Publication	Data		Fish	Anatomy
He et al., 2022	Fig. S5		WT + MO1-spns1	whole organism lysophosphatidylcholine increased amount, abnormal whole organism lysophosphatidylethanolamine increased amount, abnormal yolk physical object quality, abnormal
Coffey et al., 2021	Fig. 1		spns1hi891Tg/hi891Tg	myotome fast muscle cell degenerate, abnormal myotome muscle tendon junction disorganized, abnormal myotome skeletal muscle cell detached from vertical myoseptum muscle tendon junction, abnormal myotome skeletal muscle cell disorganized, abnormal myotome slow muscle cell degenerate, abnormal post-vent region regulation of lysosomal lumen pH decreased process quality, abnormal trunk regulation of lysosomal lumen pH decreased process quality, abnormal
	Fig. 2		spns1hi891Tg/hi891Tg	vertical myoseptum muscle tendon junction ab1-lam labeling increased amount, abnormal
	Fig. 3		spns1hi891Tg/hi891Tg	vertical myoseptum muscle tendon junction ab1-lam labeling increased amount, abnormal
	Fig. 5		spns1hi891Tg/hi891Tg	vertical myoseptum muscle tendon junction ab1-lam labeling increased amount, abnormal
	Fig. 6		spns1hi891Tg/hi891Tg	myotome sarcolemma permeable, abnormal skeletal muscle cell regulation of membrane permeability decreased process quality, abnormal
	Fig. 7		spns1hi891Tg/hi891Tg	myotome skeletal muscle cell mmp9 expression increased amount, abnormal whole organism mmp9 expression increased amount, abnormal
	Fig. 8		spns1hi891Tg/hi891Tg; nc1Tg ,   spns1hi891Tg/hi891Tg	myotome skeletal muscle cell mmp9 expression increased amount, abnormal myotome skeletal muscle cell degenerate, abnormal
	Fig. text statement (non-figure)		spns1hi891Tg/hi891Tg	whole organism tnfsf12 expression increased amount, abnormal
Sasaki et al., 2017	Fig. 1		WT + MO1-spns1 ,   spns1hi891Tg/hi891Tg	whole organism decreased life span, abnormal
	Fig. 2		WT + MO1-spns1 ,   spns1hi891Tg/hi891Tg	whole organism decreased life span, abnormal yolk opaque, abnormal
	Fig. 3		spns1hi891Tg/hi891Tg	cell phagophore ab4-map1lc3 labeling increased amount, abnormal
	Fig. 7		spns1hi891Tg/hi891Tg	whole organism decreased life span, abnormal
Sasaki et al., 2014	Fig. 2		WT + MO1-spns1	whole organism decreased life span, abnormal yolk opaque, abnormal
	Fig. 4		spns1hi891Tg/hi891Tg	cellular senescence increased process quality, abnormal
	Fig. 5		spns1hi891Tg/hi891Tg	cellular senescence increased process quality, abnormal yolk opaque, abnormal
Kishi et al., 2008	Fig. 3		spns1hi891Tg/hi891Tg	cellular senescence increased magnitude, abnormal yolk opaque, abnormal
	Fig. 8		spns1hi891Tg/hi891Tg	cellular senescence increased magnitude, abnormal liver composition, abnormal muscle composition, abnormal
	Fig. 9		spns1hi891Tg/+	whole organism viability, abnormal
	Fig. S2		WT + MO1-spns1	cellular senescence increased magnitude, abnormal yolk opaque, abnormal
	text only		spns1hi891Tg/hi891Tg	cellular senescence increased magnitude, abnormal extension decreased thickness, abnormal whole organism lethal (sensu genetics), abnormal
Phenotype Annotation (1994-2006)	Fig. for (hi891)		spns1hi891Tg	whole organism quality, abnormal
Amsterdam et al., 2004	text only		spns1hi891Tg/hi891Tg	extension absent, abnormal extension decreased thickness, abnormal trunk decreased thickness, abnormal whole organism edematous, abnormal whole organism necrotic, abnormal whole organism viability, abnormal yolk dark grey, abnormal yolk necrotic, abnormal