Phenotype
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Fish
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Conditions
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Figures
|
dorsal aorta blood vessel endothelial cell notch1b expression decreased amount, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel Notch signaling pathway decreased occurrence, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel has fewer parts of type blood vessel endothelial cell, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 4
from Savage et al., 2019
|
dorsal aorta Notch signaling pathway decreased occurrence, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel blood vessel endothelial cell notch1b expression decreased amount, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
dorsal aorta blood vessel endothelial cell hey2 expression decreased amount, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel blood vessel endothelial cell dll4 expression decreased amount, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
dorsal aorta blood vessel endothelial cell her12 expression decreased amount, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel blood vessel endothelial cell her12 expression decreased amount, abnormal
|
WT + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel blood vessel endothelial cell Venus expression decreased amount, abnormal
|
qmc61Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
dorsal aorta blood vessel endothelial cell Venus expression decreased amount, abnormal
|
qmc61Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel Notch signaling pathway decreased occurrence, abnormal
|
qmc61Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
spinal cord Venus expression decreased amount, abnormal
|
qmc61Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
dorsal aorta Notch signaling pathway decreased occurrence, abnormal
|
qmc61Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 6
from Savage et al., 2019
|
intersegmental vessel cell migration involved in sprouting angiogenesis decreased occurrence, abnormal
|
sh467Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|
intersegmental vessel endothelial tip cell filopodium assembly decreased occurrence, abnormal
|
sh467Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|
intersegmental vessel endothelial tip cell has fewer parts of type endothelial tip cell filopodium, abnormal
|
sh467Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|
intersegmental vessel endothelial tip cell spade-shaped, abnormal
|
y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
intersegmental vessel cell migration involved in sprouting angiogenesis decreased occurrence, abnormal
|
y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
dorsal longitudinal anastomotic vessel split, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
dorsal longitudinal anastomotic vessel sprouting angiogenesis decreased occurrence, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
intersegmental vessel cell migration involved in sprouting angiogenesis decreased occurrence, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
cardiovascular system lacks all parts of type vascular lymphangioblast, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
intersegmental vessel sprouting angiogenesis decreased occurrence, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
lymph vessel development decreased occurrence, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
cardiovascular system lacks all parts of type thoracic duct, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
dorsal longitudinal anastomotic vessel malformed, abnormal
|
hu5333Tg; y1Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 1
from Savage et al., 2019
|
endothelial tip cell regulation of cytosolic calcium ion concentration decreased occurrence, abnormal
|
ubs3Tg ; sh392Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|
endothelial tip cell calcium-mediated signaling decreased occurrence, abnormal
|
ubs3Tg ; sh392Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|
intersegmental vessel endothelial tip cell GCaMP expression decreased amount, abnormal
|
ubs3Tg ; sh392Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|
endothelial tip cell regulation of store-operated calcium channel activity decreased occurrence, abnormal
|
ubs3Tg ; sh392Tg + MO1-tmem33 + MO2-tmem33
|
standard conditions
|
Fig. 2
from Savage et al., 2019
|