Fig. 4

Cheng et al., 2016 - Vinculin b deficiency causes epicardial hyperplasia and coronary vessel disorganization in zebrafish
Other Figures
All Figure Page
Back to All Figure Page
Fig. 4

Ventricular expression patterns of the NVclb-EGFP fusion protein. (A,B) GFP expression in the ventricle surface (0 μm) at 52 dpf, showing the formation of plexus-like structures in the v12 mutant (B) compared with heterozygotes (A). (C,D) GFP expression patterns in the sub-epicardium of ventricles at 4.6 μm (C) and 9.2 μm (D) from the surface. Strong expression of GFP fusion protein is shown in the mutant (D) compared with heterozygotes (C). (E,F) GFP is detected at the boundary between endothelial cells and muscles of ventricles at 9.8 μm (E) and 13.8 μm (F) from the surface. (G-H''') GFP is ubiquitously expressed in the ventricle. GFP expression is detected in several layers of GFP+/MF20- epicardium-derived cells in the mutant (H'',H''') compared with a single layer of epicardial cells in heterozygotes (G'',G'''). (I-J′) Tg(flk1:mcherry) transgenic reporter is used to label endothelial cells. GFP is detected at the boundary between endothelial cells (red) and myocardium. endo, endocardium; epi, epicardium; myo, myocardium. Scale bars: 10 μm in A-F,I-J'; 20 μm in G,G'',H,H''.

Expression Data
Anatomical Terms:
Stage: Long-pec

Expression Detail
Antibody Labeling
Phenotype Data
Observed In:
Stage: Long-pec

Phenotype Detail
This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image. Full text @ Development