FIGURE

Fig. S3

ID

ZDB-FIG-110405-27

Publication

Cui et al., 2011 - Disruption of planar cell polarity activity leads to developmental biliary defects

Other Figures

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Fig. S3

Abnormal intestine localization in pk1a morphants. Whole-mount in situ hybridization of fatty acid binding protein 2 (fabp2), an intestinal marker, in 3 dpf control (A) and pk1a morphants demonstrates abnormal intestine location, including “bilateral” (B), and “right” (C). (D) Graph depicting the scoring of pk1a morphants demonstrates a significant difference in the number of abnormally localized intestines (0% vs. 33%, p<0.0001 by chi-square test). Note that the fabp2 probe also faintly stains the liver.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	WT + MO4-prickle1a
Knockdown Reagent:	MO4-prickle1a
Observed In:	intestine
Stage:	Protruding-mouth

Phenotype Detail

Acknowledgments

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Reprinted from Developmental Biology, 351(2), Cui, S., Capecci, L.M., and Matthews, R.P., Disruption of planar cell polarity activity leads to developmental biliary defects, 229-241, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.