Fig. 7
- ID
- ZDB-FIG-250304-7
- Publication
- Chen et al., 2025 - NDUFB7 mutations cause brain neuronal defects, lactic acidosis, and mitochondrial dysfunction in humans and zebrafish
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Knockdown of Ndufb7 reduces the oxygen consumption rate. We treated one-cell stage zebrafish embryos without or with indicated Ndufb7 translational-blocking morpholino oligonucleotides (tMO), and Ndufb7 mRNA or Mitoquinone mesylate (MitoQ) as described in Fig. |
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Knockdown Reagent: | |
Observed In: | |
Stage: | Prim-5 |