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Fargher et al., 2025 - A zebrafish model of acmsd deficiency does not support a prominent role for ACMSD in Parkinson's disease
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Fig. 8 acmsd-/- larvae demonstrate normal expression of three key proinflammatory mediators.

qPCR data suggested that genotype had no effect on the expression of cxcl8 (A, p = 0.9551, Mann Whitney test), il-1β (B, p = 0.7081, two-tailed unpaired t test), or mmp9 (C, p = 0.2200, two-tailed unpaired t test). n = 7 replicates for acmsd-/-; 8 replicates for acmsd+/+ (15 larvae per replicate).

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This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image. Full text @ NPJ Parkinsons Dis