FIGURE

Fig. 5

ID

ZDB-FIG-100114-29

Publication

Codina et al., 2010 - Loss of Smyhc1 or Hsp90alpha1 function results in different effects on myofibril organization in skeletal muscles of zebrafish embryos

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Fig. 5

Knockdown of smyhc1 expression or hsp90α1 mutation resulted in defective thin filament organization in skeletal muscles of zebrafish embryos.

A?C. Anti-α-actin antibody staining shows the organization of thin filaments in slow muscles of control (A), smyhc1 knockdown (B), or slo^tu44c mutant (C) embryos at 48 hpf. D, F. Anti-α-actin antibody staining shows the organization of thin filaments in fast muscles of control (D), smyhc1 knockdown (E), or slo^tu44c mutant (F) embryos at 72 hpf. Scale bar = 25 μm in A.

Expression Data

Antibody:	Ab1-actc
Fish:	WT hsp90aa1.1^tu44c/tu44c WT + MO1-smyhc1
Knockdown Reagent:	MO1-smyhc1
Anatomical Terms:	fast muscle cell striated muscle thin filament slow muscle cell striated muscle thin filament
Stage Range:	Long-pec to Protruding-mouth

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	hsp90aa1.1^tu44c/tu44c WT + MO1-smyhc1
Knockdown Reagent:	MO1-smyhc1
Observed In:	fast muscle cell striated muscle thin filament slow muscle cell striated muscle thin filament
Stage Range:	Long-pec to Protruding-mouth

Phenotype Detail

Acknowledgments

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