Fig. 2 The mibta52b and mibm132 antimorphic phenotypes are caused by Mibta52b and Mibm132 proteins, respectively. (A–C) Control (or mib-m2-injected) wt (or mibta52b) embryos probed with (A) deltaC at 10ss, dorsal view; (B) huC at 10ss, dorsal view, and (C) her4 at 22hpf, (a–d) head region, dorsal view and (a2–d2) trunk region, lateral view. (D–F) 3dpf tail pigment phenotype in panel D (a) control mibta52b or (b, c) mib-m2-injected mibta52b embryos, (E) mib-m1-injected mibta52b embryos and (F) (a) control mibm132 or (b) mib-m2-injected mibm132 embryos. All are lateral views. (G) her4 expression at 22hpf in control or mib-m2-injected mibm132 embryos, (a–b) head region, dorsal view and (a′–b′) trunk region, lateral view. All are head to the left.
Reprinted from Developmental Biology, 305(1), Zhang, C., Li, Q., Lim, C.H., Qiu, X., and Jiang, Y.J., The characterization of zebrafish antimorphic mib alleles reveals that Mib and Mind bomb-2 (Mib2) function redundantly, 14-27, Copyright (2007) with permission from Elsevier. Full text @ Dev. Biol.