FIGURE

Fig. 5

ID

ZDB-FIG-220831-27

Publication

Paul et al., 2022 - Aluminum (Al) causes a delayed suppression of nucleotide excision repair (NER) capacity in zebrafish (Danio rerio) embryos via disturbance of DNA lesion detection

Other Figures

All Figure Page

Back to All Figure Page

Fig. 5

Fig. 5. Delayed response of NER capacity in zebrafish embryos to Al. Embryos at 1 hpf were treated with Al at 15 mg/L for the indicated time periods and UV damage repair activities in the extracts of stressed and unstressed embryos at the same developmental stages were determined. (A) A representative gel showing the results of transcription-based repair assay. (B) Time-course effects of Al on NER capacity determined by quantitative analysis. Each bar graph represents the mean and standard deviation of five independent experiments. * indicates p < 0.05 when compared to protein-free negative control. CE stands for crude extracts.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Phenotype Detail

Acknowledgments

This image is the copyrighted work of the attributed author or publisher, and ZFIN has permission only to display this image to its users. Additional permissions should be obtained from the applicable author or publisher of the image. Full text @ Ecotoxicol. Environ. Saf.