Morpholino

MO2-ntn1a

ID
ZDB-MRPHLNO-060810-1
Name
MO2-ntn1a
Previous Names
  • ntn1a SBMO
Target
Sequence
5' - ATGATGGACTTACCGACACATTCGT - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
None
Genome Resources
None
Target Location
Genome Build: GRCz11Chromosome: 6
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO2-ntn1a
Phenotype
Phenotype resulting from MO2-ntn1a
Phenotype Fish Figures
angiogenesis disrupted, abnormal y1Tg + MO2-ntn1a Fig. 3 from Wilson et al., 2006
aortic arch absent, abnormal s843Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
aortic arch hypoplastic, abnormal s843Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
aortic arch morphology, abnormal s843Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
heart jogging disrupted, abnormal WT + MO2-ntn1a Fig. 3 from Opitz et al., 2015
heart looping disrupted, abnormal WT + MO2-ntn1a Fig. 3 from Opitz et al., 2015
horizontal myoseptum lacks parts or has fewer parts of type motor neuron axon, abnormal ml2Tg + MO2-ntn1a Fig. 6 with image from Lim et al., 2011
horizontal myoseptum lacks parts or has fewer parts of type RoP motor neuron axon, abnormal ml2Tg + MO2-ntn1a Fig. 6 with image from Lim et al., 2011
hypobranchial artery dysplastic, abnormal s843Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
hypobranchial artery morphology, abnormal s843Tg; ulb1Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
lymph vessel development process quality, abnormal y1Tg/+ + MO2-ntn1a Fig. S1 with image from Lim et al., 2011
parachordal vessel incomplete structure, abnormal s843Tg + MO2-ntn1a Fig. S1 from Opitz et al., 2015
parachordal vessel morphology, abnormal s843Tg + MO2-ntn1a Fig. S1 from Opitz et al., 2015
pharyngeal vasculature morphology, abnormal s843Tg; ulb1Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
thoracic duct aplastic, abnormal y1Tg/+ + MO2-ntn1a Fig. S1 with image from Lim et al., 2011
thyroid follicle mislocalised, abnormal s843Tg; ulb1Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
thyroid follicle position, abnormal WT + MO2-ntn1a Fig. 3 from Opitz et al., 2015
thyroid follicle cell decreased amount, abnormal ulb1Tg + MO2-ntn1a Fig. 6 from Opitz et al., 2015
thyroid primordium distended, abnormal s843Tg; ulb1Tg + MO2-ntn1a Fig. 5 from Opitz et al., 2015
thyroid primordium mislocalised, abnormal s843Tg; ulb1Tg + MO2-ntn1a Fig. 4Fig. 5 from Opitz et al., 2015
thyroid primordium morphology, abnormal s843Tg; ulb1Tg + MO2-ntn1a Fig. 3Fig. 5 from Opitz et al., 2015
trunk vasculature lacks all parts of type parachordal vessel, abnormal y1Tg + MO2-ntn1a Fig. 3 from Wilson et al., 2006
VaP motor neuron axon increased branchiness, abnormal WT + MO2-ntn1a Fig. 2 with image from Hale et al., 2011
VaP motor neuron axon protruding into muscle pioneer, abnormal WT + MO2-ntn1a Fig. 2 with image from Hale et al., 2011
ventral aorta morphology, abnormal s843Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
ventral aorta separated from bulbus arteriosus, abnormal s843Tg + MO2-ntn1a Fig. 4 from Opitz et al., 2015
Phenotype of all Fish created by or utilizing MO2-ntn1a
Phenotype Fish Conditions Figures
heart jogging disrupted, abnormal WT + MO2-ntn1a standard conditions Fig. 3 from Opitz et al., 2015
heart looping disrupted, abnormal WT + MO2-ntn1a standard conditions Fig. 3 from Opitz et al., 2015
VaP motor neuron axon protruding into muscle pioneer, abnormal WT + MO2-ntn1a standard conditions Fig. 2 with image from Hale et al., 2011
VaP motor neuron axon increased branchiness, abnormal WT + MO2-ntn1a standard conditions Fig. 2 with image from Hale et al., 2011
thyroid follicle position, abnormal WT + MO2-ntn1a standard conditions Fig. 3 from Opitz et al., 2015
thyroid primordium morphology, abnormal WT + MO2-ntn1a standard conditions Fig. 3 from Opitz et al., 2015
lymph vessel development process quality, abnormal y1Tg/+ + MO2-ntn1a standard conditions Fig. S1 with image from Lim et al., 2011
thoracic duct aplastic, abnormal y1Tg/+ + MO2-ntn1a standard conditions Fig. S1 with image from Lim et al., 2011
horizontal myoseptum lacks parts or has fewer parts of type RoP motor neuron axon, abnormal ml2Tg + MO2-ntn1a standard conditions Fig. 6 with image from Lim et al., 2011
horizontal myoseptum lacks parts or has fewer parts of type motor neuron axon, abnormal ml2Tg + MO2-ntn1a standard conditions Fig. 6 with image from Lim et al., 2011
ventral aorta separated from bulbus arteriosus, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
parachordal vessel incomplete structure, abnormal s843Tg + MO2-ntn1a standard conditions Fig. S1 from Opitz et al., 2015
hypobranchial artery dysplastic, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
ventral aorta morphology, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
parachordal vessel morphology, abnormal s843Tg + MO2-ntn1a standard conditions Fig. S1 from Opitz et al., 2015
pharyngeal vasculature morphology, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
aortic arch hypoplastic, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
aortic arch absent, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
aortic arch morphology, abnormal s843Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
thyroid follicle cell decreased amount, abnormal ulb1Tg + MO2-ntn1a standard conditions Fig. 6 from Opitz et al., 2015
angiogenesis disrupted, abnormal y1Tg + MO2-ntn1a standard conditions Fig. 3 from Wilson et al., 2006
trunk vasculature lacks all parts of type parachordal vessel, abnormal y1Tg + MO2-ntn1a standard conditions Fig. 3 from Wilson et al., 2006
thyroid primordium mislocalised, abnormal s843Tg; ulb1Tg + MO2-ntn1a standard conditions Fig. 4Fig. 5 from Opitz et al., 2015
thyroid primordium morphology, abnormal s843Tg; ulb1Tg + MO2-ntn1a standard conditions Fig. 5 from Opitz et al., 2015
pharyngeal vasculature morphology, abnormal s843Tg; ulb1Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
thyroid primordium distended, abnormal s843Tg; ulb1Tg + MO2-ntn1a standard conditions Fig. 5 from Opitz et al., 2015
hypobranchial artery morphology, abnormal s843Tg; ulb1Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
thyroid follicle mislocalised, abnormal s843Tg; ulb1Tg + MO2-ntn1a standard conditions Fig. 4 from Opitz et al., 2015
VaP motor neuron axon protruding into horizontal myoseptum, abnormal ml2Tg + MO1-ntn2 + MO2-ntn1a standard conditions Fig. 9 with image from Hale et al., 2011
VaP motor neuron axon protruding into horizontal myoseptum, abnormal ml2Tg + MO2-ntn1a + MO3-ntn1b standard conditions Fig. 9 with image from Hale et al., 2011
dendrite development disrupted, abnormal rw0Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 4 from Suli et al., 2006
efferent neuron dendrite mislocalised, abnormal rw0Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 4 from Suli et al., 2006
efferent neuron dendrite decreased amount, abnormal rw0Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 4 from Suli et al., 2006
olfactory receptor cell axon attached to anteromedial zone olfactory bulb, abnormal p201Tg; p202Tg + MO2-ntn1a standard conditions Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon attached to ventral zone olfactory bulb, abnormal p201Tg; p202Tg + MO2-ntn1a standard conditions Fig. 8 from Lakhina et al., 2012
olfactory bulb axon guidance disrupted, abnormal p201Tg; p202Tg + MO2-ntn1a standard conditions Fig. 5Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon position, abnormal p201Tg; p202Tg + MO2-ntn1a standard conditions Fig. 5Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon posterior to olfactory bulb, abnormal p201Tg; p202Tg + MO2-ntn1a standard conditions Fig. 8 from Lakhina et al., 2012
branching involved in lymph vessel morphogenesis process quality, abnormal plcg1y10/+; y1Tg/+ + MO2-ntn1a standard conditions Fig. 1 with image from Lim et al., 2011
parachordal vessel immature, abnormal plcg1y10/+; y1Tg/+ + MO2-ntn1a standard conditions Fig. 1 with image from Lim et al., 2011
parachordal vessel venous endothelial cell migration involved in lymph vessel development arrested, abnormal plcg1y10/+; y1Tg/+ + MO2-ntn1a standard conditions Fig. 1 with image from Lim et al., 2011
olfactory bulb axon guidance disrupted, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 5Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon attached to medial protoglomerulus, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 5 from Lakhina et al., 2012
olfactory receptor cell axon position, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 5Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon posterior to olfactory bulb, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon attached to dorsal zone olfactory bulb, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 5 from Lakhina et al., 2012
olfactory receptor cell axon attached to anteromedial zone olfactory bulb, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 8 from Lakhina et al., 2012
olfactory receptor cell axon attached to ventral zone olfactory bulb, abnormal p201Tg; p202Tg + MO1-ntn1b + MO2-ntn1a standard conditions Fig. 8 from Lakhina et al., 2012
Citations
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