Phenotype
|
Fish
|
Conditions
|
Figures
|
inner ear epithelial cell aggregated, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 6
from Croushore et al., 2005
|
pillar of the semicircular canal unfused from pillar of the semicircular canal, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 6
from Croushore et al., 2005
|
head domed, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
notochord undulate, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
post-vent region curved ventral, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
lipid phosphatase activity disrupted, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 8
from Croushore et al., 2005
|
intersegmental vessel shape, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
intersegmental vessel malformed, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
pericardium edematous, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
whole organism anterior-posterior axis decreased length, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
inner ear epithelial cell disorganized, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 6
from Croushore et al., 2005
|
otolith decreased amount, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 6
from Croushore et al., 2005
|
semicircular canal formation disrupted, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 6
from Croushore et al., 2005
|
phosphatidylinositol dephosphorylation decreased occurrence, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 8
from Croushore et al., 2005
|
blood decreased fluid flow, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
head mislocalised ventrally, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
eye decreased size, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 5
from Croushore et al., 2005
|
otolith shape, abnormal
|
WT + MO1-ptena
|
standard conditions
|
Fig. 6
from Croushore et al., 2005
|
spinal cord oligodendrocyte ab1-rps6 labeling increased amount, abnormal
|
vu12Tg + MO1-ptena + MO4-tp53
|
standard conditions
|
Fig. 3
from Mathews et al., 2016
|
oligodendrocyte TOR signaling increased occurrence, abnormal
|
vu12Tg + MO1-ptena + MO4-tp53
|
standard conditions
|
Fig. 3
from Mathews et al., 2016
|
oligodendrocyte myelin sheath length, ameliorated
|
co18Tg; co19Tg + MO1-ptena + MO4-tp53
|
chemical treatment by environment: U0126
|
Fig. 7
from Mathews et al., 2016
|
oligodendrocyte myelin sheath decreased length, ameliorated
|
co18Tg; co19Tg + MO1-ptena + MO4-tp53
|
chemical treatment by environment: sirolimus
|
Fig. 7
from Mathews et al., 2016
|
oligodendrocyte myelin sheath increased length, abnormal
|
co18Tg; co19Tg + MO1-ptena + MO4-tp53
|
control
|
Fig. 7
from Mathews et al., 2016
|
hindbrain oligodendrocyte development process quality, ameliorated
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
control
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte development decreased process quality, abnormal
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
chemical treatment by environment: sirolimus
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte mbpa expression amount, ameliorated
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
control
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte mbpa expression decreased amount, abnormal
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
chemical treatment by environment: sirolimus
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte plp1a expression amount, ameliorated
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
chemical treatment by environment: U0126
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte plp1a expression decreased amount, abnormal
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
chemical treatment by environment: sirolimus
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte plp1a expression amount, ameliorated
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
control
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte development process quality, ameliorated
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
chemical treatment by environment: U0126
|
Fig. 4
from Mathews et al., 2016
|
hindbrain oligodendrocyte mbpa expression amount, ameliorated
|
hmgcs1vu57/vu57 + MO1-ptena + MO4-tp53
|
chemical treatment by environment: U0126
|
Fig. 4
from Mathews et al., 2016
|
oligodendrocyte TOR signaling occurrence, ameliorated
|
hmgcs1vu57/vu57; vu12Tg + MO1-ptena + MO4-tp53
|
standard conditions
|
Fig. 3
from Mathews et al., 2016
|
spinal cord oligodendrocyte ab1-rps6 labeling amount, ameliorated
|
hmgcs1vu57/vu57; vu12Tg + MO1-ptena + MO4-tp53
|
standard conditions
|
Fig. 3
from Mathews et al., 2016
|
spinal cord oligodendrocyte ab20-mapk labeling increased amount, abnormal
|
hmgcs1vu57/vu57; vu12Tg + MO1-ptena + MO4-tp53
|
standard conditions
|
Fig. 3
from Mathews et al., 2016
|
oligodendrocyte MAPK cascade decreased occurrence, abnormal
|
hmgcs1vu57/vu57; vu12Tg + MO1-ptena + MO4-tp53
|
standard conditions
|
Fig. 3
from Mathews et al., 2016
|
spinal cord central nervous system myelination occurrence, ameliorated
|
hmgcs1vu57/vu57; vu16Tg + MO1-ptena + MO4-tp53
|
chemical treatment by environment: U0126
|
Fig. 6
from Mathews et al., 2016
|
spinal cord central nervous system myelination occurrence, ameliorated
|
hmgcs1vu57/vu57; vu16Tg + MO1-ptena + MO4-tp53
|
control
|
Fig. 5,
Fig. 6
from Mathews et al., 2016
|
spinal cord central nervous system myelination occurrence, ameliorated
|
hmgcs1vu57/vu57; vu16Tg + MO1-ptena + MO4-tp53
|
chemical treatment by environment: sirolimus
|
Fig. 6
from Mathews et al., 2016
|