ZFIN ID: ZDB-MRPHLNO-121026-2
Morpholino Name: MO2-s1pr1
Target: s1pr1 (1)
Sequence:
5' - AGTGTCTGGCGATTAGGTCATCCAT - 3'
   
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  (Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.)
Note: Translation-blocking MO.
TARGET LOCATIONS
View all 3 target locations
No data available
CONSTRUCTS WITH SEQUENCES FROM MO2-s1pr1 No data available
GENE EXPRESSION
Gene expression in Wild Types + MO2-s1pr1 No data available
PHENOTYPE
Phenotype resulting from MO2-s1pr1
Phenotype Figures
blood vessel development disrupted, abnormal Fig. 5 with imageFig. S2 with image from Ben Shoham et al., 2012
caudal vein plexus decreased area, abnormal Fig. 6 from Mendelson et al., 2013
caudal vein plexus decreased length, abnormal Fig. 6 from Mendelson et al., 2013
caudal vein plexus decreased width, abnormal Fig. 6 from Mendelson et al., 2013
caudal vein plexus morphology, abnormal Fig. 6 with imageFig. 9 with image from Ben Shoham et al., 2012
caudal vein plexus endothelial cell has extra parts of type endothelial cell filopodium, abnormal Fig. 7 with image from Ben Shoham et al., 2012
dorsal aorta endothelial cell decreased amount, abnormal Fig. 5 from Mendelson et al., 2013
dorsal longitudinal anastomotic vessel disorganized, abnormal Fig. 5Fig. S1 from Mendelson et al., 2013
dorsal longitudinal anastomotic vessel endothelial cell decreased amount, abnormal Fig. 5 from Mendelson et al., 2013
endothelial cell migration delayed, abnormal Fig. 5 with image from Ben Shoham et al., 2012
filopodium assembly increased occurrence, abnormal Fig. 7 with image from Ben Shoham et al., 2012
heart physical object quality, abnormal Fig. 5 with image from Ben Shoham et al., 2012
heart looping disrupted, abnormal Fig. 5 from Mendelson et al., 2013
hindbrain malformed, abnormal Fig. 5 with image from Ben Shoham et al., 2012
intersegmental vessel decreased length, abnormal Fig. 5 with image from Ben Shoham et al., 2012
intersegmental vessel shortened, abnormal Fig. 5Fig. S1 from Mendelson et al., 2013
intersegmental vessel endothelial cell decreased amount, abnormal Fig. 5 from Mendelson et al., 2013
nucleate erythrocyte increased accumulation blood island, abnormal Fig. 5 from Mendelson et al., 2013
pericardium edematous, abnormal Fig. 5 from Mendelson et al., 2013
Fig. 5 with image from Ben Shoham et al., 2012
posterior cardinal vein endothelial cell decreased amount, abnormal Fig. 5 from Mendelson et al., 2013
regulation of blood vessel remodeling process quality, abnormal Fig. 6 with imageFig. 7 with image from Ben Shoham et al., 2012
retina filopodium increased amount, abnormal Fig. 6 from Mendelson et al., 2013
sprouting angiogenesis increased occurrence, abnormal Fig. S2 with image from Ben Shoham et al., 2012
sprouting angiogenesis process quality, abnormal Fig. 7 with image from Ben Shoham et al., 2012
subintestinal vein deformed, abnormal Fig. S1 from Mendelson et al., 2013
subintestinal vein dilated, abnormal Fig. S2 with image from Ben Shoham et al., 2012
subintestinal vein has extra parts of type angiogenic sprout, abnormal Fig. S2 with image from Ben Shoham et al., 2012
subintestinal vein morphology, abnormal Fig. 9 with image from Ben Shoham et al., 2012

Phenotype of all Fish created by or utilizing MO2-s1pr1
Phenotype Fish Conditions Figures
pericardium edematous, abnormal AB/TU + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
heart looping disrupted, abnormal AB/TU + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
nucleate erythrocyte increased accumulation blood island, abnormal sd2Tg + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
subintestinal vein deformed, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. S1 from Mendelson et al., 2013
hindbrain malformed, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5 with image from Ben Shoham et al., 2012
subintestinal vein dilated, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. S2 with image from Ben Shoham et al., 2012
dorsal longitudinal anastomotic vessel disorganized, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5Fig. S1 from Mendelson et al., 2013
caudal vein plexus decreased area, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 6 from Mendelson et al., 2013
pericardium edematous, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5 with image from Ben Shoham et al., 2012
intersegmental vessel shortened, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5Fig. S1 from Mendelson et al., 2013
subintestinal vein morphology, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 9 with image from Ben Shoham et al., 2012
retina filopodium increased amount, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 6 from Mendelson et al., 2013
intersegmental vessel decreased length, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5 with image from Ben Shoham et al., 2012
sprouting angiogenesis increased occurrence, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. S2 with image from Ben Shoham et al., 2012
caudal vein plexus decreased width, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 6 from Mendelson et al., 2013
endothelial cell migration delayed, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5 with image from Ben Shoham et al., 2012
blood vessel development disrupted, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5 with imageFig. S2 with image from Ben Shoham et al., 2012
caudal vein plexus morphology, abnormal y1Tg + MO2-s1pr1 chemical treatment: semaxanib Fig. 9 with image from Ben Shoham et al., 2012
sprouting angiogenesis process quality, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 7 with image from Ben Shoham et al., 2012
caudal vein plexus endothelial cell has extra parts of type endothelial cell filopodium, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 7 with image from Ben Shoham et al., 2012
caudal vein plexus morphology, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 6 with imageFig. 9 with image from Ben Shoham et al., 2012
filopodium assembly increased occurrence, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 7 with image from Ben Shoham et al., 2012
regulation of blood vessel remodeling process quality, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 6 with imageFig. 7 with image from Ben Shoham et al., 2012
heart physical object quality, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 5 with image from Ben Shoham et al., 2012
subintestinal vein morphology, abnormal y1Tg + MO2-s1pr1 chemical treatment: semaxanib Fig. 9 with image from Ben Shoham et al., 2012
caudal vein plexus decreased length, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. 6 from Mendelson et al., 2013
subintestinal vein has extra parts of type angiogenic sprout, abnormal y1Tg + MO2-s1pr1 standard conditions Fig. S2 with image from Ben Shoham et al., 2012
posterior cardinal vein endothelial cell decreased amount, abnormal s896Tg; ubs1Tg + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
dorsal aorta endothelial cell decreased amount, abnormal s896Tg; ubs1Tg + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
intersegmental vessel endothelial cell decreased amount, abnormal s896Tg; ubs1Tg + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
dorsal longitudinal anastomotic vessel endothelial cell decreased amount, abnormal s896Tg; ubs1Tg + MO2-s1pr1 standard conditions Fig. 5 from Mendelson et al., 2013
intersegmental vessel shortened, abnormal y1Tg + MO1-s1pr2 + MO2-s1pr1 standard conditions Fig. 7 from Mendelson et al., 2013
intersegmental vessel morphology, abnormal y1Tg + MO1-s1pr2 + MO2-s1pr1 standard conditions Fig. 7 from Mendelson et al., 2013
intersegmental vessel shortened, abnormal y1Tg + MO2-s1pr1 + MO2-spns2 standard conditions Fig. 7 from Mendelson et al., 2013
vasculature development disrupted, abnormal y1Tg + MO2-s1pr1 + MO2-spns2 standard conditions Fig. 7 from Mendelson et al., 2013
OTHER MO2-s1pr1 MORPHOLINO PAGESNo links to external sites
CITATIONS (4)