Fig. 1
- ID
- ZDB-FIG-220831-23
- Publication
- Paul et al., 2022 - Aluminum (Al) causes a delayed suppression of nucleotide excision repair (NER) capacity in zebrafish (Danio rerio) embryos via disturbance of DNA lesion detection
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Fig. 1. Effects of increasing Al concentration on the survival of developing zebrafish. Zebrafish embryos at 1 hpf were exposed to Al at the indicated concentrations until the larval stage at 96 hpf. Exposure was carried out by individually transferring embryos to the wells of 24-well plates with 2 mL of test solution per well and endpoints described in OECD guideline TG 236 were used as the indicators for lethal responses. Each bar graph represents the percentage survival rate of embryos or larvae as the mean and standard deviation of four separate experiments. * indicates p < 0.05 and ** indicates p < 0.01 when compared to the survival rate of nonexposed embryos at the same developmental stage. |