Fig. 4
- ID
- ZDB-FIG-160218-17
- Publication
- Jahangiri et al., 2016 - The AP-1 transcription factor component Fosl2 potentiates the rate of myocardial differentiation from the zebrafish second heart field
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fosl2 mutants recover from their ventricular deficit. (A,B) Confocal images of hearts from 72hpf control sibling (CTRL; A; n=12) and fosl2 (B; n=5) mutant Tg(cmlc2:DsRed2-nuc) embryos. (C) Bar graph showing the mean numbers of total, atrial and ventricular cardiomyocytes in control, fosl2fb15-/- and fosl2fb16-/- (n=3) embryos. Error bars represent s.d. n.s., not significant. (D,E) Confocal images of OFT regions in control (D; n=15) and fosl2 mutant (E; n=4) embryos co-stained with antibodies recognizing striated muscle (MF20, red) or OFT smooth muscle (Elastin2, ELN2, green). Scale bars: 50µm. |
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Stage: | Protruding-mouth |
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Stage: | Protruding-mouth |