ZFIN ID: ZDB-FIG-200806-3
Espín-Palazón et al., 2014 - Proinflammatory signaling regulates hematopoietic stem cell emergence. Cell   159:1070-85 Full text @ Cell
ADDITIONAL FIGURES
EXPRESSION / LABELING:
Genes:
Fish:
Condition:
Knockdown Reagents:
Anatomical Terms:
Stage: Prim-5
PHENOTYPE:
Fish:
Condition:
Knockdown Reagents:
Observed In:
Stage: Prim-5

Fig. 3 ZFIN is incorporating published figure images and captions as part of an ongoing project. Figures from some publications have not yet been curated, or are not available for display because of copyright restrictions.

Gene Expression Details
Gene Antibody Fish Conditions Stage Anatomy Assay
EGFP s896Tg; um14Tg standard conditions Prim-5 ventral wall of dorsal aorta IFL
s896Tg; um14Tg + MO2-tnfrsf1b standard conditions Prim-5 ventral wall of dorsal aorta IFL
s896Tg; um14Tg + MO4-tnfa standard conditions Prim-5 ventral wall of dorsal aorta IFL
mCherry s896Tg; um14Tg standard conditions Prim-5 endothelial cell IFL
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell IFL
s896Tg; um14Tg + MO2-tnfrsf1b standard conditions Prim-5 endothelial cell IFL
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell IFL
s896Tg; um14Tg + MO4-tnfa standard conditions Prim-5 endothelial cell IFL
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell IFL
runx1 bw9Tg; kca3Tg standard conditions Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
bw9Tg; kca3Tg + MO2-tnfrsf1b standard conditions Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
bw9Tg; kca3Tg + MO4-tnfa standard conditions Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
kca3Tg; kca4Tg standard conditions Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
kca3Tg; kca4Tg heat shock Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
kca3Tg; kca4Tg + MO2-tnfrsf1b standard conditions Prim-5 ventral wall of dorsal aorta ISH
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
kca3Tg; kca4Tg + MO2-tnfrsf1b heat shock Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
kca3Tg; kca4Tg + MO4-tnfa standard conditions Prim-5 ventral wall of dorsal aorta ISH
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
kca3Tg; kca4Tg + MO4-tnfa heat shock Prim-5 ventral wall of dorsal aorta hematopoietic stem cell ISH
Antibody Labeling Details No data available
Phenotype Details
Fish Conditions Stage Phenotype
bw9Tg; kca3Tg + MO2-tnfrsf1b standard conditions Prim-5 ventral wall of dorsal aorta hematopoietic stem cell amount, ameliorated
bw9Tg; kca3Tg + MO4-tnfa standard conditions Prim-5 ventral wall of dorsal aorta hematopoietic stem cell amount, ameliorated
kca3Tg; kca4Tg + MO2-tnfrsf1b standard conditions Prim-5 ventral wall of dorsal aorta runx1 expression decreased distribution, abnormal
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell decreased amount, abnormal
kca3Tg; kca4Tg + MO2-tnfrsf1b heat shock Prim-5 ventral wall of dorsal aorta hematopoietic stem cell amount, ameliorated
kca3Tg; kca4Tg + MO4-tnfa standard conditions Prim-5 ventral wall of dorsal aorta runx1 expression decreased distribution, abnormal
Prim-5 ventral wall of dorsal aorta hematopoietic stem cell decreased amount, abnormal
kca3Tg; kca4Tg + MO4-tnfa heat shock Prim-5 ventral wall of dorsal aorta hematopoietic stem cell amount, ameliorated
s896Tg; um14Tg + MO2-tnfrsf1b standard conditions Prim-5 ventral wall of dorsal aorta EGFP expression decreased distribution, abnormal
s896Tg; um14Tg + MO4-tnfa standard conditions Prim-5 ventral wall of dorsal aorta EGFP expression decreased distribution, abnormal
Acknowledgments:
ZFIN wishes to thank the journal Cell for permission to reproduce figures from this article. Please note that this material may be protected by copyright.

Reprinted from Cell, 159, Espín-Palazón, R., Stachura, D.L., Campbell, C.A., García-Moreno, D., Del Cid, N., Kim, A.D., Candel, S., Meseguer, J., Mulero, V., Traver, D., Proinflammatory signaling regulates hematopoietic stem cell emergence, 1070-85, Copyright (2014) with permission from Elsevier. Full text @ Cell