Publication	Data		Fish	Anatomy
Mauri et al., 2021	Fig. 8		hu5333Tg; hu7135Tg + MO4-dll4	intersegmental artery decreased amount, abnormal intersegmental vein increased amount, abnormal
Helker et al., 2020	Figure 4		bns157Tg + MO4-dll4	angiogenic sprout increased length, abnormal blood vessel EGFP expression increased amount, abnormal
Ando et al., 2019	Fig. 4		ncv69Tg; s939Tg + MO3-dll4	trunk vasculature blood vessel endothelial cell mCherry expression decreased amount, abnormal
Bonkhofer et al., 2019	Fig. 6		dll4sa9436/sa9436 ,   WT + MO1-dll4 ,   WT + MO4-dll4	ventral wall of dorsal aorta runx1 expression decreased amount, abnormal
Page et al., 2019	Fig. 2		ubs1Tg + MO1-dll4	blood vessel endothelial cell cell division increased duration, abnormal intersegmental vein blood vessel endothelial cell increased amount, abnormal
	Fig. 3		WT + MO1-dll4	dorsal aorta tm4sf18 expression increased distribution, abnormal whole organism kdrl expression increased amount, abnormal whole organism tm4sf18 expression increased amount, abnormal
Savage et al., 2019	Fig. 3		sh512Tg; y1Tg + CRISPR2-dll4	dorsal longitudinal anastomotic vessel increased diameter, abnormal dorsal longitudinal anastomotic vessel malformed, abnormal
Bower et al., 2017	Fig. 6		y1Tg + MO4-dll4	intersegmental artery increased branchiness, abnormal
Henke et al., 2017	Fig. 6		dll4dmh34/+ ,   dll4dmh33/+ ,   dll4dmh32/+ ,   dll4dmh20/+	lepidotrichium shortened, abnormal pelvic fin deformed, abnormal
Shin et al., 2016	Fig. S5		la116Tg + MO2-dll4	intersegmental vessel increased branchiness, abnormal
Rochon et al., 2015	Fig. 4		la116Tg + MO2-dll4	basal communicating artery increased size, abnormal
Yokota et al., 2015	Fig. 10		ncv31Tg; ubs4Tg; zf415Tg + MO3-dll4	endothelial cell cytoplasm GCaMP expression spatial pattern, abnormal
	Fig. 8		ncv31Tg; ubs4Tg; zf415Tg + MO3-dll4	endothelial tip cell GCaMP expression spatial pattern, abnormal
Zhou et al., 2015	Fig. 5		AB + MO1-dll4	whole organism uxt expression increased amount, abnormal
Fukuhara et al., 2014	Fig. 3		ncv3Tg; ncv5Tg + MO3-dll4	intersegmental vessel has extra parts of type endothelial cell, abnormal
Le Guen et al., 2014	Fig. 3		y1Tg + MO4-dll4	intersegmental artery increased branchiness, abnormal sprouting angiogenesis growth quality of occurrent, abnormal
Okigawa et al., 2014	Fig. 3		nns5Tg + MO2-dll4	CiD increased amount, abnormal VeLD increased amount, abnormal
Quillien et al., 2014	Fig. S5		dll4j16e1/j16e1	intersegmental artery decreased amount, abnormal
Sacilotto et al., 2013	Fig. S8		WT + MO1-dll4	blood vessel development disrupted, abnormal
Villefranc et al., 2013	Fig. 3		WT + MO2-dll4	angiogenesis disrupted, abnormal intersegmental vessel has extra parts of type endothelial cell, abnormal intersegmental vessel increased branchiness, abnormal
Weijts et al., 2013	Fig. 4		hu5333Tg; y1Tg + MO4-dll4	intersegmental artery decreased amount, abnormal intersegmental vessel increased branchiness, abnormal lymphangioblast cord lymphangioblast decreased amount, abnormal lymphangioblast decreased amount, abnormal lymphangiogenesis disrupted, abnormal sprouting angiogenesis disrupted, abnormal thoracic duct absent, abnormal
Biyashev et al., 2012	Fig. 3		WT + MO6-dll4	intersegmental vessel morphology, abnormal
Bridge et al., 2012	Fig. 4		WT + MO1-dll4	dorsal aorta decreased diameter, abnormal
	Fig. 5		y1Tg + MO7-dll4	angiogenesis process quality, abnormal intersegmental vessel decreased amount, abnormal intersegmental vessel decreased size, abnormal
Geudens et al., 2010	Fig. 1		y1Tg + MO3-dll4 + MO4-dll4	lymphangiogenesis disrupted, abnormal thoracic duct absent, abnormal thoracic duct wholeness, abnormal
	Fig. 2		WT + MO3-dll4 + MO4-dll4 ,   hu4453Tg + MO3-dll4 + MO4-dll4	lymphangioblast cord absent, abnormal
	Fig. 4		y1Tg + MO3-dll4 + MO4-dll4	branching involved in lymph vessel morphogenesis disrupted, abnormal lymphangioblast cord absent, abnormal lymphangiogenesis disrupted, abnormal lymphangiogenic sprout decreased amount, abnormal thoracic duct hypotrophic, abnormal thoracic duct wholeness, abnormal venous endothelial cell migration involved in lymph vessel development disrupted, abnormal
Fukui et al., 2009	Fig. S8		y1Tg + MO3-dll4	cranial vasculature branchiness, abnormal intersegmental vessel cell increased amount, abnormal pericardium edematous, abnormal trunk vasculature branchiness, abnormal
Hogan et al., 2009	Fig. 4		hu4624Tg + MO4-dll4	blood vessel development disrupted, abnormal dorsal aorta has extra parts of type endothelial tip cell, abnormal posterior cardinal vein lacks parts or has fewer parts of type endothelial tip cell, abnormal
	Fig. 5		hu4624Tg + MO4-dll4	cellular response to growth factor stimulus increased occurrence, abnormal
Leslie et al., 2007	Fig. 2		WT + MO3-dll4 ,   y1Tg + MO3-dll4 ,   dll4j16e1/j16e1; y1Tg	blood circulation decreased rate, abnormal blood vessel endothelial cell increased amount, abnormal blood vessel morphogenesis disrupted, abnormal dorsal aorta morphology, abnormal gut blood vasculature increased amount, abnormal intersegmental vessel morphology, abnormal pectoral fin blood vasculature increased amount, abnormal posterior cardinal vein morphology, abnormal
	Fig. 3		y1Tg + MO3-dll4	intersegmental vessel morphology, abnormal regulation of blood vessel endothelial cell migration disrupted, abnormal
Siekmann et al., 2007	Fig. 4		y7Tg + MO1-dll4	intersegmental artery blood vessel endothelial cell increased amount, abnormal
	Fig. S1		y1Tg + MO1-dll4	blood circulation disrupted, abnormal intersegmental artery structure, abnormal
	Fig. S7		y7Tg + MO2-dll4 ,   y7Tg + MO1-dll4	intersegmental artery blood vessel endothelial cell increased amount, abnormal