Phenotype
|
Fish
|
Conditions
|
Figures
|
whole organism decreased length, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
somite morphology, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
vasculature increased accumulation blood, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S6
from Hartnett et al., 2010
|
post-vent vasculature increased accumulation blood, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S6
from Hartnett et al., 2010
|
blood decreased fluid flow, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S6
from Hartnett et al., 2010
|
blood circulation disrupted, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 6 ,
Fig. S6
from Hartnett et al., 2010
|
heart increased size, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 6
from Hartnett et al., 2010
|
eye decreased size, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
spinal cord neural keel apoptotic, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S4
from Hartnett et al., 2010
|
brain structure, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
apoptotic process increased occurrence, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S4
from Hartnett et al., 2010
|
intermediate cell mass of mesoderm distended, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
dorsal/ventral pattern formation disrupted, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S5
from Hartnett et al., 2010
|
heart looping arrested, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 6
from Hartnett et al., 2010
|
bulbus arteriosus morphology, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 6
from Hartnett et al., 2010
|
trunk vasculature increased accumulation blood, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. S6
from Hartnett et al., 2010
|
whole organism wholly ventralized, abnormal
|
AB + MO1-igf2a
|
standard conditions
|
Fig. 1 ,
Fig. S5
from Hartnett et al., 2010
|
dorsal/ventral axis specification disrupted, abnormal
|
AB/TU + MO1-igf2a
|
standard conditions
|
Fig. 3
from White et al., 2009
|
notochord undulate, abnormal
|
AB/TU + MO1-igf2a
|
standard conditions
|
Fig. 3
from White et al., 2009
|
convergent extension involved in axis elongation delayed, abnormal
|
AB/TU + MO1-igf2a
|
standard conditions
|
Fig. 3
from White et al., 2009
|
whole organism curved ventral, abnormal
|
AB/TU + MO1-igf2a
|
standard conditions
|
Fig. 3
from White et al., 2009
|
notochord development delayed, abnormal
|
AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53
|
standard conditions
|
Fig. 4
from White et al., 2009
|
somite specification disrupted, abnormal
|
AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53
|
standard conditions
|
Fig. 4
from White et al., 2009
|
notochord undulate, abnormal
|
AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53
|
standard conditions
|
Fig. 4
from White et al., 2009
|
trunk has fewer parts of type somite, abnormal
|
AB/TU + MO1-igf2a + MO2-igf2a + MO4-tp53
|
standard conditions
|
Fig. 4
from White et al., 2009
|
angiogenesis disrupted, abnormal
|
y1Tg + MO1-igf2a
|
standard conditions
|
text only
from Hartnett et al., 2010
|
intersegmental vessel sprouting angiogenesis delayed, abnormal
|
y7Tg + MO1-igf2a + MO2-igf2a + MO4-tp53
|
standard conditions
|
Fig. 4
from Dallinga et al., 2018
|
intersegmental vessel filopodium absent, abnormal
|
y7Tg + MO1-igf2a + MO2-igf2a + MO4-tp53
|
standard conditions
|
Fig. 4
from Dallinga et al., 2018
|
blood circulation disrupted, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 4 ,
Fig. 6
from Hartnett et al., 2010
|
blood decreased fluid flow, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 4
from Hartnett et al., 2010
|
dorsal/ventral pattern formation disrupted, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 3
from Hartnett et al., 2010
|
heart looping arrested, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 6
from Hartnett et al., 2010
|
whole organism decreased length, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
brain structure, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
vasculature increased accumulation blood, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 4
from Hartnett et al., 2010
|
intermediate cell mass of mesoderm distended, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
somite morphology, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
post-vent vasculature increased accumulation blood, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 4
from Hartnett et al., 2010
|
apoptotic process increased occurrence, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 2
from Hartnett et al., 2010
|
eye decreased size, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 1
from Hartnett et al., 2010
|
bulbus arteriosus morphology, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 6
from Hartnett et al., 2010
|
trunk vasculature increased accumulation blood, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 4
from Hartnett et al., 2010
|
heart increased size, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 6
from Hartnett et al., 2010
|
whole organism wholly ventralized, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 1 ,
Fig. 3
from Hartnett et al., 2010
|
heart contraction decreased occurrence, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
text only
from Hartnett et al., 2010
|
neural keel apoptotic, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 2
from Hartnett et al., 2010
|
spinal cord neural keel apoptotic, abnormal
|
AB + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 2
from Hartnett et al., 2010
|
whole organism curved ventral, abnormal
|
AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53
|
standard conditions
|
text only
from White et al., 2009
|
somite specification disrupted, abnormal
|
AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53
|
standard conditions
|
Fig. 4
from White et al., 2009
|
notochord undulate, abnormal
|
AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53
|
standard conditions
|
text only
from White et al., 2009
|
trunk has fewer parts of type somite, abnormal
|
AB/TU + MO1-igf2a + MO1-igf2b + MO2-igf2a + MO2-igf2b + MO4-tp53
|
standard conditions
|
Fig. 4
from White et al., 2009
|
cranial blood vessel decreased size, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
angiogenesis disrupted, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5 ,
Fig. S7 ,
text only
from Hartnett et al., 2010
|
parachordal vessel hypoplastic, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
intersegmental vessel decreased size, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
parachordal vessel morphology, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
intermediate cell mass of mesoderm distended, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
intersegmental vessel disorganized, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
vasculature disorganized, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. S7
from Hartnett et al., 2010
|
ocular blood vessel decreased size, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|
sprouting angiogenesis decreased occurrence, abnormal
|
y1Tg + MO1-igf2a + MO3-igf2b
|
standard conditions
|
Fig. 5
from Hartnett et al., 2010
|