| Phenotype | Fish | Conditions | Figures | 
    
    
        
            | blood vessel nr2f1b expression increased distribution, abnormal | TL + MO4-rbpja,rbpjb | control | Fig. 6  from Li et al., 2015 
 | 
    
        
            | whole organism nr2f1b expression increased distribution, abnormal | TL + MO4-rbpja,rbpjb | control | Fig. 6  from Li et al., 2015 
 | 
    
        
            | pronephric tubule morphology, abnormal | TU + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | interrenal primordium increased size, abnormal | TU + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | pronephric podocyte decreased amount, abnormal | TU + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | central nervous system cellular quality, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 5
                    
                    from Yeo et al., 2007 
 | 
    
        
            | angiogenic sprout increased amount, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | nucleate erythrocyte mislocalised, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. S2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | fin regeneration decreased process quality, abnormal | WT + MO4-rbpja,rbpjb | physical alteration: anatomical structure | Fig. 3  from Münch et al., 2013 
 | 
    
        
            | ventricular system distended, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. S2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | posterior lateral line ganglion neuron increased amount, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 1
                    
                    from Mizoguchi et al., 2011 
 | 
    
        
            | ventricular system increased accumulation nucleate erythrocyte, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. S2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | hindbrain epithelium disorganized, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Qiu et al., 2009 
 | 
    
        
            | cranial vasculature broken, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. S2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | intersegmental vessel structure, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | rhombomere boundary formation disrupted, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Qiu et al., 2009 
 | 
    
        
            | blood vessel endothelial cell proliferation involved in sprouting angiogenesis process quality, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | hindbrain development disrupted, abnormal | WT + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Qiu et al., 2009 
 | 
    
        
            | pronephric podocyte decreased amount, abnormal | TU + MO1-foxc1a + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | pronephric tubule morphology, abnormal | TU + MO1-foxc1a + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | interrenal primordium increased size, abnormal | TU + MO1-foxc1a + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | pronephric tubule absent, abnormal | TU + MO3-wt1a + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | interrenal primordium increased size, abnormal | TU + MO3-wt1a + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | pronephric podocyte decreased amount, abnormal | TU + MO3-wt1a + MO4-rbpja,rbpjb | standard conditions | Fig. 3  from O'Brien et al., 2011 
 | 
    
        
            | whole organism lacks all parts of type dorsal aorta, abnormal | WT + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  from Sacilotto et al., 2013 
 | 
    
        
            | axial blood vessel decreased amount, abnormal | WT + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 5  from Sacilotto et al., 2013 
 | 
    
        
            | artery development arrested, abnormal | WT + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 4  , 
                    
                        Fig. 5  , 
                    
                        Fig. S8  from Sacilotto et al., 2013 
 | 
    
        
            | pronephros lacks all parts of type pronephric podocyte, abnormal | hnf1bahi1843Tg/hi1843Tg + MO1-hnf1bb + MO3-wt1a + MO4-rbpja,rbpjb | standard conditions | Fig. 4
                    
                    from Naylor et al., 2013 
 | 
    
        
            | podocyte differentiation decreased occurrence, abnormal | hnf1bahi1843Tg/hi1843Tg + MO1-hnf1bb + MO3-wt1a + MO4-rbpja,rbpjb | standard conditions | Fig. 4
                    
                    from Naylor et al., 2013 
 | 
    
        
            | peripheral neuron decreased length, abnormal | knu2Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4
                    
                    from So et al., 2009 
 | 
    
        
            | blood vessel endothelial cell proliferation involved in sprouting angiogenesis process quality, abnormal | lcr1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | trunk vasculature EGFP expression decreased amount, abnormal | lcr1Tg + MO4-rbpja,rbpjb | control | Fig. 3  from Overman et al., 2017 
 | 
    
        
            | angiogenic sprout increased amount, abnormal | lcr1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | intersegmental vessel structure, abnormal | lcr1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | trunk vasculature EGFP expression decreased amount, abnormal | lcr1Tg + MO4-rbpja,rbpjb | chemical treatment by environment: pharmaceutical | Fig. 3  from Overman et al., 2017 
 | 
    
        
            | whole organism EGFP expression decreased amount, abnormal | lcr1Tg + MO4-rbpja,rbpjb | chemical treatment by environment: pharmaceutical | Fig. 3  from Overman et al., 2017 
 | 
    
        
            | intersegmental vessel increased branchiness, abnormal | s843Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 7  from Zhou et al., 2015 
 | 
    
        
            | intersegmental vessel angiogenesis increased process quality, abnormal | s843Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 7  from Zhou et al., 2015 
 | 
    
        
            | blood circulation disrupted, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. S1
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | intersegmental vessel structure, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 1
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | intersegmental vessel increased mass density, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Jensen et al., 2012 
 | 
    
        
            | blood vessel development disrupted, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 5  from Sacilotto et al., 2013 
 | 
    
        
            | intersegmental vessel disorganized, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Jensen et al., 2012 
 | 
    
        
            | intersegmental vessel has extra parts of type blood vessel, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Jensen et al., 2012 
 | 
    
        
            | intersegmental vessel mislocalised, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 1
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | intersegmental vessel has extra parts of type vascular sprouts, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 4  from Jensen et al., 2012 
 | 
    
        
            | intersegmental artery structure, abnormal | y1Tg + MO4-rbpja,rbpjb | standard conditions | Fig. S1
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | intersegmental vessel structure, abnormal | y7Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | blood vessel endothelial cell increased amount, abnormal | y7Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | blood vessel endothelial cell proliferation involved in sprouting angiogenesis process quality, abnormal | y7Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | cell migration involved in sprouting angiogenesis process quality, abnormal | y7Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 2
                    
                    from Siekmann et al., 2007 
 | 
    
        
            | intersegmental artery blood vessel endothelial cell increased amount, abnormal | y7Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 5  from Lamont et al., 2016 Fig. 3, 
                    
                        Fig. S5
                    
                    from Siekmann et al., 2007
 
 | 
    
        
            | dorsal longitudinal anastomotic vessel blood vessel development occurrence, ameliorated | s843Tg + MO2-uxt + MO4-rbpja,rbpjb | standard conditions | Fig. 7  from Zhou et al., 2015 
 | 
    
        
            | intersegmental vessel angiogenesis process quality, ameliorated | s843Tg + MO2-uxt + MO4-rbpja,rbpjb | standard conditions | Fig. 7  from Zhou et al., 2015 
 | 
    
        
            | intersegmental artery blood vessel endothelial cell amount, ameliorated | y7Tg + MO4-isl2a + MO4-rbpja,rbpjb + MO5-isl2a | control | Fig. 5  from Lamont et al., 2016 
 | 
    
        
            | artery development arrested, abnormal | lcr1Tg + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | whole organism lacks all parts of type dorsal aorta, abnormal | lcr1Tg + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 4  from Sacilotto et al., 2013 
 | 
    
        
            | artery development arrested, abnormal | y1Tg + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 5  from Sacilotto et al., 2013 
 | 
    
        
            | axial blood vessel decreased amount, abnormal | y1Tg + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. 5  from Sacilotto et al., 2013 
 | 
    
        
            | intersegmental vessel cell increased amount, abnormal | ci5Tg; y7Tg + MO4-rbpja,rbpjb | standard conditions | Fig. 6  from Li et al., 2015 
 | 
    
        
            | intersegmental vessel cell amount, ameliorated | ci5Tg; y7Tg + MO4-nr2f1b + MO4-rbpja,rbpjb | standard conditions | Fig. 6  from Li et al., 2015 
 | 
    
        
            | ventral wall of dorsal aorta endothelial cell EGFP expression absent, abnormal | gfi1aaqmc551Gt/+; sd2Tg + MO4-rbpja,rbpjb | control | Fig. 4  from Thambyrajah et al., 2016 
 | 
    
        
            | hematopoietic multipotent progenitor cell EGFP expression absent, abnormal | gfi1aaqmc551Gt/+; sd2Tg + MO4-rbpja,rbpjb | control | Fig. 4  from Thambyrajah et al., 2016 
 | 
    
        
            | ventral wall of dorsal aorta hematopoietic cell EGFP expression absent, abnormal | gfi1aaqmc551Gt/+; sd2Tg + MO4-rbpja,rbpjb | control | Fig. 4  from Thambyrajah et al., 2016 
 | 
    
        
            | blood circulation arrested, abnormal | s843Tg; sd2Tg + MO4-rbpja,rbpjb + MO5-sox18 + MO5-sox7 | standard conditions | Fig. S7  from Sacilotto et al., 2013 
 |