FIGURE
            Fig. 10
- ID
 - ZDB-FIG-151008-2
 - Publication
 - Aspatwar et al., 2015 - Inactivation of ca10a and ca10b Genes Leads to Abnormal Embryonic Development and Alters Movement Pattern in Zebrafish
 - Other Figures
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                        Fig. 10
                    
                    
                
                
            
        
        
    
        
            
            
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 Knockdown of ca10a and ca10b genes leads to apoptosis in the morphant zebrafish. Results of the TUNEL assay detecting apoptotic cells in 5 dpf morphant embryos. A) head region of a ca10a morphant; B) eye region of a ca10a morphant; C) head region of a ca10b morphant; and D) tail regions of a ca10b morphant. (Original magnification 100X).  | 
    
                
                    
                        Expression Data
                    
                    
                
                
            
        
        
    
        
            
            
            
            
    
    
                
                    
                        Expression Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Antibody Labeling
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Phenotype Data
                    
                    
                
                
            
        
        
    
        
            
            
  
    | Fish: | |
|---|---|
| Knockdown Reagents: | |
| Observed In: | |
| Stage: | Day 5 | 
                
                    
                        Phenotype Detail
                    
                    
                
                
            
        
        
    
        
            
                
            
        
    
    
    
                
                    
                        Acknowledgments
                    
                    
                
                
            
        
        
    
        
            
            
                
                    
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      Full text @ PLoS One