Fig. S6
- ID
- ZDB-FIG-150422-70
- Publication
- Olena et al., 2015 - miR-216a regulates snx5, a novel notch signaling pathway component, during zebrafish retinal development
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Co-injection with p53 morpholino causes no changes in cell death. WT embryos were injected with miR-216aMOs (A), miR-216aMOs and p53MO (B), snx5MOs (C), or snx5MOs and p53MO (D) at the 1-cell stage, fixed at 65 hpf, and transverse sections of developing retinas were obtained. TUNEL labeling was performed and TUNEL+ cells counted. No difference in number of TUNEL+ cells was found between miR-216aMOs and miR-216aMOs+p53MO co-injection or between, snx5MOs and snx5MOs+p53MO co-injection (E). Nuclei are marked by staining with To-Pro. Error bars = SD. |
Reprinted from Developmental Biology, 400(1), Olena, A.F., Rao, M.B., Thatcher, E.J., Wu, S.Y., Patton, J.G., miR-216a regulates snx5, a novel notch signaling pathway component, during zebrafish retinal development, 72-81, Copyright (2015) with permission from Elsevier. Full text @ Dev. Biol.