FIGURE

Fig. S4

ID

ZDB-FIG-110405-28

Publication

Cui et al., 2011 - Disruption of planar cell polarity activity leads to developmental biliary defects

Other Figures

All Figure Page

Back to All Figure Page

Fig. S4

Marginally abnormal exocrine pancreas localization in pk1a morphants. Whole-mount in situ hybridization of trypsin (try), an exocrine pancreas marker, in 3 dpf control (A) and pk1a morphants demonstrates abnormal exocrine pancreas location, including “bilateral” (B), “midline” (C) and “left” (D). (E) Graph depicting the scoring of pk1a morphants demonstrates a slight difference in localization of the exocrine pancreas (p=NS by chi-square test).

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	WT + MO4-prickle1a
Knockdown Reagent:	MO4-prickle1a
Observed In:	exocrine pancreas
Stage:	Protruding-mouth

Phenotype Detail

Acknowledgments

This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image.

Reprinted from Developmental Biology, 351(2), Cui, S., Capecci, L.M., and Matthews, R.P., Disruption of planar cell polarity activity leads to developmental biliary defects, 229-241, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.