Publication	Data		Fish	Anatomy
Schenk et al., 2019	Fig. 1		WT + MO4-sulf1 ,   zf3202Tg + MO4-sulf1	caudal artery dissociated from caudal vein, abnormal caudal fin blood circulation decreased occurrence, abnormal post-vent vasculature morphology, abnormal
	Fig. 2		WT + MO4-sulf1 ,   lri500Tg + MO4-sulf1	caudal fin curved, abnormal glomerular filtration process quality, abnormal optic choroid vascular plexus EGFP expression decreased amount, abnormal pericardium edematous, abnormal whole organism sulf2a expression increased amount, abnormal whole organism sulf2b expression increased amount, abnormal yolk edematous, abnormal
	Fig. 3		WT + MO4-sulf1	pronephric glomerular basement membrane structure, abnormal pronephric glomerulus morphology, abnormal pronephric podocyte morphology, abnormal
Al Oustah et al., 2014	Fig. 1		vu12Tg + MO5-sulf1 ,   sulf1sa199/sa199 ,   sulf1sa199/+ ,   vu17Tg; vu19Tg + MO5-sulf1	spinal cord has fewer parts of type oligodendrocyte, abnormal spinal cord oligodendrocyte cell differentiation decreased occurrence, abnormal
	Fig. 2		WT + MO5-sulf1 ,   sulf1sa199/sa199 ,   sulf1sa199/+	spinal cord has fewer parts of type Kolmer-Agduhr neuron, abnormal spinal cord has fewer parts of type spinal cord interneuron, abnormal ventral spinal cord interneuron differentiation decreased occurrence, abnormal
	Fig. 5		WT + MO5-sulf1 ,   sulf1sa199/sa199	spinal cord ventral region poorly differentiated, abnormal ventral spinal cord development process quality, abnormal
	Fig. S5		WT + MO5-sulf1 ,   sulf1sa199/sa199	neural tube smoothened signaling pathway decreased process quality, abnormal spinal cord smoothened signaling pathway decreased process quality, abnormal
Gorsi et al., 2014	Fig. 1		s844Tg; sd2Tg + MO3-sulf1	head hemorrhagic, abnormal hindbrain morphology, abnormal post-vent region edematous, abnormal
	Fig. 2		WT + MO3-sulf1	heparan sulfate 6-O-sulfotransferase activity process quality, abnormal
	Fig. 3		sd2Tg + MO3-sulf1	cranial vasculature blood circulation disrupted, abnormal post-vent vasculature blood circulation disrupted, abnormal
	Fig. 4		WT + MO4-sulf1 ,   WT + MO3-sulf1	axial vasculature blood circulation disrupted, abnormal cranial vasculature blood circulation disrupted, abnormal
	Fig. 5		s844Tg; sd2Tg + MO3-sulf1	blood vessel lumenization disrupted, abnormal caudal artery immature, abnormal caudal artery morphology, abnormal caudal vein plexus disorganized, abnormal central artery blood vessel lumenization disrupted, abnormal
	Fig. S3		s844Tg; sd2Tg + MO3-sulf1	blood circulation disrupted, abnormal central artery immature, abnormal cranial vasculature lacks parts or has fewer parts of type central artery, abnormal head hemorrhagic, abnormal hindbrain edematous, abnormal spinal cord curved, abnormal
	Fig. S5		s844Tg; sd2Tg + MO3-sulf1 ,   sd2Tg + MO3-sulf1	blood accumulation caudal vein plexus, abnormal cranial vasculature blood circulation disrupted, abnormal cranial vasculature lacks parts or has fewer parts of type central artery, abnormal dorsal aorta blood circulation disrupted, abnormal
	Fig. S6		s844Tg + MO3-sulf1	cranial vasculature broken, abnormal
Meyers et al., 2013	Fig. 2		WT + MO2-sulf1	caudal fin curved, abnormal caudal fin increased width, abnormal horizontal myoseptum absent, abnormal muscle cell sparse, abnormal somite decreased width, abnormal somite obtuse angle to somite, abnormal somite shape, abnormal somite tight, abnormal
	Fig. 3		WT + MO2-sulf1	lateral larval melanophore stripe absent, abnormal muscle pioneer decreased amount, abnormal somite decreased width, abnormal somite obtuse angle to somite, abnormal
	Fig. 4		WT + MO2-sulf1	somite shape, abnormal
	Fig. 5		WT + MO2-sulf1	somite has fewer parts of type fast muscle cell, abnormal somite has fewer parts of type muscle pioneer, abnormal
	Fig. 6		WT + MO2-sulf1	horizontal myoseptum absent, abnormal muscle cell sparse, abnormal
	Fig. 7		WT + MO2-sulf1	lateral larval melanophore stripe has fewer parts of type melanocyte, abnormal somite decreased width, abnormal somite obtuse angle to somite, abnormal
	Fig. 8		zf106Tg + MO2-sulf1	lateral line development process quality, abnormal lateral line morphology, abnormal
	Fig. 9		zf106Tg + MO2-sulf1 ,   WT + MO2-sulf1	neuromast decreased amount, abnormal neuromast decreased size, abnormal neuromast deposition decreased process quality, abnormal neuromast dispersed, abnormal neuromast primordium migration process quality, abnormal