Phenotype
|
Fish
|
Conditions
|
Figures
|
whole organism gata1a expression increased amount, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
FIGURE 4
from Mazzola et al., 2020
|
mitotic sister chromatid cohesion process quality, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
Fig. 7
from Horsfield et al., 2007
|
cell spindle morphology, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
Fig. 7
from Horsfield et al., 2007
|
embryo development delayed, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
Fig. 4
from Horsfield et al., 2007
|
whole organism deformed, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
Fig. 8
from Muto et al., 2011
|
mitotic cell cycle arrested, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
Fig. 7
from Horsfield et al., 2007
|
whole organism spi1b expression increased amount, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
FIGURE 4
from Mazzola et al., 2020
|
whole organism morphology, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
Fig. 4
from Horsfield et al., 2007
|
whole organism runx1 expression increased amount, abnormal
|
AB + MO2-rad21a
|
standard conditions
|
FIGURE 4
from Mazzola et al., 2020
|
whole organism itga9 expression decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 7
from Schuster et al., 2015
|
cell population proliferation increased occurrence, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 7
from Xu et al., 2015
|
developmental growth disrupted, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1
from Schuster et al., 2015
|
cranial cartilage cartilage development decreased process quality, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 5
from Xu et al., 2015
|
melanocyte mislocalised, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 5
from Schuster et al., 2015
|
heart edematous, abnormal
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. S7
from Xu et al., 2015
|
whole organism lethal (sensu genetics), ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. S4
from Xu et al., 2015
|
whole organism pdgfaa expression decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 7
from Schuster et al., 2015
|
cell population proliferation increased occurrence, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 7
from Xu et al., 2015
|
head decreased size, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: 4-methyl-2-oxopentanoic acid
|
Fig. 8
from Xu et al., 2015
|
whole organism decreased length, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1
from Xu et al., 2015
|
head apoptotic process increased occurrence, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 6
from Xu et al., 2015
|
eye decreased size, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 2
from Xu et al., 2015
|
whole organism bmp3 expression decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 7
from Schuster et al., 2015
|
whole organism lethal (sensu genetics), abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 8,
Fig. S4
from Xu et al., 2015
|
head apoptotic process increased occurrence, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 6
from Xu et al., 2015
|
rRNA transcription decreased process quality, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 4,
Fig. 9
from Xu et al., 2015
|
trunk decreased length, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 2
from Xu et al., 2015
|
translation decreased process quality, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 3,
Fig. 4,
Fig. 9
from Xu et al., 2015
|
heart malformed, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1,
Fig. S2
from Schuster et al., 2015
|
eye decreased size, abnormal
|
WT + MO2-rad21a
|
chemical treatment: 4-methyl-2-oxopentanoic acid
|
Fig. 8
from Xu et al., 2015
|
cranial cartilage cartilage development disrupted, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 5
from Xu et al., 2015
|
rRNA transcription decreased process quality, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 4
from Xu et al., 2015
|
post-vent region curved, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 2
from Xu et al., 2015
|
neural tube pigmentation increased occurrence, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 5
from Schuster et al., 2015
|
whole organism dead, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1
from Schuster et al., 2015
|
translation decreased process quality, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 3,
Fig. 4
from Xu et al., 2015
|
heart contraction decreased rate, abnormal
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. S7
from Xu et al., 2015
|
post-vent region curved, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1
from Xu et al., 2015
|
heart edematous, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1,
Fig. S7
from Xu et al., 2015
|
whole organism decreased size, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1
from Schuster et al., 2015
|
whole organism sema3aa expression decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 7
from Schuster et al., 2015
|
post-vent region apoptotic process increased occurrence, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 6
from Xu et al., 2015
|
embryo development disrupted, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: 4-methyl-2-oxopentanoic acid
|
Fig. 8
from Xu et al., 2015
|
whole organism gja1b expression decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 7
from Schuster et al., 2015
|
head decreased size, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1,
Fig. 8
from Xu et al., 2015
|
whole organism sox10 expression decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. S4
from Schuster et al., 2015
|
embryo development disrupted, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 8
from Xu et al., 2015
|
eye decreased size, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1,
Fig. 8
from Xu et al., 2015
|
whole organism tp53 expression increased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 1
from Xu et al., 2015
|
translation decreased process quality, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: 4-methyl-2-oxopentanoic acid
|
Fig. 9
from Xu et al., 2015
|
rRNA transcription decreased process quality, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: 4-methyl-2-oxopentanoic acid
|
Fig. 9
from Xu et al., 2015
|
heart contraction decreased rate, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. S7
from Xu et al., 2015
|
whole organism lethal (sensu genetics), ameliorated
|
WT + MO2-rad21a
|
chemical treatment: 4-methyl-2-oxopentanoic acid
|
Fig. 8
from Xu et al., 2015
|
epidermis melanocyte position, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 5
from Schuster et al., 2015
|
heart cardiac neural crest decreased amount, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 4
from Schuster et al., 2015
|
head decreased size, ameliorated
|
WT + MO2-rad21a
|
chemical treatment: L-leucine zwitterion
|
Fig. 2
from Xu et al., 2015
|
post-vent region apoptotic process increased occurrence, abnormal
|
WT + MO2-rad21a
|
standard conditions
|
Fig. 6
from Xu et al., 2015
|
heart malformed, abnormal
|
rad21anz171/+ + MO2-rad21a
|
standard conditions
|
Fig. S3
from Schuster et al., 2015
|
pharyngeal arch morphology, abnormal
|
ba2Tg + MO2-rad21a
|
standard conditions
|
Fig. 6
from Schuster et al., 2015
|
pharyngeal arch disorganized, abnormal
|
ba2Tg + MO2-rad21a
|
standard conditions
|
Fig. 6
from Schuster et al., 2015
|
pharyngeal arch aplastic, abnormal
|
ba2Tg + MO2-rad21a
|
standard conditions
|
Fig. 6
from Schuster et al., 2015
|
atrioventricular valve non-functional, abnormal
|
c262Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
atrioventricular valve decreased size, abnormal
|
c262Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
ventriculo bulbo valve decreased size, abnormal
|
c262Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
atrioventricular valve orientation ventriculo bulbo valve, abnormal
|
c262Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
ventriculo bulbo valve aplastic, abnormal
|
c262Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
cardiac ventricle blood decreased amount, abnormal
|
c262Tg; sd2Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
blood circulation decreased process quality, abnormal
|
c262Tg; sd2Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
atrium blood decreased amount, abnormal
|
c262Tg; sd2Tg + MO2-rad21a
|
standard conditions
|
Fig. 3
from Schuster et al., 2015
|
heart malformed, abnormal
|
sd2Tg; zp5Tg + MO2-rad21a
|
standard conditions
|
Fig. 1
from Schuster et al., 2015
|
heart looping disrupted, abnormal
|
sd2Tg; zp5Tg + MO2-rad21a
|
standard conditions
|
Fig. 2
from Schuster et al., 2015
|
heart morphology, abnormal
|
sd2Tg; zp5Tg + MO2-rad21a
|
standard conditions
|
Fig. 2
from Schuster et al., 2015
|
nucleate erythrocyte increased accumulation heart, abnormal
|
sd2Tg; zp5Tg + MO2-rad21a
|
standard conditions
|
Fig. 2
from Schuster et al., 2015
|
blood circulation decreased rate, abnormal
|
sd2Tg; zp5Tg + MO2-rad21a
|
standard conditions
|
Fig. 1,
Fig. 2
from Schuster et al., 2015
|
heart contraction decreased rate, abnormal
|
sd2Tg; zp5Tg + MO2-rad21a
|
standard conditions
|
Fig. 2
from Schuster et al., 2015
|
heart malformed, ameliorated
|
tp53zdf1/zdf1 + MO2-rad21a
|
standard conditions
|
Fig. S2
from Schuster et al., 2015
|