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Fig. S2
Tp53 mutant fish do not display phenotypes of morpholino toxicity. Wild-type (A, B, C) and tp53 mutant (D, E, F) embryos were injected with control or wnt1 MOs. In situ hybridization for rfng mRNA revealed extensive ectopic rfng expression in the wnt1 MO-injected wild-type embryos, while no ectopic rfng was observed in the wnt1 MO-injected tp53 mutant fish (compare C and F).
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Reprinted from Developmental Biology, 350(2), Gerety, S.S., and Wilkinson, D.G., Morpholino artifacts provide pitfalls and reveal a novel role for pro-apoptotic genes in hindbrain boundary development, 279-289, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.