Publication	Data		Fish	Anatomy
Liu et al., 2020	Fig. 1		spi1bhkz4/hkz4	osteoclast decreased amount, abnormal
	Fig. 2		spi1bhkz4/hkz4	osteoclast decreased amount, abnormal
	Fig. 3		spi1bhkz4/hkz4	osteoclast apoptotic process decreased occurrence, abnormal osteoclast apoptotic process increased occurrence, abnormal
	Fig. 4		spi1bhkz4/hkz4	osteoclast decreased amount, abnormal
	Fig. 6		spi1bhkz4/hkz4	osteoclast decreased amount, abnormal
Hassan-Abdi et al., 2019	Figure 2		WT + MO1-spi1b	optic tectum microglial cell ab2-lcp1 labeling absent, abnormal optic tectum microglial cell absent, abnormal whole organism cxcl8a expression decreased amount, abnormal whole organism il1b expression decreased amount, abnormal
Mikdache et al., 2019	Fig. 4		WT + MO1-spi1b	neuron axon decreased amount, abnormal posterior lateral line ganglion apoptotic process increased occurrence, abnormal posterior lateral line ganglion has fewer parts of type neuron, abnormal
Villani et al., 2019	Fig. 1		WT + MO1-spi1b	brain lacks all parts of type microglial cell, abnormal brain phagocytosis decreased occurrence, abnormal
Chernyavskaya et al., 2017	Fig. 4		gl23Tg + MO1-spi1b ,   nz50Tg + MO1-spi1b	head macrophage mCherry expression decreased distribution, abnormal head neutrophil DsRed2 expression decreased distribution, abnormal
Roh-Johnson et al., 2017	Fig. S3		spi1bfh509/fh509	macrophage absent, abnormal
Yu et al., 2017	Fig. 1		spi1bhkz24/hkz24	common myeloid progenitor cebpa expression decreased amount, abnormal macrophage lyz expression decreased amount, abnormal macrophage mfap4.1 expression decreased amount, abnormal macrophage decreased amount, abnormal
	Fig. 4		spi1bhkz24/hkz24	whole organism spi1a expression increased amount, abnormal
	Fig. 6		spi1bhkz4/hkz4 ,   spi1bhkz24/hkz24	central nervous system microglial cell decreased amount, abnormal macrophage mfap4.1 expression decreased amount, abnormal macrophage spi1a expression decreased amount, abnormal
	Fig. 7		spi1bhkz4/hkz4 ,   spi1bhkz24/hkz24	myeloid cell irf8 expression decreased amount, abnormal
Bernut et al., 2015	Fig. 5		ump2Tg + MO2-spi1b + MO3-spi1b	macrophage absent, abnormal macrophage mCherry expression absent, abnormal
He et al., 2015	Fig. S8		TU + MO2-spi1b	ventral wall of dorsal aorta hematopoietic multipotent progenitor cell decreased amount, abnormal
Rossi et al., 2015	Fig. 3		WT + MO1-spi1b	brain lacks all parts of type microglial cell, abnormal microglia development decreased occurrence, abnormal
Tenor et al., 2015	Fig. 1		nz117Tg + MO1-spi1b ,   WT + MO1-spi1b	blood island macrophage decreased amount, abnormal blood island neutrophil decreased amount, abnormal
Espín-Palazón et al., 2014	Fig. 7		WT + MO1-spi1b	whole organism tnfa expression decreased amount, abnormal
Mazaheri et al., 2014	Fig. 2		WT + MO1-spi1b	brain apoptotic cell clearance decreased occurrence, abnormal microglial cell absent, abnormal
Pimtong et al., 2014	Fig. 6		AB + MO1-spi1b	intermediate cell mass of mesoderm drl expression increased amount, abnormal intermediate cell mass of mesoderm drll.1 expression increased amount, abnormal intermediate cell mass of mesoderm drll.2 expression increased amount, abnormal intermediate cell mass of mesoderm drll.3 expression increased amount, abnormal monocyte lcp1 expression decreased amount, abnormal
Flinn et al., 2013	Fig. S7		WT + MO1-spi1b	brain lacks all parts of type microglial cell, abnormal microglia development arrested, abnormal
Helker et al., 2013	Fig. 8		WT + MO1-spi1b	common cardinal vein blood vessel endothelial cell increased amount, abnormal macrophage decreased amount, abnormal
van der Vaart et al., 2013	Fig. 1		AB + MO1-spi1b	phagocytosis decreased occurrence, abnormal
Chen et al., 2012	Fig. 7		la4Tg; zf149Tg + MO1-spi1b	midbrain blood vasculature has fewer parts of type macrophage, abnormal
Jin et al., 2012	Fig. 1		spi1bhkz4/hkz4	macrophage differentiation disrupted, abnormal
Yoo et al., 2012	Fig. 2		uwm4Tg + MO1-spi1b	neutrophil absent, abnormal
Li et al., 2011	Fig. 6		WT + MO6-spi1b	myeloid cell decreased amount, abnormal
Zakrzewska et al., 2010	Fig. 1		WT + MO1-spi1b	myeloid cell absent, abnormal
	Fig. 3		WT + MO1-spi1b	myeloid cell absent, abnormal
Bukrinsky et al., 2009	Fig. 3		WT + MO1-spi1b	blood island lacks parts or has fewer parts of type granulocyte, abnormal blood island lacks parts or has fewer parts of type macrophage, abnormal regulation of myeloid leukocyte differentiation disrupted, abnormal yolk lacks parts or has fewer parts of type granulocyte, abnormal yolk lacks parts or has fewer parts of type macrophage, abnormal
	Fig. 4		WT + MO1-spi1b	regulation of myeloid leukocyte differentiation disrupted, abnormal
	Fig. 5		WT + MO1-spi1b	blood island lacks parts or has fewer parts of type granulocyte, abnormal blood island lacks parts or has fewer parts of type macrophage, abnormal regulation of myeloid leukocyte differentiation disrupted, abnormal yolk lacks parts or has fewer parts of type granulocyte, abnormal yolk lacks parts or has fewer parts of type macrophage, abnormal
Liongue et al., 2009	Fig. 1		WT + MO5-spi1b	erythroid lineage cell increased amount, abnormal erythroid progenitor cell increased amount, abnormal
Walters et al., 2009	Fig. 8		WT + MO1-spi1b	muscle degenerate, abnormal neutrophil decreased amount, abnormal
Peri et al., 2008	Fig. 4		WT + MO1-spi1b	microglial cell absent, abnormal
Mathew et al., 2007	Fig. 9		AB + MO1-spi1b	macrophage absent, abnormal neutrophil absent, abnormal
Mathias et al., 2007	Fig. 4		AB + MO1-spi1b	neutrophil absent, abnormal
Su et al., 2007	Fig. 4		WT + MO3-spi1b	macrophage differentiation disrupted, abnormal myeloid cell decreased amount, abnormal