PUBLICATION

Vibrio cholerae arrests intestinal epithelial proliferation through T6SS-dependent activation of the bone morphogenetic protein pathway

Authors
Xu, X., Foley, E.
ID
ZDB-PUB-240211-11
Date
2024
Source
Cell Reports   43: 113750113750 (Journal)
Registered Authors
Foley, Edan
Keywords
BMP, CP: Immunology, Drosophila, IMD, Vibrio cholerae, host-microbe interactions, intestinal progenitors, type VI secretion system, zebrafish
MeSH Terms
  • Zebrafish*
  • Animals
  • Bone Morphogenetic Proteins
  • Vibrio cholerae*
  • Anti-Bacterial Agents
  • Cell Proliferation
  • Drosophila
PubMed
38340318 Full text @ Cell Rep.
Abstract
To maintain an effective barrier, intestinal progenitor cells must divide at a rate that matches the loss of dead and dying cells. Otherwise, epithelial breaches expose the host to systemic infection by gut-resident microbes. Unlike most pathogens, Vibrio cholerae blocks tissue repair by arresting progenitor proliferation in the Drosophila model. At present, we do not understand how V. cholerae circumvents such a critical antibacterial defense. We find that V. cholerae blocks epithelial repair by activating the growth inhibitor bone morphogenetic protein (BMP) pathway in progenitors. Specifically, we show that interactions between V. cholerae and gut commensals initiate BMP signaling via host innate immune defenses. Notably, we find that V. cholerae also activates BMP and arrests proliferation in zebrafish intestines, indicating an evolutionarily conserved link between infection and failure in tissue repair. Our study highlights how enteric pathogens engage host immune and growth regulatory pathways to disrupt intestinal epithelial repair.
Genes / Markers
Figures
Show all Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping