| Phenotype | Fish | Conditions | Figures | 
    
    
        
            | heart rudiment morphology, abnormal | WT + MO1-gata5 | standard conditions | Fig. 2
                    
                    from Hempel et al., 2017 
 | 
    
        
            | heart physical object quality, abnormal | WT + MO1-gata5 | standard conditions | Fig. 1  from Holtzinger et al., 2007 
 | 
    
        
            | heart cardiac muscle cell decreased amount, abnormal | WT + MO1-gata5 | standard conditions | Fig. 5
                    
                    from Jia et al., 2007 
 | 
    
        
            | heart tube malformed, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | anterior lateral plate mesoderm nkx2.5 expression decreased amount, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4
                    
                    from Wen et al., 2017 
 | 
    
        
            | heart morphology, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4
                    
                    from Wen et al., 2017 
 | 
    
        
            | heart primordium left side unfused from heart primordium right side, abnormal | WT + MO1-gata5 | standard conditions | Fig. 1  from Holtzinger et al., 2007 
 | 
    
        
            | anterior lateral plate mesoderm myocardial precursor myl7 expression decreased amount, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4
                    
                    from Wen et al., 2017 
 | 
    
        
            | heart bifurcated, abnormal | WT + MO1-gata5 | standard conditions | Fig. 1  from Holtzinger et al., 2007 
 | 
    
        
            | heart rudiment bifurcated, abnormal | WT + MO1-gata5 | standard conditions | Fig. 2
                    
                    from Hempel et al., 2017 
 | 
    
        
            | embryonic heart tube development disrupted, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | heart nppa expression spatial pattern, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4
                    
                    from Wen et al., 2017 
 | 
    
        
            | heart decreased size, abnormal | WT + MO1-gata5 | standard conditions | Fig. 5
                    
                    from Jia et al., 2007 
 | 
    
        
            | anterior lateral plate mesoderm myocardial precursor myh7 expression decreased amount, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4
                    
                    from Wen et al., 2017 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | heart nppa expression increased distribution, abnormal | WT + MO1-gata5 | standard conditions | Fig. 4
                    
                    from Wen et al., 2017 
 | 
    
        
            | heart morphogenesis process quality, abnormal | knu9Tg + MO1-gata5 (AB) | standard conditions | Fig. 6  from Park et al., 2009 
 | 
    
        
            | heart bifurcated, abnormal | knu9Tg + MO1-gata5 (AB) | standard conditions | Fig. 6  from Park et al., 2009 
 | 
    
        
            | heart cardiac muscle cell decreased amount, abnormal | hey2m145/m145 + MO1-gata5 | standard conditions | Fig. 5
                    
                    from Jia et al., 2007 
 | 
    
        
            | heart decreased size, abnormal | hey2m145/m145 + MO1-gata5 | standard conditions | Fig. 5
                    
                    from Jia et al., 2007 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | heart formation disrupted, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 3  from Peterkin et al., 2009 
 | 
    
        
            | fin bud aplastic, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 6  from Peterkin et al., 2009 
 | 
    
        
            | whole organism mir210 expression increased amount, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 2
                    
                    from Jia et al., 2018 
 | 
    
        
            | cardioblast migration to the midline involved in heart field formation disrupted, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 3  from Peterkin et al., 2009 
 | 
    
        
            | myeloid cell differentiation decreased occurrence, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 3
                    
                    from Jia et al., 2018 
 | 
    
        
            | otic placode foxj1b expression decreased amount, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. S3
                    
                    from Jia et al., 2018 
 | 
    
        
            | embryonic heart tube development disrupted, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | yolk syncytial layer slc3a2a expression decreased amount, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. S3
                    
                    from Jia et al., 2018 
 | 
    
        
            | granulocyte mpx expression decreased amount, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 3
                    
                    from Jia et al., 2018 
 | 
    
        
            | myeloid cell differentiation disrupted, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 1  from Peterkin et al., 2009 
 | 
    
        
            | heart tube malformed, abnormal | WT + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | WT + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart tube bifurcated, abnormal | WT + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell decreased amount, abnormal | WT + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell differentiation disrupted, abnormal | WT + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell differentiation disrupted, abnormal | WT + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart tube bifurcated, abnormal | WT + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | WT + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell decreased amount, abnormal | WT + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart tube malformed, abnormal | WT + MO1-gata5 + MO3-gata4 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | WT + MO1-gata5 + MO3-gata4 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | embryonic heart tube development disrupted, abnormal | WT + MO1-gata5 + MO3-gata4 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Peterkin et al., 2007 
 | 
    
        
            | otic placode foxj1b expression amount, ameliorated | mir210nju49/nju49 + MO1-gata5 + MO1-gata6 | standard conditions | Fig. S3
                    
                    from Jia et al., 2018 
 | 
    
        
            | myeloid cell differentiation occurrence, ameliorated | mir210nju49/nju49 + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 3
                    
                    from Jia et al., 2018 
 | 
    
        
            | yolk syncytial layer slc3a2a expression amount, ameliorated | mir210nju49/nju49 + MO1-gata5 + MO1-gata6 | standard conditions | Fig. S3
                    
                    from Jia et al., 2018 
 | 
    
        
            | granulocyte mpx expression amount, ameliorated | mir210nju49/nju49 + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 3
                    
                    from Jia et al., 2018 
 | 
    
        
            | heart morphogenesis arrested, abnormal | WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell decreased amount, abnormal | WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell differentiation disrupted, abnormal | WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart tube bifurcated, abnormal | WT + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart duplicated, abnormal | cy13Tg + MO1-gata5 | standard conditions | Fig. 3  from Lin et al., 2013 
 | 
    
        
            | lateral mesoderm EGFP expression decreased amount, abnormal | hsc70Tg + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 2  from Yuan et al., 2018 
 | 
    
        
            | margin EGFP expression absent, abnormal | hsc70Tg + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 2  from Yuan et al., 2018 
 | 
    
        
            | heart physical object quality, abnormal | twu34Tg + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 2  from Holtzinger et al., 2007 
 | 
    
        
            | cardiac muscle cell decreased amount, abnormal | twu34Tg + MO1-gata5 + MO1-gata6 | standard conditions | Fig. 2  from Holtzinger et al., 2007 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | twu34Tg + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart bifurcated, abnormal | twu34Tg + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell differentiation disrupted, abnormal | twu34Tg + MO1-gata5 + MO1-smarcd3b | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell differentiation disrupted, abnormal | twu34Tg + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | twu34Tg + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart bifurcated, abnormal | twu34Tg + MO1-gata5 + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart physical object quality, abnormal | twu34Tg + MO1-gata5 + MO2-gata4 | standard conditions | Fig. 2  from Holtzinger et al., 2007 
 | 
    
        
            | heart bifurcated, abnormal | twu34Tg + MO1-gata5 + MO2-gata4 | standard conditions | Fig. 2  from Holtzinger et al., 2007 
 | 
    
        
            | heart morphology, abnormal | el133Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. S1  from Lou et al., 2011 
 | 
    
        
            | pharyngeal musculature morphology, abnormal | el133Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. S1  from Lou et al., 2011 
 | 
    
        
            | whole organism EGFP expression absent, abnormal | knu9Tg + MO1-gata5 + MO1-gata6 + MO5-gata4 (AB) | standard conditions | Fig. 6  from Park et al., 2009 
 | 
    
        
            | heart absent, abnormal | knu9Tg + MO1-gata5 + MO1-gata6 + MO5-gata4 (AB) | standard conditions | Fig. 6  from Park et al., 2009 
 | 
    
        
            | endocardium aplastic, abnormal | s843Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. S1  from Lou et al., 2011 
 | 
    
        
            | heart morphogenesis disrupted, abnormal | twu34Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | cardiac muscle cell differentiation disrupted, abnormal | twu34Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 | 
    
        
            | heart bifurcated, abnormal | twu34Tg + MO1-gata5 + MO1-smarcd3b + MO1-tbx5a | standard conditions | Fig. 1  from Lou et al., 2011 
 |