FIGURE
            Fig. S2
- ID
 - ZDB-FIG-130411-23
 - Publication
 - Ju et al., 2009 - Co-activation of hedgehog and AKT pathways promote tumorigenesis in zebrafish
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 - All Figure Page
 - Back to All Figure Page
 
                
                    
                        Fig. S2
                    
                    
                
                
            
        
        
    
        
            
            
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 Overall strategy for co-expression of oncogenes in zebrafish. Stable transgenic lines expressing zebrafish Smoa1 were generated using a Tol2-based vector (A). Constitutively active human AKT1 (myrhAKT1) was incorporated into a meganuclease-based vector (B). The zebrafish krt4 promoter could simultaneously activate smoa1-EGFP and myrhAKT1 expression through Gal4VP16-UAS.  | 
    
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ Mol. Cancer