Research
Search
Genes / Clones
Expression
Mutants / Tg
Antibodies
Anatomy / GO / Human Disease / Chemical
Publications
Data Mining
Downloads
Data Model
AllianceMine
BioMart
Genomics
BLAST
ZFIN
Ensembl
NCBI
UCSC
Genome Browsers
ZFIN
Ensembl
Vega
GRC
UCSC
NCBI
Resources
Zebrafish Genomics
Other Genome Databases
Resources
General
The Zebrafish Book
Protocol Wiki
Antibody Wiki
Anatomy Atlases
Resources for Students and Educators
Zebrafish Programs
ZF-Health
Husbandry Resources
More...
Resource Centers
Zebrafish International Resource Center (ZIRC)
China Zebrafish Resource Center (CZRC)
European Zebrafish Resource Center (EZRC)
Community
Announcements
News
Meetings
Jobs
Alliance Community Forum
Search
People
Labs
Companies
Societies
International Zebrafish Society (IZFS)
Zebrafish Disease Models Society (ZDMS)
Genetics Society of America (GSA)
Zebrafish Husbandry Association
Support
Nomenclature
Nomenclature Conventions
Line Designations
Wild-Type Lines
Submit a Proposed Gene Name
Submit a Proposed Mutant/Tg Line Name
Publications
Guidelines for Authors
Zebrashare
Citing ZFIN
Using ZFIN
Help & Tips
Glossary
Single Box Search Help
Submit Data
Terms of Use
About Us
About ZFIN
Contact Information
Statistics
Committees
Jobs at ZFIN
Sign In
Phenotype Figure Summary
Marker:
hoxb1a
Conditions:
Standard or Control
Publication
Data
Fish
Anatomy
Ghosh
et al.
, 2018
Fig. 1
hoxb1a
um191/um191
rhombomere 4
hoxb1a
expression decreased amount, abnormal
rhombomere 4 Mauthner neuron decreased amount, abnormal
Fig. 3
hoxb1a
um191/um191
rhombomere 1
gbx1
expression mislocalised, abnormal
rhombomere 2
gbx1
expression mislocalised, abnormal
rhombomere 3
gbx1
expression mislocalised, abnormal
rhombomere 4
gbx1
expression mislocalised, abnormal
Fig. 5
hoxb1a
um191/um191
rhombomere 4
gbx1
expression mislocalised, abnormal
Fig. 7
hoxb1a
um191/um191
rhombomere 4
hoxb1a
expression decreased amount, abnormal
rhombomere 4 Mauthner neuron decreased amount, abnormal
Fig. S2
hoxb1a
um191/um191
rhombomere 4
hoxb1a
expression decreased amount, abnormal
Selland
et al.
, 2018
Fig. 2
hoxb1a
sa1191/sa1191
rhombomere 4
hoxa2b
expression decreased amount, abnormal
rhombomere 4
hoxb1a
expression decreased amount, abnormal
rhombomere 4
hoxb2a
expression decreased amount, abnormal
rhombomere 5
hoxb2a
expression decreased amount, abnormal
Fig. 3
hoxb1a
sa1191/sa1191
; rw0Tg
, 
hoxb1a
sa1191/sa1191
Mauthner neuron ab1-neurofilament-m labeling absent, abnormal
Mauthner neuron absent, abnormal
motor neuron migration decreased occurrence, abnormal
motor neuron migration disrupted, abnormal
rhombomere 4 branchiomotor neuron mislocalised, abnormal
rhombomere 6 branchiomotor neuron absent, abnormal
Fig. 7
hoxb1a
sa1191/sa1191
presumptive rhombomere 3
cyp26b1
expression decreased amount, abnormal
Fig. 9
hoxb1a
sa1191/sa1191
midbrain hindbrain boundary neural keel
fgf8a
expression spatial pattern, abnormal
midbrain hindbrain boundary neural keel morphology, abnormal
presumptive rhombomere 2
fgf8a
expression spatial pattern, abnormal
Fig. S4
hoxb1a
sa1191/sa1191
rhombomere 4
fgf3
expression decreased amount, abnormal
Fig. S5
hoxb1a
sa1191/sa1191
midbrain hindbrain boundary neural keel
fgf8a
expression spatial pattern, abnormal
Weicksel
et al.
, 2014
Fig. 3
hoxb1a
um191/+
; hoxb1a
um192/+
facial nerve motor nucleus accumulation rhombomere 4, abnormal
facial nerve motor nucleus motor neuron migration decreased process quality, abnormal
Mauthner neuron absent, abnormal
rhombomere 4 has extra parts of type cranial motor neuron, abnormal
rhombomere 6 has fewer parts of type cranial motor neuron, abnormal
rhombomere 7 has fewer parts of type cranial motor neuron, abnormal
Hurley
et al.
, 2007
Fig. 5
rw0Tg + MO1-hoxb1a
cell migration in hindbrain disrupted, abnormal
facial nerve development disrupted, abnormal
hindbrain neuron mislocalised, abnormal
neuron migration disrupted, abnormal
Rohrschneider
et al.
, 2007
Fig. 3
WT + MO1-hoxb1a
rhombomere 4 development disrupted, abnormal
Fig. 4
WT + MO1-hoxb1a
rhombomere 4 development disrupted, abnormal
Your Input Welcome
Your Input Welcome
We welcome your input and comments. Please use this form to recommend updates to the information in ZFIN. We appreciate as much detail as possible and references as appropriate. We will review your comments promptly.
Please check the highlighted fields and try again.
Name:
Institution:
Email address:
Please leave blank:
Subject:
Comments:
Send your comments
Thank you for submitting comments. Your input has been emailed to ZFIN curators who may contact you if additional information is required.
Oops. Something went wrong. Please try again later.