FIGURE

Fig. 4

ID

ZDB-FIG-160218-17

Publication

Jahangiri et al., 2016 - The AP-1 transcription factor component Fosl2 potentiates the rate of myocardial differentiation from the zebrafish second heart field

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Fig. 4

fosl2 mutants recover from their ventricular deficit. (A,B) Confocal images of hearts from 72hpf control sibling (CTRL; A; n=12) and fosl2 (B; n=5) mutant Tg(cmlc2:DsRed2-nuc) embryos. (C) Bar graph showing the mean numbers of total, atrial and ventricular cardiomyocytes in control, fosl2^fb15-/- and fosl2^fb16-/- (n=3) embryos. Error bars represent s.d. n.s., not significant. (D,E) Confocal images of OFT regions in control (D; n=15) and fosl2 mutant (E; n=4) embryos co-stained with antibodies recognizing striated muscle (MF20, red) or OFT smooth muscle (Elastin2, ELN2, green). Scale bars: 50µm.

Expression Data

Gene:	DsRed2
Antibodies:	Ab-MF20 Ab1-elnb
Fish:	WT fosl2^fb15/fb15 f2Tg fosl2^fb15/fb15; f2Tg
Anatomical Terms:	bulbus arteriosus smooth muscle cardiac muscle cell cardiac muscle cell nucleus striated muscle cell
Stage:	Protruding-mouth

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	fosl2^fb15/fb15; f2Tg
Observed In:	atrium cardiac muscle cell cardiac ventricle cardiac muscle cell
Stage:	Protruding-mouth

Phenotype Detail

Acknowledgments

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