ZFIN ID: ZDB-FIG-130710-9
Rodríguez-Aznar et al., 2013 - Scratch2 Prevents Cell Cycle Re-Entry by Repressing miR-25 in Postmitotic Primary Neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience   33(12):5095-5105 Full text @ J. Neurosci.
ADDITIONAL FIGURES
EXPRESSION / LABELING:
Genes:
Antibody:
Fish:
Condition:
Anatomical Terms:
Stage: Prim-5
PHENOTYPE:
Fish:
Condition:
Observed In:
Stage: Prim-5

Fig. 6 ZFIN is incorporating published figure images and captions as part of an ongoing project. Figures from some publications have not yet been curated, or are not available for display because of copyright restrictions.

Gene Expression Details
Gene Antibody Fish Conditions Stage Anatomy Assay
EGFP in1Tg; knu3Tg chemical treatment: messenger RNA Prim-5 spinal cord neuron IFL
knu3Tg control Prim-5 spinal cord neuron IHC
knu3Tg chemical treatment: messenger RNA Prim-5 spinal cord neuron IHC
mRFP1 in1Tg; knu3Tg chemical treatment: messenger RNA Prim-5 neuron nucleus IFL
Prim-5 spinal cord neuron IFL
Antibody Labeling Details
Antibody Assay Fish Conditions Stage Anatomy
Ab1-elavl IHC WT control Prim-5 spinal cord neuron
IHC WT chemical treatment: messenger RNA Prim-5 spinal cord neuron
Phenotype Details
Fish Conditions Stage Phenotype
WT chemical treatment: messenger RNA Prim-5 primary neuron proliferative, abnormal
Prim-5 proliferative region mislocalised, abnormal
Prim-5 re-entry into mitotic cell cycle increased occurrence, abnormal
Prim-5 regulation of cyclin-dependent protein serine/threonine kinase activity decreased process quality, abnormal
Prim-5 regulation of neural precursor cell proliferation decreased process quality, abnormal
in1Tg; knu3Tg chemical treatment: messenger RNA Prim-5 primary neuron proliferative, abnormal
Prim-5 proliferative region mislocalised, abnormal
Prim-5 re-entry into mitotic cell cycle increased occurrence, abnormal
Prim-5 regulation of neural precursor cell proliferation decreased process quality, abnormal
knu3Tg chemical treatment: messenger RNA Prim-5 primary neuron proliferative, abnormal
Prim-5 proliferative region mislocalised, abnormal
Prim-5 re-entry into mitotic cell cycle increased occurrence, abnormal
Prim-5 regulation of neural precursor cell proliferation decreased process quality, abnormal
Full text @ J. Neurosci.