Phenotype
|
Fish
|
Conditions
|
Figures
|
pronephric proximal convoluted tubule slc20a1a expression increased distribution, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 7 S2
from Poureetezadi et al., 2016
Fig. 3
from Cheng et al., 2015
|
heart edematous, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2
from Cheng et al., 2015
|
pronephric distal early tubule mislocalised posteriorly, abnormal
|
TU + MO1-sim1a
|
chemical treatment: all-trans-retinoic acid
|
Fig. 6
from Cheng et al., 2015
|
pronephric proximal convoluted tubule slc20a1a expression mislocalised, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3
from Cheng et al., 2015
|
pronephric distal late tubule increased length, abnormal
|
TU + MO1-sim1a
|
chemical treatment: 4-(diethylamino)benzaldehyde
|
Fig. 7
from Cheng et al., 2015
|
pronephric proximal straight tubule decreased size, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3 ,
Fig. S6
from Cheng et al., 2015
|
pronephric distal late tubule slc12a3 expression decreased distribution, abnormal
|
TU + MO1-sim1a
|
control
|
Fig. 7 S2
from Poureetezadi et al., 2016
|
pronephric distal late tubule decreased length, abnormal
|
TU + MO1-sim1a
|
chemical treatment: all-trans-retinoic acid
|
Fig. 6
from Cheng et al., 2015
|
pronephric distal early tubule slc12a1 expression spatial pattern, ameliorated
|
TU + MO1-sim1a
|
chemical treatment by environment: 16,16-dimethylprostaglandin E2
|
Fig. 7 S2
from Poureetezadi et al., 2016
|
pronephric distal early tubule mislocalised anteriorly, abnormal
|
TU + MO1-sim1a
|
chemical treatment: 4-(diethylamino)benzaldehyde
|
Fig. 7
from Cheng et al., 2015
|
pronephric proximal straight tubule trpm7 expression absent, abnormal
|
TU + MO1-sim1a
|
chemical treatment by environment: 16,16-dimethylprostaglandin E2
|
Fig. 7
from Poureetezadi et al., 2016
|
pronephric proximal convoluted tubule mislocalised posteriorly, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3
from Cheng et al., 2015
|
pronephric proximal tubule morphogenesis process quality, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3
from Cheng et al., 2015
|
pronephros lacks all parts of type pronephric proximal straight tubule, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 6
from Cheng et al., 2015
|
pronephric proximal convoluted tubule distended, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3 ,
Fig. 6 ,
Fig. S6
from Cheng et al., 2015
|
pronephric nephron tubule development process quality, abnormal
|
TU + MO1-sim1a
|
chemical treatment: 4-(diethylamino)benzaldehyde
|
Fig. 7
from Cheng et al., 2015
|
renal filtration decreased process quality, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2
from Cheng et al., 2015
|
pronephric proximal convoluted tubule distended, abnormal
|
TU + MO1-sim1a
|
chemical treatment: all-trans-retinoic acid
|
Fig. 6
from Cheng et al., 2015
|
pronephros lacks all parts of type corpuscles of Stannius, abnormal
|
TU + MO1-sim1a
|
chemical treatment: all-trans-retinoic acid
|
Fig. 6
from Cheng et al., 2015
|
pronephric proximal straight tubule absent, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 7
from Poureetezadi et al., 2016
|
pronephros lacks all parts of type corpuscles of Stannius, abnormal
|
TU + MO1-sim1a
|
chemical treatment: 4-(diethylamino)benzaldehyde
|
Fig. 7
from Cheng et al., 2015
|
pronephros lacks all parts of type corpuscles of Stannius, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3 ,
Fig. 6 ,
Fig. 7 ,
Fig. S6
from Cheng et al., 2015
|
head apoptotic, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2
from Cheng et al., 2015
|
eye decreased size, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2
from Cheng et al., 2015
|
pronephric proximal straight tubule absent, abnormal
|
TU + MO1-sim1a
|
chemical treatment by environment: 16,16-dimethylprostaglandin E2
|
Fig. 7
from Poureetezadi et al., 2016
|
pronephric proximal convoluted tubule straight, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2
from Cheng et al., 2015
|
pronephric nephron tubule development process quality, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 6 ,
Fig. 7
from Cheng et al., 2015
|
head decreased size, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2
from Cheng et al., 2015
|
pronephric nephron tubule development process quality, abnormal
|
TU + MO1-sim1a
|
chemical treatment: all-trans-retinoic acid
|
Fig. 6
from Cheng et al., 2015
|
pronephros lacks all parts of type pronephric proximal straight tubule, abnormal
|
TU + MO1-sim1a
|
chemical treatment: all-trans-retinoic acid
|
Fig. 6
from Cheng et al., 2015
|
pronephric proximal tubule morphogenesis decreased process quality, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 2 ,
Fig. 3
from Cheng et al., 2015
|
corpuscles of Stannius stc1 expression absent, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 3
from Cheng et al., 2015
|
pronephric proximal straight tubule trpm7 expression absent, abnormal
|
TU + MO1-sim1a
|
standard conditions
|
Fig. 7
from Poureetezadi et al., 2016
Fig. 3
from Cheng et al., 2015
|
pronephric distal early tubule increased length, abnormal
|
TU + MO1-sim1a
|
chemical treatment: 4-(diethylamino)benzaldehyde
|
Fig. 7
from Cheng et al., 2015
|
pronephric distal late tubule slc12a3 expression decreased distribution, abnormal
|
TU + MO1-sim1a
|
chemical treatment by environment: 16,16-dimethylprostaglandin E2
|
Fig. 7 S2
from Poureetezadi et al., 2016
|
pronephric proximal convoluted tubule slc20a1a expression increased distribution, abnormal
|
TU + MO1-sim1a
|
chemical treatment by environment: 16,16-dimethylprostaglandin E2
|
Fig. 7 S2
from Poureetezadi et al., 2016
|
pronephros lacks all parts of type corpuscles of Stannius, abnormal
|
TU + MO1-sim1a + MO1-tp53
|
standard conditions
|
Fig. S6
from Cheng et al., 2015
|
pronephric proximal convoluted tubule distended, abnormal
|
TU + MO1-sim1a + MO1-tp53
|
standard conditions
|
Fig. S6
from Cheng et al., 2015
|
pronephric proximal tubule development decreased process quality, abnormal
|
TU + MO1-sim1a + MO1-tp53
|
standard conditions
|
Fig. S6
from Cheng et al., 2015
|
pronephros lacks all parts of type pronephric proximal straight tubule, abnormal
|
TU + MO1-sim1a + MO1-tp53
|
standard conditions
|
Fig. S6
from Cheng et al., 2015
|
embryo development delayed, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 6 ,
Fig. 7
from Eaton et al., 2006
|
preoptic area pou3f2a expression decreased amount, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 3
from Kasher et al., 2016
|
eye decreased size, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 5
from Eaton et al., 2008
Fig. 6 ,
Fig. 7
from Eaton et al., 2006
|
head decreased size, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 5
from Eaton et al., 2008
Fig. 6 ,
Fig. 7
from Eaton et al., 2006
|
neuroendocrine cell decreased amount, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 5
from Eaton et al., 2006
|
preoptic area pou3f2b expression decreased amount, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 3
from Kasher et al., 2016
|
preoptic area morphology, abnormal
|
WT + MO1-sim1a
|
standard conditions
|
Fig. 5
from Eaton et al., 2008
|