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Gessler et al., 2022 - Control of Directed Cell Migration after Tubular Cell Injury by Nucleotide Signaling

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Figure 2

The involvement of adenosine deaminase family members in zebrafish pronephros repair. (A) In situ hybridization revealed upregulation of zebrafish ada. Depletion of zebrafish ada by a splice—(SBM) blocking morpholino oligonucleotides (MO) delayed the repair process. (B) In situ hybridization revealed upregulation of zebrafish ada2a. Depletion of zebrafish ada2a by a translation- (TBM) blocking morpholino oligonucleotides (MO) had no effect on the repair process. (C) In situ hybridization revealed upregulation of zebrafish ada2b. Depletion of zebrafish ada2b by a TBM delayed the repair process. (D) In situ hybridization revealed upregulation of zebrafish adal. Depletion of zebrafish adal by a TBM had no significant influence on the repair process (mean ± SEM; 2-way ANOVA).

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This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image. Full text @ Int. J. Mol. Sci.