PUBLICATION

Prion protein-related proteins from zebrafish are complex glycosylated and contain a glycosylphosphatidylinositol anchor

Authors
Miesbauer, M., Bamme, T., Riemer, C., Oidtmann, B., Winklhofer, K.F., Baier, M., and Tatzelt, J.
ID
ZDB-PUB-060124-19
Date
2006
Source
Biochemical and Biophysical Research Communications   341(1): 218-224 (Journal)
Registered Authors
Keywords
none
MeSH Terms
  • Amino Acid Sequence
  • Animals
  • Endoplasmic Reticulum/metabolism*
  • Glycosylation
  • Glycosylphosphatidylinositols/chemistry*
  • Glycosylphosphatidylinositols/metabolism*
  • Mice
  • Molecular Sequence Data
  • Prions/chemistry*
  • Prions/metabolism*
  • Zebrafish/metabolism*
PubMed
16414019 Full text @ Biochem. Biophys. Res. Commun.
Abstract
A hallmark of prion diseases in mammals is a conformational transition of the cellular prion protein (PrP(C)) into a pathogenic isoform termed PrP(Sc). PrP(C) is highly conserved in mammals, moreover, genes of PrP-related proteins have been recently identified in fish. While there is only little sequence homology to mammalian PrP, PrP-related fish proteins were predicted to be modified with N-linked glycans and a C-terminal glycosylphosphatidylinositol (GPI) anchor. We biochemically characterized two PrP-related proteins from zebrafish in cultured cells and show that both zePrP1 and zeSho2 are imported into the endoplasmic reticulum and are post-translationally modified with complex glycans and a C-terminal GPI anchor.
Genes / Markers
Figures
Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping