 3		mib1ta52b/ta52b	brain hk1 expression decreased amount, abnormal brain hk2 expression increased amount, abnormal brain ldha expression decreased amount, abnormal brain pkma expression decreased amount, abnormal brain slc2a3a expression decreased amount, abnormal retina hk2 expression increased amount, abnormal retina slc2a1a expression decreased amount, abnormal
	Fig. 4		mib1ta52b/ta52b	whole organism elavl3 expression decreased amount, abnormal whole organism hk2 expression increased amount, abnormal whole organism ldha expression decreased amount, abnormal whole organism pkma expression decreased amount, abnormal whole organism slc2a1a expression decreased amount, abnormal whole organism slc2a3a expression decreased amount, abnormal
Anuppalle et al., 2017	Fig. 5		mib1ta52b/ta52b	hindbrain prune2 expression increased amount, abnormal spinal cord prune2 expression increased amount, abnormal telencephalon prune2 expression increased amount, abnormal
Mizoguchi et al., 2017	Fig. 6		mib1ta52b/ta52b; zf106Tg ,   mib1ta52b/ta52b; chi4Tg; nkhspGFF15ATg	posterior lateral line neuromast cell projection direction, abnormal posterior lateral line neuromast primordium migration delayed, abnormal posterior lateral line neuromast primordium migration process quality, abnormal
	Fig. 7		mib1ta52b/ta52b; zf106Tg	posterior lateral line neuromast primordium migration delayed, abnormal posterior lateral line neuromast primordium migration process quality, abnormal
Gore et al., 2016	Fig. 1		mib1ta52b/ta52b	ventral wall of dorsal aorta dnmt3bb.1 expression decreased amount, abnormal whole organism dnmt3bb.1 expression decreased amount, abnormal
Mahony et al., 2016	Fig. 1		mib1ta52b/ta52b	neural crest tfec expression absent, abnormal ventral wall of dorsal aorta runx1 expression absent, abnormal
Nakahara et al., 2016	Fig. S4		mib1ta52b/ta52b	adenohypophyseal placode her4.1 expression absent, abnormal adenohypophyseal placode hey1 expression absent, abnormal brain her4.1 expression absent, abnormal brain hey1 expression absent, abnormal
Shin et al., 2016	Fig. 7		mib1ta52b/ta52b	dorsal aorta flt4 expression increased amount, abnormal
Thambyrajah et al., 2016	Fig. 4		mib1ta52b/ta52b	hematopoietic multipotent progenitor cell gfi1aa expression absent, abnormal inner ear gfi1aa expression increased amount, abnormal
Wang et al., 2016	Fig. S5		WT + MO1-mib1	whole organism flt1 expression decreased amount, abnormal
Cheng et al., 2015	Fig. 2		mib1ta52b/ta52b	nervous system dtx1 expression increased amount, abnormal whole organism dtx1 expression increased amount, abnormal
Ghaye et al., 2015	Fig. 5		mib1ta52b/ta52b	nervous system ascl1b expression increased amount, abnormal nervous system nkx6.1 expression decreased amount, abnormal pancreas primordium ascl1b expression increased amount, abnormal pancreas primordium nkx6.1 expression absent, abnormal pancreas primordium nkx6.1 expression decreased amount, abnormal
Hsu et al., 2015	Fig. 2		mib1nn2000/nn2000 ,   mib1nn2001/nn2001	post-vent region curved, abnormal post-vent region decreased pigmentation, abnormal post-vent region hemorrhagic, abnormal
Kressmann et al., 2015	Fig. 4		AB + MO1-mib1	spinal cord generation of neurons process quality, abnormal
Mikami et al., 2015	Fig. 3		mib1tfi91/tfi91 ,   mib1ta52b/ta52b	neuroblast elavl3 expression increased amount, abnormal neuron her4.1 expression decreased amount, abnormal
	Fig. 5		mib1tfi91/tfi91 ,   mib1ta52b/ta52b	post-vent region decreased pigmentation, abnormal post-vent region white, abnormal
Rider et al., 2015	Fig. 5		mib1ta52b/ta52b; ue301Tg ,   mib1ta52b/ta52b	anterior mesenteric artery ren expression absent, abnormal anterior mesenteric artery TagRFP expression absent, abnormal blood circulation arrested, abnormal brain hemorrhagic, abnormal caudal fin malformed, abnormal caudal fin unpigmented, abnormal embryonic structure ren expression absent, abnormal pericardium edematous, abnormal
Rodríguez-Fraticelli et al., 2015	Fig. 6		mib1ta52b/ta52b	enterocyte differentiation decreased occurrence, abnormal intestinal epithelium endocytosis decreased occurrence, abnormal intestinal epithelium has fewer parts of type enterocyte, abnormal
Stooke-Vaughan et al., 2015	Fig. 5		mib1ta52b/ta52b	otic vesicle has extra parts of type hair cell, abnormal otic vesicle has fewer parts of type auditory epithelial support cell, abnormal
Zhou et al., 2015	Fig. 2		mib1ta52b/ta52b	pronephric tubule mcidas expression increased distribution, abnormal pronephric tubule has extra parts of type pronephric tubule multi-ciliated epithelial cell, abnormal pronephros multi-ciliated epithelial cell differentiation increased occurrence, abnormal
Imai et al., 2014	Fig. 7		mib1ta52b/ta52b	neural retina development disrupted, abnormal
	Fig. 8		mib1ta52b/ta52b	retinal ganglion cell axon increased accumulation retina ventral region, abnormal
Okigawa et al., 2014	Fig. 1		mib1ta52b/ta52b	CiD increased amount, abnormal VeLD decreased amount, abnormal
	Fig. 3		nns5Tg + MO1-mib1	CiD increased amount, abnormal
Sawamiphak et al., 2014	Fig. S4		la2Tg; s896Tg + MO4-mib1	ventral wall of dorsal aorta has fewer parts of type hematopoietic stem cell, abnormal
Kondrychyn et al., 2013	Fig. 4		mib1ta52b/ta52b; sqet33Et/+	central canal morphology, abnormal roof plate spinal cord region cell projection absent, abnormal roof plate spinal cord region malformed, abnormal roof plate spinal cord region separated from roof plate spinal cord region, abnormal spinal cord development disrupted, abnormal
Luxan et al., 2013	Fig. 4		mib1ta52b/ta52b; twu34Tg	heart looping disrupted, abnormal pericardium distended, abnormal post-vent region kinked, abnormal
Pascoal et al., 2013	Fig. 2		mib1ta52b/ta52b	pectoral fin endoskeletal disc disorganized, abnormal pectoral fin fold morphology, abnormal
	Fig. 4		mib1m178/m178 ,   mib1ta52b/ta52b	pectoral fin actin filament mislocalised, abnormal pectoral fin musculature contractile muscle fiber malformed, abnormal
	Fig. 5		mib1ta52b/ta52b	pectoral fin myofibril disheveled, abnormal
	Fig. 7		mib1ta52b/ta52b	pectoral fin myoblast decreased amount, abnormal
Jensen et al., 2012	Fig. 4		mib1ta52b/ta52b; y1Tg	intersegmental vessel disorganized, abnormal intersegmental vessel has extra parts of type blood vessel, abnormal intersegmental vessel has extra parts of type vascular sprouts, abnormal intersegmental vessel increased mass density, abnormal
Obholzer et al., 2012	Fig. S7		mib1ta52b/ta52b	inner ear morphology, abnormal
Porazzi et al., 2012	Fig. 1		mib1ta52b/ta52b	thyroid gland development decreased process quality, abnormal thyroid primordium disorganized, abnormal thyroid primordium mislocalised, abnormal thyroid primordium physical object quality, abnormal
	Fig. 2		mib1ta52b/ta52b	thyroid gland development decreased process quality, abnormal thyroid primordium increased volume, abnormal
	Fig. 3		mib1ta52b/ta52b	pharyngeal vasculature separated from thyroid primordium, abnormal thyroid primordium mislocalised ventrally, abnormal
	Fig. S1		mib1ta52b/ta52b	thyroid gland development decreased process quality, abnormal thyroid primordium disorganized, abnormal thyroid primordium increased size, abnormal
Hortopan et al., 2011	Fig. 4		mib1hi904Tg/hi904Tg (AB)	retina layer formation arrested, abnormal retinal neural layer undifferentiated, abnormal
Kapsimali et al., 2011	Fig. 6		mib1ta52b/ta52b	pharyngeal arch 3 epithelium physical object quality, abnormal taste bud formation decreased process quality, abnormal taste bud physical quality, abnormal
Mizoguchi et al., 2011	Fig. 1		mib1ta52b/ta52b	posterior lateral line ganglion neuron increased amount, abnormal
	Fig. 5		mib1ta52b/ta52b	posterior lateral line ganglion neuron increased amount, abnormal posterior lateral line primordium cell decreased amount, abnormal
Untergasser et al., 2011	Fig. 4		mib1m178/m178	central nervous system dkk3a expression decreased amount, abnormal peripheral nervous system dkk3a expression decreased amount, abnormal
Zygmunt et al., 2011	Fig. 6		mib1m178/m178; y7Tg	endothelial cell increased amount, abnormal
Barresi et al., 2010	Fig. 3		mib1hi904Tg/hi904Tg (AB/TU)	anterior commissure astrocyte mislocalised, abnormal anterior commissure commissural neuron axon guidance disrupted, abnormal anterior commissure decreased size, abnormal anterior commissure has fewer parts of type astrocyte, abnormal cranial nerve II increased thickness, abnormal postoptic commissure astrocyte mislocalised, abnormal postoptic commissure commissural neuron axon guidance disrupted, abnormal postoptic commissure decreased size, abnormal postoptic commissure has fewer parts of type astrocyte, abnormal
	Fig. 4		mib1hi904Tg/hi904Tg (AB/TU)	hindbrain axon morphology, abnormal hindbrain radial glial cell mislocalised, abnormal hindbrain radial glial cell morphology, abnormal
	Fig. 5		mib1hi904Tg/hi904Tg (AB/TU)	spinal cord lacks all parts of type ventricular zone radial glial cell, abnormal
Herrgen et al., 2010	Fig. 2		mib1ta52b/ta52b	somitogenesis delayed, abnormal
	Fig. 3		mib1ta52b/ta52b	somite 2 increased length, abnormal somite 3 increased length, abnormal somite 4 increased length, abnormal somite 5 increased length, abnormal somitogenesis delayed, abnormal
	Fig. 4		mib1ta52b/ta52b	somite increased length, abnormal somitogenesis delayed, abnormal
	Fig. 5		mib1ta52b/ta52b	somitogenesis delayed, abnormal
Hortopan et al., 2010	Fig. 1		mib1hi904Tg/hi904Tg	forebrain action potential increased occurrence, abnormal optic tectum action potential increased occurrence, abnormal
	Fig. 2		mib1hi904Tg/hi904Tg	swimming behavior increased occurrence, abnormal swimming behavior irregular duration, abnormal
	Fig. 3		mib1hi904Tg/hi904Tg	hindbrain bdnf expression increased amount, abnormal midbrain bdnf expression increased amount, abnormal optic tectum bdnf expression increased amount, abnormal telencephalon bdnf expression increased amount, abnormal whole organism bdnf expression increased amount, abnormal
	Fig. 4		mib1hi904Tg/hi904Tg	hindbrain pyya expression increased amount, abnormal locus coeruleus pyya expression decreased amount, abnormal olfactory bulb pyya expression decreased amount, abnormal spinal cord pyya expression increased amount, abnormal whole organism pyya expression increased amount, abnormal
	Fig. 5		mib1hi904Tg/hi904Tg	brain gamma-aminobutyric acid signaling pathway decreased occurrence, abnormal forebrain ventral region gad1b expression decreased amount, abnormal whole organism gad1b expression decreased amount, abnormal
	Fig. 6		mib1hi904Tg/hi904Tg	brain gamma-aminobutyric acid signaling pathway decreased occurrence, abnormal hindbrain gabra1 expression decreased amount, abnormal optic tectum gabra1 expression decreased amount, abnormal telencephalon gabra1 expression decreased amount, abnormal whole organism gabra1 expression decreased amount, abnormal
	Fig. 7		mib1hi904Tg/hi904Tg	cerebellum calb2b expression decreased amount, abnormal hindbrain calb2b expression decreased amount, abnormal optic tectum calb2b expression decreased amount, abnormal telencephalon calb2b expression decreased amount, abnormal whole organism calb2b expression decreased amount, abnormal
	Fig. 8		mib1hi904Tg/hi904Tg	hypothalamus interneuron pvalb5 expression decreased amount, abnormal telencephalon interneuron pvalb5 expression decreased amount, abnormal whole organism pvalb5 expression decreased amount, abnormal
	Fig. S4		mib1hi904Tg/hi904Tg	brain glra4b expression decreased amount, abnormal whole organism glra4b expression decreased amount, abnormal
Mahler et al., 2010	Fig. 7		mib1ta52b/ta52b	caudal tuberculum morphology, abnormal
Song et al., 2010	Fig. 4		mib1hi904Tg	Mauthner neuron increased amount, abnormal secondary motor neuron amount, abnormal secondary motor neuron axon decreased amount, abnormal spinal cord glial cell decreased amount, abnormal
	Fig. 9		mib1hi904Tg	cell death increased occurrence, abnormal retinal ganglion cell dead, abnormal retinal neural layer morphology, abnormal
Yamaguchi et al., 2010	Fig. 5		mib1ta52b/ta52b	eye proliferative region increased size, abnormal eye proliferative region mislocalised, abnormal retina disorganized, abnormal
Burns et al., 2009	text only		mib1hi904Tg/hi904Tg	hematopoietic stem cell differentiation disrupted, abnormal ventral wall of dorsal aorta hematopoietic stem cell absent, abnormal
Qiu et al., 2009	Fig. 1		mib1tfi91/tfi91 ,   mib1ta52b/ta52b	hindbrain development disrupted, abnormal hindbrain epithelium disorganized, abnormal hindbrain neuroepithelial cell aggregated, abnormal neuroepithelial cell circular, abnormal rhombomere boundary formation disrupted, abnormal
	Fig. 2		mib1tfi91/tfi91 ,   mib1ta52b/ta52b	hindbrain development disrupted, abnormal rhombomere boundary formation disrupted, abnormal
	Fig. 3		mib1ta52b/ta52b	hindbrain development disrupted, abnormal rhombomere boundary formation disrupted, abnormal
So et al., 2009	Fig. 3		mib1ta52b/ta52b	apoptotic process increased occurrence, abnormal trigeminal ganglion sensory neuron apoptotic, abnormal
Batista et al., 2008	Fig. 8		mib1ta52b/ta52b	CiD increased amount, abnormal VeLD decreased amount, abnormal ventral spinal cord interneuron differentiation disrupted, abnormal
Cau et al., 2008	Fig. 3		mib1ta52b/ta52b	epiphysis has extra parts of type neuron, abnormal
García-Lecea et al., 2008	Fig. 5		mib1tfi91/tfi91; sqet33e20Et	rhombic lip cellular motility, abnormal tela chorioidea mislocalised, abnormal tela chorioidea morphology, abnormal
Pendeville et al., 2008	Fig. 6		mib1ta52b/ta52b	vasculogenesis disrupted, abnormal
Aizawa et al., 2007	Fig. 4		mib1ta52b/ta52b	determination of left/right symmetry disrupted, abnormal generation of neurons increased rate, abnormal habenula cellular quality, abnormal habenula development disrupted, abnormal habenula neuron morphology, abnormal
Akanuma et al., 2007	Fig. 3		mib1ta52b/ta52b	trunk somite border disorganized, abnormal
Hsiao et al., 2007	Fig. 2		mib1ta52b/ta52b	ectoderm development disrupted, abnormal
	Fig. 3		mib1ta52b/ta52b	epidermal cell fate specification disrupted, abnormal
	Fig. 5		mib1ta52b/ta52b	ectoderm development disrupted, abnormal
Jänicke et al., 2007	Fig. 4		mib1ta52b/ta52b	ionocyte progenitor cell foxi3a expression spatial pattern, abnormal ionocyte progenitor cell aggregated, abnormal ionocyte progenitor cell irregular spatial pattern, abnormal NaK ionocyte atp1b1b expression increased amount, abnormal NaK ionocyte increased amount, abnormal vH ionocyte atp6v1aa expression increased amount, abnormal vH ionocyte increased amount, abnormal
	Fig. 5		mib1ta52b/ta52b	NaK ionocyte increased amount, abnormal vH ionocyte increased amount, abnormal
Leslie et al., 2007	Fig. 1		mib1unspecified	artery endothelial cell morphology, abnormal blood vessel endothelial cell migration disrupted, abnormal endothelial cell development disrupted, abnormal neural tube development disrupted, abnormal neural tube morphology, abnormal
Liu et al., 2007	Fig. 7		mib1m132/m132 (AB)	cell differentiation involved in pronephros development disrupted, abnormal pronephros cellular quality, abnormal
Schafer et al., 2007	Fig. 3		mib1ta52b/ta52b	floor plate formation disrupted, abnormal lateral floor plate composition, abnormal ventral spinal cord interneuron specification disrupted, abnormal
Shin et al., 2007	Fig. 2		mib1ta52b/ta52b; vu12Tg	spinal cord CNS interneuron decreased amount, abnormal spinal cord motor neuron differentiation disrupted, abnormal spinal cord primary motor neuron increased amount, abnormal ventral spinal cord interneuron differentiation disrupted, abnormal
Yeo et al., 2007	Fig. 7		mib1ta52b/ta52b	spinal cord GABAergic neuron decreased amount, abnormal
Yeo et al., 2007	Fig. 5		mib1ta52b/ta52b	central nervous system cellular quality, abnormal
Zhang et al., 2007	Fig. 1		mib1m132/m132 ,   mib1tfi91/tfi91 ,   mib1ta52b/ta52b	post-vent region increased curvature, abnormal
	Fig. 2		mib1m132/m132 + MO4-mib1 ,   mib1m132/m132 ,   mib1ta52b/ta52b + MO4-mib1 ,   mib1ta52b/ta52b + MO3-mib1 ,   mib1ta52b/ta52b	post-vent region increased curvature, abnormal post-vent region melanocyte decreased amount, abnormal
	Fig. 4		mib1tfi91/tfi91	post-vent region increased curvature, abnormal post-vent region melanocyte decreased amount, abnormal
	Table 1		mib1m132/+; mib1tfi91/+ ,   mib1m132/m132 ,   mib1ta52b/+; mib1tfi91/+ ,   mib1tfi91/tfi91 ,   mib1ta52b/+; mib1tfi91/+ ,   mib1m132/+; mib1ta52b/+ ,   mib1ta52b/ta52b ,   mib1m132/+; mib1tfi91/+ ,   mib1m132/+; mib1ta52b/+	post-vent region melanocyte decreased amount, abnormal somite decreased amount, abnormal
	Table 2		mib1m132/m132 + MO4-mib1 ,   mib1ta52b/ta52b + MO4-mib1 ,   mib1ta52b/ta52b + MO3-mib1	post-vent region melanocyte decreased amount, abnormal
Phenotype Annotation (1994-2006)	Fig. for (hi904)		mib1hi904Tg	brain quality, abnormal nervous system quality, abnormal post-vent region quality, abnormal whole organism shape, abnormal
	Fig. for (m132)		mib1m132/m132 (AB)	brain quality, abnormal hindbrain quality, abnormal inner ear quality, abnormal melanocyte irregular spatial pattern, abnormal musculoskeletal movement quality, abnormal nervous system quality, abnormal notochord undulate, abnormal otolith quality, abnormal pigment cell quality, abnormal response to external stimulus quality, abnormal sensory system quality, abnormal somite quality, abnormal
	Fig. for (m178)		mib1m178/m178 (AB)	brain quality, abnormal hindbrain quality, abnormal inner ear quality, abnormal melanocyte irregular spatial pattern, abnormal musculoskeletal movement quality, abnormal nervous system quality, abnormal notochord undulate, abnormal otolith quality, abnormal pigment cell quality, abnormal response to external stimulus quality, abnormal sensory system quality, abnormal somite quality, abnormal
	Fig. for (ta52b)		mib1ta52b/ta52b	brain quality, abnormal eye decreased size, abnormal gut quality, abnormal hindbrain quality, abnormal melanocyte irregular spatial pattern, abnormal nervous system quality, abnormal pigment cell quality, abnormal post-vent region quality, abnormal sensory system quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
Kawamura et al., 2005	Fig. 2		mib1ta52b/ta52b	somitogenesis disrupted, abnormal
Amsterdam et al., 2004	text only		mib1hi904Tg/hi904Tg	brain cell circular, abnormal brain cell dispersed, abnormal brain disorganized, abnormal brain surface feature shape, abnormal eye decreased size, abnormal forebrain increased size, abnormal head decreased size, abnormal head surface feature shape, abnormal mandibular arch skeleton increased thickness, abnormal pericardium edematous, abnormal trunk curved dorsal, abnormal yolk circular, abnormal
Riley et al., 2004	Fig. 6		mib1ta52b/ta52b	axon guidance disrupted, abnormal embryonic pattern specification disrupted, abnormal hindbrain commissure neuron absent, abnormal hindbrain disorganized, abnormal hindbrain radial glial cell disorganized, abnormal hindbrain radial glial cell increased amount, abnormal Mauthner neuron axon fasciculation, abnormal Mauthner neuron increased amount, abnormal primary neuron axon fasciculation, abnormal rhombomere morphology, abnormal rhombomere primary neuron increased amount, abnormal
Bae et al., 2003	Fig. 7		mib1m178/m178	Rohon-Beard neuron increased amount, abnormal
Choi et al., 2003	Fig. 4		mib1ta52b/ta52b	diencephalon celf2 expression increased amount, abnormal diencephalon celf2 expression increased distribution, abnormal hindbrain celf2 expression increased amount, abnormal hindbrain celf2 expression increased distribution, abnormal telencephalon celf2 expression increased amount, abnormal telencephalon celf2 expression increased distribution, abnormal
Golling et al., 2002	Fig. 1		mib1hi904Tg/hi904Tg	brain hemorrhagic, abnormal post-vent region bent, abnormal
	Fig. 2		mib1hi904Tg/hi904Tg	brain morphology, abnormal brain neuron disorganized, abnormal central nervous system neuron disorganized, abnormal
Heller et al., 2002	Fig. 4		mib1ta52b/ta52b	auditory receptor cell fate specification process quality, abnormal hair cell anterior macula increased amount, abnormal hair cell posterior macula increased amount, abnormal
Lawson et al., 2001	Fig. 4		mib1ta52b/ta52b	trunk curved, abnormal trunk pigmentation decreased process quality, abnormal
	Fig. 6		mib1ta52b/ta52b	dorsal aorta disorganized, abnormal dorsal aorta fused with posterior cardinal vein, abnormal head hemorrhagic, abnormal intersegmental vessel disorganized, abnormal intersegmental vessel vascular sprouts mislocalised, abnormal posterior cardinal vein disorganized, abnormal
Gothilf et al., 1999	Fig. 4		mib1ta52b/ta52b	pineal complex aanat2 expression increased distribution, abnormal
Halloran et al., 1999	Fig. 4		mib1ta52b/ta52b	axon guidance process quality, abnormal diencephalon sema3d expression decreased amount, abnormal dorsoventral diencephalic tract axon guidance process quality, abnormal Rohon-Beard neuron axon guidance process quality, abnormal roof plate spinal cord region sema3d expression absent, abnormal
Riley et al., 1999	text only		mib1ta52b/ta52b	otic vesicle has extra parts of type hair cell, abnormal
Haddon et al., 1998	Fig. 1		mib1ta52b/ta52b	otic placode formation increased process quality, abnormal
	Fig. 4		mib1ta52b/ta52b	neural plate primary neuron increased amount, abnormal
	Fig. 5		mib1ta52b/ta52b	auditory epithelial support cell absent, abnormal hair cell anterior macula decreased amount, abnormal hair cell anterior macula increased amount, abnormal hair cell posterior macula decreased amount, abnormal hair cell posterior macula increased amount, abnormal lapillus decreased size, abnormal lapillus hypotrophic, abnormal otolith development arrested, abnormal sagitta decreased size, abnormal sagitta hypotrophic, abnormal
	Fig. 7		mib1ta52b/ta52b	statoacoustic (VIII) ganglion hyperplastic, abnormal statoacoustic (VIII) ganglion increased size, abnormal
	text only		mib1ta52b/ta52b	anterior crista absent, abnormal auditory epithelial support cell absent, abnormal cristae formation absent, abnormal hair cell anterior macula absent, abnormal hair cell posterior macula absent, abnormal inner ear hair cell absent, abnormal posterior crista absent, abnormal
Jiang et al., 1996	Fig. 1		mib1ta52b/ta52b	hindbrain neural keel concave, abnormal rhombomere morphology, abnormal somite border morphology, abnormal trunk melanocyte absent, abnormal
	Fig. 2		mib1ta52b/ta52b	anterior lateral line placode has extra parts of type sensory neuron, abnormal CoSA increased amount, abnormal epiphysis has extra parts of type neuron, abnormal hindbrain interneuron increased amount, abnormal Mauthner neuron increased amount, abnormal medial longitudinal fasciculus increased width, abnormal midbrain hindbrain boundary increased size, abnormal polster increased size, abnormal posterior lateral line placode has extra parts of type sensory neuron, abnormal pronephric duct increased size, abnormal rhombomere increased size, abnormal trigeminal ganglion has extra parts of type sensory neuron, abnormal trigeminal placode increased size, abnormal whole organism anterior-posterior axis has extra parts of type primary motor neuron, abnormal whole organism anterior-posterior axis has extra parts of type Rohon-Beard neuron, abnormal whole organism medial-lateral axis has extra parts of type Rohon-Beard neuron, abnormal
	Fig. 4		mib1ta52b/ta52b	hindbrain dorsal region has fewer parts of type neuron, abnormal hindbrain has fewer parts of type radial glial cell, abnormal optic cup has fewer parts of type radial glial cell, abnormal trunk anterior region has fewer parts of type melanocyte, abnormal
	Fig. 5		mib1ta52b/ta52b	somite decreased amount, abnormal somitogenesis delayed, abnormal
Kelsh et al., 1996	Table 2		mib1ta52b/ta52b	integument has fewer parts of type pigment cell, abnormal post-vent region lacks all parts of type pigment cell, abnormal
Malicki et al., 1996	Fig. 3		mib1m178/m178 (AB)	inner ear has fewer parts of type otolith, abnormal otic vesicle decreased size, abnormal otic vesicle shape, abnormal otolith decreased size, abnormal semicircular canal morphology, abnormal semicircular canal spatial pattern, abnormal
	Table 1		mib1m132/m132 (AB)	inner ear has fewer parts of type otolith, abnormal otic vesicle decreased size, abnormal otic vesicle shape, abnormal otolith decreased distance otolith, abnormal semicircular canal morphology, abnormal
Schier et al., 1996	Fig. 9		mib1m178/m178 (AB)	hindbrain morphology, abnormal neurogenesis disrupted, abnormal
	Fig. T1		mib1m178/m178 (AB) ,   mib1m132/m132 (AB)	fourth ventricle decreased size, abnormal hindbrain morphology, abnormal neurogenesis disrupted, abnormal primary neuron increased amount, abnormal
Stemple et al., 1996	Fig. 6		mib1m132/m132 (AB)	notochord cell spherical, abnormal notochord structure, abnormal post-vent region bent, abnormal
	Fig. T1		mib1m178/m178 (AB) ,   mib1m132/m132 (AB)	central nervous system morphology, abnormal notochord cell spherical, abnormal notochord decreased thickness, abnormal
van Eeden et al., 1996	Table 1		mib1ta52b/ta52b	somite morphology, abnormal somitogenesis disrupted, abnormal