FIGURE
            Fig. S2
- ID
- ZDB-FIG-180725-24
- Publication
- Berrun et al., 2018 - Isthmin 1 (ism1) is required for normal hematopoiesis in developing zebrafish
- Other Figures
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                        Fig. S2
                    
                    
                
                
            
        
        
    
        
            
            
| 
 ism1 knockdown leads to temporally shortened intersegmental vessels. flk1:GFP single-cell-stage embryos were injected with 7 ng of ism1 MO (bottom); uninjected embryos served as controls (top). 24 hpf (left column) and 48 hpf (right column) zebrafish were visualized at 40x for flk1:GFP fluorescence within the trunk area denoted by black box in brightfield image at top center. Arrows indicate shortened intersegmental vessels in ism1 morphants. Numbers in corner of images denote the number of embryos displaying the imaged phenotype. | 
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
  
    | Fish: | |
|---|---|
| Knockdown Reagent: | |
| Observed In: | |
| Stage Range: | Prim-5 to Long-pec | 
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ PLoS One
                
                
            
        
        
    
    
     
        