Morpholino

MO3-ccn2a

ID

ZDB-MRPHLNO-150126-6

Name

MO3-ccn2a

Previous Names

MO3-ctgfa

Target

ccn2a (1)

Sequence

5' - GAGTCATTCCAGAAAACATGATGAC - 3'

Disclaimer

Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.

Note

Translation-blocking MO.

Genome Resources

None

Target Location

Genomic Features

No data available

Expression

Gene expression in Wild Types + MO3-ccn2a

Expressed Gene	Anatomy	Figures
coro1a	caudal hematopoietic tissue	Fig. S6 from Xue et al., 2019
gata1a	caudal hematopoietic tissue whole organism	Fig. S6 from Xue et al., 2019
mfap4.1	caudal hematopoietic tissue	Fig. S6 from Xue et al., 2019
myb	caudal hematopoietic tissue whole organism	Fig. S6 from Xue et al., 2019
runx1	ventral wall of dorsal aorta	Fig. S6 from Xue et al., 2019
spi1b	whole organism	Fig. S6 from Xue et al., 2019

Phenotype

Phenotype resulting from MO3-ccn2a

Phenotype	Fish	Figures
cardiac muscle progenitor cell migration to the midline involved in heart field formation decreased occurrence, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	Fig. 3 , Fig. 4 from Fukui et al., 2014
caudal hematopoietic tissue mfap4.1 expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue myb expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue coro1a expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue gata1a expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue GFP expression increased amount, abnormal	ioz1Tg; pku6Tg + MO3-ccn2a	Fig. 5 from Xue et al., 2019
caudal hematopoietic tissue GFP expression increased distribution, abnormal	ioz1Tg; pku6Tg + MO3-ccn2a	Fig. 5 from Xue et al., 2019
caudal hematopoietic tissue hematopoietic cell increased amount, abnormal	ioz1Tg; pku6Tg + MO3-ccn2a	Fig. 5 from Xue et al., 2019
endoderm anterior region apoptotic, abnormal	ha01Tg + MO3-ccn2a	Fig. 3 from Fukui et al., 2014
endoderm antero-medial region decreased object quality, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	Fig. 4 from Fukui et al., 2014
endoderm apoptotic process increased occurrence, abnormal	ha01Tg + MO3-ccn2a	Fig. 3 from Fukui et al., 2014
heart split bilaterally, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	Fig. 4 from Fukui et al., 2014
heart morphogenesis decreased process quality, abnormal	ha01Tg + MO3-ccn2a	Fig. 3 , Fig. 4 from Fukui et al., 2014
heart tube split bilaterally, abnormal	ha01Tg + MO3-ccn2a	Fig. 3 from Fukui et al., 2014
myocardial precursor mislocalised laterally, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	Fig. 4 from Fukui et al., 2014
whole organism spi1b expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019
whole organism myb expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019
whole organism gata1a expression increased amount, abnormal	TU + MO3-ccn2a	Fig. S6 from Xue et al., 2019

Phenotype of all Fish created by or utilizing MO3-ccn2a

Phenotype	Fish	Conditions	Figures
caudal hematopoietic tissue coro1a expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue mfap4.1 expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue gata1a expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
whole organism gata1a expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
whole organism myb expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
whole organism spi1b expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
caudal hematopoietic tissue myb expression increased amount, abnormal	TU + MO3-ccn2a	control	Fig. S6 from Xue et al., 2019
heart tube split bilaterally, abnormal	ha01Tg + MO3-ccn2a	standard conditions	Fig. 3 from Fukui et al., 2014
endoderm apoptotic process increased occurrence, abnormal	ha01Tg + MO3-ccn2a	standard conditions	Fig. 3 from Fukui et al., 2014
cardiac muscle progenitor cell migration to the midline involved in heart field formation decreased occurrence, abnormal	ha01Tg + MO3-ccn2a	standard conditions	Fig. 3 from Fukui et al., 2014
endoderm anterior region apoptotic, abnormal	ha01Tg + MO3-ccn2a	standard conditions	Fig. 3 from Fukui et al., 2014
heart morphogenesis decreased process quality, abnormal	ha01Tg + MO3-ccn2a	standard conditions	Fig. 3 from Fukui et al., 2014
heart split bilaterally, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
endoderm antero-medial region decreased object quality, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
heart morphogenesis decreased process quality, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
cardiac muscle progenitor cell migration to the midline involved in heart field formation decreased occurrence, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
myocardial precursor mislocalised laterally, abnormal	ha01Tg; ncv11Tg + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
caudal hematopoietic tissue GFP expression increased distribution, abnormal	ioz1Tg; pku6Tg + MO3-ccn2a	control	Fig. 5 from Xue et al., 2019
caudal hematopoietic tissue hematopoietic cell increased amount, abnormal	ioz1Tg; pku6Tg + MO3-ccn2a	control	Fig. 5 from Xue et al., 2019
caudal hematopoietic tissue GFP expression increased amount, abnormal	ioz1Tg; pku6Tg + MO3-ccn2a	control	Fig. 5 from Xue et al., 2019
caudal hematopoietic tissue myb expression amount, ameliorated	pd97Tg + MO3-ccn2a	heat shock	Fig. S6 from Xue et al., 2019
heart split bilaterally, abnormal	ha01Tg; ncv11Tg + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
myocardial precursor mislocalised laterally, abnormal	ha01Tg; ncv11Tg + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
heart morphogenesis decreased process quality, abnormal	ha01Tg; ncv11Tg + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
cardiac muscle progenitor cell migration to the midline involved in heart field formation decreased occurrence, abnormal	ha01Tg; ncv11Tg + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
endoderm antero-medial region decreased object quality, abnormal	ha01Tg; ncv11Tg + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 4 from Fukui et al., 2014
endoderm antero-medial region decreased object quality, abnormal	ha01Tg; ncv11Tg + MO1-gna13a + MO1-gna13b + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 2 from Fukui et al., 2014
heart morphogenesis decreased process quality, abnormal	ha01Tg; ncv11Tg + MO1-gna13a + MO1-gna13b + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 2 from Fukui et al., 2014
myocardial precursor mislocalised laterally, abnormal	ha01Tg; ncv11Tg + MO1-gna13a + MO1-gna13b + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 2 from Fukui et al., 2014
heart tube split bilaterally, abnormal	ha01Tg; ncv11Tg + MO1-gna13a + MO1-gna13b + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 2 from Fukui et al., 2014
cardiac muscle progenitor cell migration to the midline involved in heart field formation decreased occurrence, abnormal	ha01Tg; ncv11Tg + MO1-gna13a + MO1-gna13b + MO1-yap1 + MO3-ccn2a	standard conditions	Fig. 2 from Fukui et al., 2014

Citations