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ZFIN ID:
ZDB-PERS-031009-2
Sarras, Michael P., Jr.
URL:
Affiliation:
Address:
Department of Anatomy and Cell Biology University of Kansas Medical Center 3901 Rainbow Boulevard Kansas City, KS 66160 USA
Country:
Phone:
(913) 588-2730
Fax:
(913) 588-2710
ORCID ID:
BIOGRAPHY AND RESEARCH INTERESTS
PUBLICATIONS
Leontovich, A.A., Intine, R.V., Sarras, M.P. (2016) Epigenetic Studies Point to DNA Replication/Repair Genes as a Basis for the Heritable Nature of Long Term Complications in Diabetes. Journal of Diabetes Research. 2016:2860780
Sarras, M.P., Leontovich, A.A., Intine, R.V. (2015) Use of zebrafish as a model to investigate the role of epigenetics in propagating the secondary complications observed in diabetes mellitus. Comparative biochemistry and physiology. Toxicology & pharmacology : CBP. 178:3-7
Dhliwayo, N., Sarras, M.P., Luczkowski, E., Mason, S., Intine, R.V. (2014) Parp inhibition prevents ten eleven translocase enzyme activation and hyperglycemia induced DNA demethylation. Diabetes. 63(9):3069-76
Pisano, G.C., Mason, S.M., Dhliwayo, N., Intine, R.V., Sarras, M.P. (2014) An assay for lateral line regeneration in adult zebrafish. Journal of visualized experiments : JoVE. (86)
Sarras, M.P., Leontovich, A.A., Olsen, A.S., and Intine, R.V. (2013) Impaired tissue regeneration corresponds with altered expression of developmental genes that persists in the metabolic memory state of diabetic zebrafish. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society. 21(2):320-328
Intine, R.V., Olsen, A.S., and Sarras, M.P. (2013) A zebrafish model of diabetes mellitus and metabolic memory. Journal of visualized experiments : JoVE. (72):e50232
Olsen, A.S., Sarras, M.P., Leontovich, A., and Intine, R.V. (2012) Heritable Transmission of Diabetic Metabolic Memory in Zebrafish Correlates With DNA Hypomethylation and Aberrant Gene Expression. Diabetes. 61(2):485-491
Olsen, A.S., Sarras, M.P. Jr, and Intine, R.V. (2010) Limb regeneration is impaired in an adult zebrafish model of diabetes mellitus. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society. 18(5):532-542
Thummel, R., Ju, M., Sarras, M.P. Jr., and Godwin, A.R. (2007) Both Hoxc13 orthologs are functionally important for zebrafish tail fin regeneration. Development genes and evolution. 217(6):413-420
Thummel, R., Bai, S., Sarras, M.P. Jr, Song, P., McDermott, J., Brewer, J., Perry, M., Zhang, X., Hyde, D.R., and Godwin, A.R. (2006) Inhibition of zebrafish fin regeneration using in vivo electroporation of morpholinos against fgfr1 and msxb. Developmental Dynamics : an official publication of the American Association of Anatomists. 235(2):336-346
Bai, S., Thummel, R., Godwin, A.R., Nagase, H., Itoh, Y., Li, L., Evans, R., McDermott, J., Seiki, M., and Sarras, M.P. Jr. (2005) Matrix metalloproteinase expression and function during fin regeneration in zebrafish: Analysis of MT1-MMP, MMP2 and TIMP2. Matrix biology : journal of the International Society for Matrix Biology. 24(4):247-260
Thummel, R., Li, L., Tanase, C., Sarras, M.P. Jr., and Godwin, A.R. (2004) Differences in expression pattern and function between zebrafish hoxc13 orthologs: recruitment of Hoxc13b into an early embryonic role. Developmental Biology. 274(2):318-333
Zhang, J., Bai, S., Zhang, X., Nagase, H., and Sarras, M.P. (2003) The expression of novel membrane-type matrix metalloproteinase isoforms is required for normal development of zebrafish embryos. Matrix biology : journal of the International Society for Matrix Biology. 22(3):279-293
NON-ZEBRAFISH PUBLICATIONS
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