FIGURE

Fig. 3

ID

ZDB-FIG-210205-20

Publication

Zhang et al., 2020 - Genetic evidence for Amh modulation of gonadotropin actions to control gonadal homeostasis and gametogenesis in zebrafish and its noncanonical signalling through Bmpr2a receptor

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Fig. 3

Gonadal hypertrophy in bmpr2a mutant. (A) Ovaries of different genotypes at 5 mpf. The bmpr2a mutation (bmpr2a−/−) caused significant enlargement of the ovary, which contained a large number of PG follicles, with only a few entering PV and vitellogenic growth. (B) Testes of different genotypes at 5 mpf. The bmpr2a mutation also induced hypertrophic growth of the testis, which contained abundant spermatogonia with limited meiosis. (C) Body length, body weight and GSI of different genotypes (n=5; *P<0.05, **P<0.01; two-tailed Student's t-test for unpaired data). (D) Sex ratios in different genotypes. SC, spermatocytes; SG, spermatogonia; ST, spermatids; SZ, spermatozoa. Arrows indicate ovaries or testes.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	bmpr2a^umo25/umo25 (AB) bmpr2a^umo25/+ (AB)
Observed In:	female organism gonad male organism ovarian follicle stage I ovary ovary cell population proliferation ovary gamete generation spermatogonium testis testis cell population proliferation testis gamete generation testis meiosis I testis spermatid differentiation whole organism
Stage:	Adult

Phenotype Detail

Acknowledgments

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