Phenotype
|
Fish
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Conditions
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Figures
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endoderm increased width, abnormal
|
ha01Tg + MO1-fn1a + MO4-tp53
|
standard conditions
|
Fig. S6
from Hu et al., 2018
|
lateral rectus tissue regeneration delayed, abnormal
|
zf13Tg + MO1-fn1a
|
ablation: lateral rectus
|
Fig. 6
from Louie et al., 2017
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lateral rectus tissue regeneration decreased efficacy, abnormal
|
zf13Tg + MO1-fn1a
|
ablation: lateral rectus
|
Fig. 6
from Louie et al., 2017
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pharyngeal arch 1 decreased length, abnormal
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s1pr1ko311/ko311 + MO1-fn1a
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standard conditions
|
Fig. 3
from Hisano et al., 2013
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pharyngeal arch 1 decreased length, abnormal
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s1pr2ko322/ko322 + MO1-fn1a
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standard conditions
|
Fig. 3
from Hisano et al., 2013
|
ventral mandibular arch disorganized, abnormal
|
s1pr2ko322/ko322 + MO1-fn1a
|
standard conditions
|
Fig. 3
from Hisano et al., 2013
|
myotome myosin filament disorganized, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 2 ,
Fig. 3 ,
Fig. 4
from Snow et al., 2008
|
myotome actin filament increased length, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 2 ,
Fig. 3
from Snow et al., 2008
|
myotome variant shape, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 1 ,
Fig. 2
from Snow et al., 2008
|
somite border aplastic, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 1
from Snow et al., 2008
|
somitogenesis disrupted, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 1
from Snow et al., 2008
|
myotome actin filament disorganized, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 4
from Snow et al., 2008
|
somite border shape, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 1 ,
Fig. 2
from Snow et al., 2008
|
myotome muscle tendon junction morphology, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 2
from Snow et al., 2008
|
somite shape, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 1
from Snow et al., 2008
|
skeletal muscle fiber development disrupted, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 2 ,
Fig. 3
from Snow et al., 2008
|
myotome increased size, abnormal
|
WT + MO1-fn1a + MO1-fn1b + MO2-fn1b
|
standard conditions
|
Fig. 1
from Snow et al., 2008
|
endoderm increased width, abnormal
|
ha01Tg + MO1-fn1a + MO1-lamb1a + MO4-tp53
|
standard conditions
|
Fig. 6 ,
Fig. S6
from Hu et al., 2018
|
endoderm increased width, abnormal
|
ha01Tg + MO1-fn1a + MO4-tp53 + MO5-lama1
|
standard conditions
|
Fig. S6
from Hu et al., 2018
|
ventral wall of dorsal aorta runx1 expression decreased amount, abnormal
|
ncv2Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 6
from Rho et al., 2019
|
ventral wall of dorsal aorta myb expression decreased amount, abnormal
|
ncv2Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 6
from Rho et al., 2019
|
somite border ab2-fn labeling absent, abnormal
|
ncv2Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 5
from Rho et al., 2019
|
angioblast cell migration from lateral mesoderm to midline process quality, abnormal
|
ncv2Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 5
from Rho et al., 2019
|
posterior lateral plate mesoderm cell morphology, abnormal
|
ncv2Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 5
from Rho et al., 2019
|
angioblast cell migration from lateral mesoderm to midline process quality, abnormal
|
y1Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 5
from Rho et al., 2019
|
posterior lateral plate mesoderm cell decreased adhesivity, abnormal
|
hzm7Et; ncv2Tg + MO1-fn1a + MO1-fn1b
|
control
|
Fig. 5
from Rho et al., 2019
|
heart primordium mislocalised laterally, abnormal
|
s1pr2ko322/ko322; ko07Tg + MO1-fn1a
|
standard conditions
|
Fig. 3
from Hisano et al., 2013
|
cardioblast migration to the midline involved in heart rudiment formation absent, abnormal
|
s1pr2ko322/ko322; ko07Tg + MO1-fn1a
|
standard conditions
|
Fig. 3
from Hisano et al., 2013
|
cardioblast migration to the midline involved in heart rudiment formation disrupted, abnormal
|
s1pr2ko322/ko322; ko07Tg + MO1-fn1a
|
standard conditions
|
Fig. 3
from Hisano et al., 2013
|
heart rudiment increased amount, abnormal
|
s1pr2ko322/ko322; ko07Tg + MO1-fn1a
|
standard conditions
|
Fig. 3
from Hisano et al., 2013
|
endoderm increased width, ameliorated
|
gpc4fr6/fr6; ha01Tg + MO1-fn1a + MO1-lamb1a + MO4-tp53
|
standard conditions
|
Fig. 6
from Hu et al., 2018
|