Publication	Data		Fish	Anatomy
Thyme et al., 2019	Fig. S4		ambra1ba224/a224	prepulse inhibition increased magnitude, abnormal response to absence of light decreased process quality, abnormal startle response decreased magnitude, abnormal swimming decreased frequency, abnormal swimming increased linear velocity, abnormal swimming increased spatial extent of a process, abnormal
Skobo et al., 2014	Fig. 1		AB + MO1-ambra1b	embryo development disrupted, abnormal post-vent region curved, abnormal whole organism decreased size, abnormal whole organism refractivity, abnormal
	Fig. 3		AB + MO2-ambra1b ,   AB + MO1-ambra1b	muscle cell disorganized, abnormal muscle cell nucleus increased amount, abnormal myoseptum decreased thickness, abnormal myoseptum morphology, abnormal
	Fig. 4		AB + MO1-ambra1b	muscle cell nucleus morphology, abnormal myoseptum unstructured, abnormal trunk musculature muscle cell disorganized, abnormal
	Fig. 5		AB + MO1-ambra1b	cell population proliferation increased occurrence, abnormal
	Fig. 7		AB + MO2-ambra1b ,   AB + MO1-ambra1b	muscle cell degenerate, abnormal muscle cell disorganized, abnormal muscle disorganized, abnormal muscle sarcomere decreased amount, abnormal muscle sarcomere disorganized, abnormal
	Fig. 8		AB + MO1-ambra1b	muscle cell disorganized, abnormal muscle cell nucleus morphology, abnormal muscle mitochondrion morphology, abnormal muscle sarcoplasmic reticulum dilated, abnormal muscle striated muscle thin filament separated from muscle myosin filament, abnormal
	Fig. 9		AB + MO2-ambra1b ,   AB + MO1-ambra1b	fast muscle cell undulate, abnormal muscle cell disorganized, abnormal myoseptum morphology, abnormal myosin filament organization disrupted, abnormal skeletal myofibril assembly disrupted, abnormal somite shape, abnormal
	Fig. S3		AB + MO1-ambra1b	hatching disrupted, abnormal
	Fig. S4		AB + MO1-ambra1b	swimming process quality, abnormal thigmotaxis disrupted, abnormal
Benato et al., 2013	Fig. 11		WT + MO1-ambra1b ,   WT + MO2-ambra1b	autophagy decreased process quality, abnormal
	Fig. 12		WT + MO1-ambra1b ,   WT + MO2-ambra1b	apoptotic process increased occurrence, abnormal head apoptotic, abnormal
	Fig. 6		WT + MO1-ambra1b ,   WT + MO1-ambra1b + MO4-tp53 ,   WT + MO2-ambra1b	embryo development disrupted, abnormal eye decreased size, abnormal head decreased size, abnormal otolith decreased size, abnormal pigmentation delayed, abnormal post-vent region kinked, abnormal trunk decreased size, abnormal whole organism deformed, abnormal
	Fig. 7		WT + MO1-ambra1b	eye decreased size, abnormal fourth ventricle hydrocephalic, abnormal notochord disorganized, abnormal post-vent region deformed, abnormal tectal ventricle hydrocephalic, abnormal
	Fig. 8		WT + MO1-ambra1b	eye decreased size, abnormal pericardium edematous, abnormal yolk syncytial layer edematous, abnormal
	Fig. 9		WT + MO2-ambra1b	eye decreased size, abnormal fourth ventricle hydrocephalic, abnormal otic vesicle decreased size, abnormal pericardium edematous, abnormal post-vent region kinked, abnormal
	Fig. S3		WT + MO1-ambra1b	intersegmental vessel morphology, abnormal subintestinal vein morphology, abnormal
	text only		WT + MO1-ambra1b ,   WT + MO2-ambra1b	whole organism dead, abnormal