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Fig. S4 Marginally abnormal exocrine pancreas localization in pk1a morphants. Whole-mount in situ hybridization of trypsin (try), an exocrine pancreas marker, in 3 dpf control (A) and pk1a morphants demonstrates abnormal exocrine pancreas location, including “bilateral” (B), “midline” (C) and “left” (D). (E) Graph depicting the scoring of pk1a morphants demonstrates a slight difference in localization of the exocrine pancreas (p=NS by chi-square test).
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Reprinted from Developmental Biology, 351(2), Cui, S., Capecci, L.M., and Matthews, R.P., Disruption of planar cell polarity activity leads to developmental biliary defects, 229-241, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.