Publication	Data		Fish	Anatomy
Nittoli et al., 2019	Fig. 7		gli2aty119/ty119	somite uncx4.1 expression spatial pattern, abnormal
Milla et al., 2012	Fig. 5		gli2aty17a/ty17a	eye myca expression decreased amount, abnormal liver myca expression decreased amount, abnormal optic tectum myca expression decreased amount, abnormal
	Fig. 6		gli2aty17a/ty17a	somite sfrp2 expression decreased amount, abnormal
Wiweger et al., 2012	Fig. 4		gli2aty119/ty119	ceratobranchial 5 bone decreased length, abnormal ceratobranchial 5 bone has fewer parts of type ceratobranchial 5 tooth, abnormal ceratobranchial 5 tooth decreased length, abnormal ceratobranchial 5 tooth increased width, abnormal
Bergeron et al., 2011	Fig. 1		gli2aty119/ty119	forebrain physical object quality, abnormal forebrain regionalization process quality, abnormal
Lobbardi et al., 2011	Fig. 4		WT + MO2-gli2a	adaxial cell physical object quality, abnormal myotome malformed, abnormal skeletal muscle tissue development process quality, abnormal slow muscle cell mislocalised, abnormal smoothened signaling pathway process quality, abnormal
	Fig. 6		gli2aty119/ty119	muscle cell differentiation decreased occurrence, abnormal muscle pioneer absent, abnormal slow muscle cell absent, abnormal smoothened signaling pathway decreased occurrence, abnormal
Schwend et al., 2010	Fig. 1		gli2aty119/ty119	post-vent region curved ventral, abnormal somite U-shaped, abnormal
Devine et al., 2009	Fig. 3		gli2aty17a/ty17a ,   WT + MO1-gli2a	adenohypophysis morphogenesis process quality, abnormal adenohypophysis wholly anteriorized, abnormal pars anterior physical object quality, abnormal
	Fig. 4		WT + MO1-gli2a	adrenocorticotropic hormone secreting cell decreased amount, abnormal corticotropin hormone secreting cell differentiation process quality, abnormal somatotropin secreting cell decreased amount, abnormal
	Fig. 5		WT + MO1-gli2a	adenohypophysis morphogenesis process quality, abnormal adenohypophysis physical object quality, abnormal
Parkin et al., 2009	Fig. 3		gli2aty119/ty119	proctodeum atretic, abnormal proctodeum malformed, abnormal
Paulus et al., 2009	Fig. 3		gli2aty17a/ty17a (AB)	axon guidance disrupted, abnormal Rohon-Beard neuron morphology, abnormal sensory neuron mislocalised, abnormal sensory neuron morphology, abnormal
Ke et al., 2008	Fig. 5		gli2aty17a/ty17a	facial nerve motor nucleus mislocalised anteriorly, abnormal trigeminal motor nucleus aplastic, abnormal vagal lobe aplastic, abnormal
	Fig. 6		gli2aty17a/ty17a	floor plate formation disrupted, abnormal
	Fig. S1		WT + MO2-gli2a	horizontal myoseptum morphology, abnormal
Swindell et al., 2008	Fig. 3		gli2aty119/ty119	adenohypophysis aplastic, abnormal lens increased amount, abnormal lens mislocalised, abnormal
Daggett et al., 2007	Fig. 4		gli2aty119/ty119 (AB)	adaxial cell disorganized, abnormal
Jin et al., 2007	Fig. Table 1		gli2as406/s406 (TL)	blood vessel morphogenesis disrupted, abnormal dorsal aorta morphology, abnormal posterior cardinal vein morphology, abnormal
Schafer et al., 2007	Fig. 4		gli2aty17a/ty17a ,   gli2aty17a/+	floor plate formation disrupted, abnormal lateral floor plate composition, abnormal
Feng et al., 2006	Fig. 2		gli2aty119/ty119 (AB)	muscle cell differentiation decreased occurrence, abnormal somite has extra parts of type muscle precursor cell, abnormal
Phenotype Annotation (1994-2006)	Fig. for (ty119)		gli2aty119/ty119	brain quality, abnormal musculoskeletal movement quality, abnormal nervous system quality, abnormal neural tube quality, abnormal response to external stimulus quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
	Fig. for (ty17a)		gli2aty17a/ty17a	brain quality, abnormal hindbrain quality, abnormal myoseptum quality, abnormal nervous system quality, abnormal retinal ganglion cell axon guidance quality, abnormal somite quality, abnormal whole organism lethal (sensu genetics), abnormal
Karlstrom et al., 2003	Fig. 1		gli2aty119/ty119	somite U-shaped, abnormal whole organism curved ventral, abnormal
Wallace et al., 2003	Fig. 9		gli2aty119/ty119	esophageal epithelium disorganized, abnormal esophageal epithelium epithelial cell differentiation disrupted, abnormal esophagus malformed, abnormal
Wolff et al., 2003	Fig. 5		gli2aty119/ty119 + MO2-gli2a ,   WT + MO2-gli2a	fast muscle cell increased amount, abnormal muscle pioneer mislocalised, abnormal somite U-shaped, abnormal
	Fig. 7		gli2aty119/ty119	fast muscle cell absent, abnormal muscle pioneer absent, abnormal
Du et al., 2001	Fig. 2		gli2aty119/ty119	slow muscle cell absent, abnormal striated muscle cell development arrested, abnormal
Majumdar et al., 2000	Fig. 2		gli2aty119/ty119	cell migration involved in kidney development disrupted, abnormal pronephric glomerulus increased amount, abnormal pronephric glomerulus mislocalised laterally, abnormal
Karlstrom et al., 1999	Fig. 4		gli2aty119/ty119	anterior commissure decreased size, abnormal optic chiasm absent, abnormal optic nerve formation disrupted, abnormal postoptic commissure absent, abnormal retinal ganglion cell axon guidance disrupted, abnormal
van Eeden et al., 1998	Fig. 1		gli2aty119/ty119	adaxial cell absent, abnormal
	Fig. 2		gli2aty119/ty119	horizontal myoseptum absent, abnormal
Brand et al., 1996	Fig. 2		gli2aty17a/ty17a ,   gli2aty119/ty119	eye decreased distance eye, abnormal eye mislocalised medially, abnormal
	Fig. 7		gli2aty17a/ty17a ,   gli2aty119/ty119	optic chiasm malformed, abnormal retinal ganglion cell axon guidance disrupted, abnormal
	Fig. 8		gli2aty17a/ty17a ,   gli2aty119/ty119	ethmoid cartilage aplastic, abnormal trabecula cranii fused with trabecula cranii, abnormal
	Table 1		gli2aty17a/ty17a ,   gli2aty119/ty119	floor plate hypoplastic, abnormal myoseptum aplastic, abnormal post-vent region curved ventral, abnormal whole organism movement behavioral quality, abnormal
Chen et al., 1996	Table 1		gli2aty17a/ty17a ,   gli2aty119/ty119	blood circulation disrupted, abnormal
Granato et al., 1996	Table 4		gli2aty17a/ty17a ,   gli2aty119/ty119	larval locomotory behavior process quality, abnormal whole organism decreased behavioural activity, abnormal
Karlstrom et al., 1996	Fig. 1		gli2aty17a/ty17a	retinal ganglion cell axon guidance process quality, abnormal retinal ganglion cell axon ipsilateral to eye, abnormal retinal ganglion cell axon misrouted, abnormal
	Fig. 2		gli2aty17a/ty17a	retinal ganglion cell axon displaced to diencephalon, abnormal retinal ganglion cell axon ipsilateral to eye, abnormal third ventricle increased size, abnormal
	Table 1		gli2aty119/ty119	retinal ganglion cell axon displaced to diencephalon, abnormal retinal ganglion cell axon guidance process quality, abnormal retinal ganglion cell axon ipsilateral to eye, abnormal retinal ganglion cell axon misrouted, abnormal third ventricle increased size, abnormal
Piotrowski et al., 1996	Table 2		gli2aty17a/ty17a ,   gli2aty119/ty119	chondrocranium morphology, abnormal cranial neural crest cell mislocalised ventrally, abnormal
van Eeden et al., 1996	Fig. 5		gli2aty119/ty119	CaP motoneuron axon decreased amount, abnormal central nervous system projection neuron axonogenesis disrupted, abnormal MiP motor neuron axon decreased amount, abnormal
	Fig. 8		gli2aty119/ty119	blood circulation disrupted, abnormal eye decreased distance eye, abnormal horizontal myoseptum aplastic, abnormal pericardium edematous, abnormal pharyngeal arch 3-7 mislocalised ventrally, abnormal post-vent region curved, abnormal
	Fig. 9		gli2aty119/+ ,   gli2aty119/ty119	adaxial cell absent, abnormal adaxial cell decreased amount, abnormal somite lacks all parts of type muscle pioneer, abnormal
	Table 1		gli2aty17a/ty17a	blood circulation disrupted, abnormal horizontal myoseptum hypoplastic, abnormal retinal ganglion cell axon guidance disrupted, abnormal somite morphology, abnormal somitogenesis disrupted, abnormal whole organism movement behavioral quality, abnormal
	Table 3		gli2aty119/ty119	retinal ganglion cell axon guidance disrupted, abnormal whole organism movement behavioral quality, abnormal