Image
|
Figure Caption
Fig. S2 Endothelial fate specification appears unaffected in mutants with defective lumen formation. (A–H) In situ hybridization with the arterial specific marker ephrinB2a (A to D) and the venous specific marker flt4 (E to H) in 36 hpf logs231 mutant embryos (B and F) and wild-type siblings (A and E), and in 36 hpf mtss233 mutant embryos (D and H) and wild-type siblings (C and G). Black arrows point to the vascular expression of ephrinB2a and flt4. Although these mutants display defective vascular lumen formation, the specification of endothelial cells appears relatively unaffected.
Figure Data
Acknowledgments
This image is the copyrighted work of the attributed author or publisher, and
ZFIN has permission only to display this image to its users.
Additional permissions should be obtained from the applicable author or publisher of the image.
Reprinted from Developmental Biology, 307(1), Jin, S.W., Herzog, W., Santoro, M.M., Mitchell, T.S., Frantsve, J., Jungblut, B., Beis, D., Scott, I.C., D'Amico, L.A., Ober, E.A., Verkade, H., Field, H.A., Chi, N.C., Wehman, A.M., Baier, H., and Stainier, D.Y., A transgene-assisted genetic screen identifies essential regulators of vascular development in vertebrate embryos, 29-42, Copyright (2007) with permission from Elsevier. Full text @ Dev. Biol.