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Fig. S5 p53 MO does not rescue the phenotypes of smarcal1 morphants. Compared with smarcal1 morphants, p53 MO and smarcal1 MO co-injected embryos show similar developmental defects in gross morphology, erythrogenesis and apoptosis. Blood cells were stained with O-dianisidine and apoptosis was assayed with TUNEL at 2 dpf embryos. The higher magnification images of TUNEL staining were shown in the right panel.
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Reprinted from Developmental Biology, 339(1), Huang, C., Gu, S., Yu, P., Yu, F., Feng, C., Gao, N., and Du, J., Deficiency of smarcal1 causes cell cycle arrest and developmental abnormalities in zebrafish, 89-100, Copyright (2010) with permission from Elsevier. Full text @ Dev. Biol.