FIGURE

Fig. 1

ID

ZDB-FIG-200604-20

Publication

Budine et al., 2020 - Gon4l/Udu Regulates Cardiomyocyte Proliferation and Maintenance of Ventricular Chamber Identity During Zebrafish Development

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Fig. 1

Loss of zygotic Gon4l function affects cardiac chamber patterning and causes a reduction in number of cardiomyocytes. Lateral view of WT embryos (A) and udu?/? (B) and their heart region (C, D) at 48 hpf. Dorsal view of WT (E, F, G) and udu?/- embryos (H, I,J) showing the expression of myl7 (E, H), myh6 (F, I), and myh7 (G, J) at 24 hpf by WISH. Dorsal view of myl7 (K, N), myh6 (L, O), and myh7 (M, P) expression at 48 hpf by WISH in WT (K, L, M) udu?/? (N, O, P) embryos.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Phenotype Detail

Acknowledgments

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Reprinted from Developmental Biology, 462(2), Budine, T.E., de Sena-Tomás, C., Williams, M.L.K., Sepich, D.S., Targoff, K.L., Solnica-Kreze, L., Gon4l/Udu Regulates Cardiomyocyte Proliferation and Maintenance of Ventricular Chamber Identity During Zebrafish Development, 223-234, Copyright (2020) with permission from Elsevier. Full text @ Dev. Biol.