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Fig. 6

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ZDB-FIG-250225-54

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Stooke-Vaughan et al., 2025 - The physical roles of different posterior tissues in zebrafish axis elongation

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Fig. 6

The role of different tissues in posterior body axis elongation.

Diagrams of posterior elongating tissues in wild-type, tbx16^−/−, noto^−/−, and vangl2^−/− mutants, illustrating the high-level tissue flows caused by cell movements. The near-complete failure to differentiate PSM in tbx16^−/− does not disrupt posterior body axis elongation, as the enlarged MPZ continues to move posteriorly. The loss of the notochord in noto^−/− reduces fluidity in the MPZ and vortices are lost from the MPZ/PSM boundary, but posterior body axis elongation is not disrupted. Disrupted planar polarity movements and dorsal cell movements in vangl2^−/− leads to greatly slowed posterior body axis elongation, with ventral tissue movements scaled to the altered proportions of the posterior tissues.

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This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image. Full text @ Nat. Commun.