Morpholino

MO2-cbfb

ID
ZDB-MRPHLNO-101021-5
Name
MO2-cbfb
Previous Names
None
Target
Sequence
5' - GTATTTAATCTGTAAACAGGGATGA - 3'
ZFIN BLAST NCBI BLAST Ensembl UCSC BLAT
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
Splice-blocking MO.
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO2-cbfb
Expressed Gene Anatomy Figures
nppa Fig. 5 from Meder et al., 2010
vegfaa Fig. 5 from Meder et al., 2010
Phenotype
Phenotype resulting from MO2-cbfb
Phenotype Fish Figures
atrium dilated, abnormal AB/TU + MO2-cbfb Fig. 3 from Meder et al., 2010
blood circulation disrupted, abnormal AB/TU + MO2-cbfb Fig. 3 from Meder et al., 2010
cardiac muscle cell has fewer parts of type cardiac muscle cell myosin filament, abnormal AB/TU + MO2-cbfb Fig. 4 from Meder et al., 2010
cardiac muscle cell intercalated disc malformed, abnormal AB/TU + MO2-cbfb Fig. 4 from Meder et al., 2010
cardiac muscle cell sarcomere decreased thickness, abnormal AB/TU + MO2-cbfb Fig. 4 from Meder et al., 2010
cardiac muscle cell Z disc malformed, abnormal AB/TU + MO2-cbfb Fig. 4 from Meder et al., 2010
cardiac ventricle dilated, abnormal AB/TU + MO2-cbfb Fig. 3 from Meder et al., 2010
heart non-functional, abnormal AB/TU + MO2-cbfb Fig. 3 from Meder et al., 2010
heart contraction disrupted, abnormal AB/TU + MO2-cbfb Fig. 3,  text only from Meder et al., 2010
locomotory behavior disrupted, abnormal AB/TU + MO2-cbfb text only from Meder et al., 2010
sarcomerogenesis disrupted, abnormal AB/TU + MO2-cbfb Fig. 4 from Meder et al., 2010
skeletal muscle decreased functionality, abnormal AB/TU + MO2-cbfb text only from Meder et al., 2010
skeletal muscle cell has fewer parts of type skeletal muscle cell myosin filament, abnormal AB/TU + MO2-cbfb Fig. 4 from Meder et al., 2010
Phenotype of all Fish created by or utilizing MO2-cbfb
Phenotype Fish Conditions Figures
skeletal muscle decreased functionality, abnormal AB/TU + MO2-cbfb standard conditions text only from Meder et al., 2010
blood circulation disrupted, abnormal AB/TU + MO2-cbfb standard conditions Fig. 3 from Meder et al., 2010
cardiac ventricle dilated, abnormal AB/TU + MO2-cbfb standard conditions Fig. 3 from Meder et al., 2010
locomotory behavior disrupted, abnormal AB/TU + MO2-cbfb standard conditions text only from Meder et al., 2010
cardiac muscle cell has fewer parts of type cardiac muscle cell myosin filament, abnormal AB/TU + MO2-cbfb standard conditions Fig. 4 from Meder et al., 2010
heart contraction disrupted, abnormal AB/TU + MO2-cbfb standard conditions Fig. 3,  text only from Meder et al., 2010
atrium dilated, abnormal AB/TU + MO2-cbfb standard conditions Fig. 3 from Meder et al., 2010
cardiac muscle cell Z disc malformed, abnormal AB/TU + MO2-cbfb standard conditions Fig. 4 from Meder et al., 2010
skeletal muscle cell has fewer parts of type skeletal muscle cell myosin filament, abnormal AB/TU + MO2-cbfb standard conditions Fig. 4 from Meder et al., 2010
cardiac muscle cell intercalated disc malformed, abnormal AB/TU + MO2-cbfb standard conditions Fig. 4 from Meder et al., 2010
sarcomerogenesis disrupted, abnormal AB/TU + MO2-cbfb standard conditions Fig. 4 from Meder et al., 2010
heart non-functional, abnormal AB/TU + MO2-cbfb standard conditions Fig. 3 from Meder et al., 2010
cardiac muscle cell sarcomere decreased thickness, abnormal AB/TU + MO2-cbfb standard conditions Fig. 4 from Meder et al., 2010
Citations
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