Publication	Data		Fish	Anatomy
Anderson et al., 2009	Fig. 2		AB + MO6-nkx2.2a	endocrine pancreas development disrupted, abnormal endocrine pancreas physical object quality, abnormal
Kucenas et al., 2008	Fig. 5		vu17Tg; vu19Tg + MO4-nkx2.2a	spinal cord has extra parts of type oligodendrocyte, abnormal spinal cord has fewer parts of type oligodendrocyte, abnormal
	Fig. 6		AB + MO4-nkx2.2a	oligodendrocyte differentiation decreased frequency, abnormal
	Fig. 7		vu12Tg + MO4-nkx2.2a	spinal cord oligodendrocyte cell differentiation delayed, abnormal
	Fig. 8		vu234Tg + MO4-nkx2.2a	spinal cord oligodendrocyte cell differentiation delayed, abnormal spinal cord oligodendrocyte cell fate specification process quality, abnormal
Kucenas et al., 2008	Fig. 6		vu17Tg; vu19Tg + MO4-nkx2.2a (AB)	glial cell migration disrupted, abnormal glial cell mislocalised, abnormal motor neuron axon decreased thickness, abnormal motor neuron axon defasciculated, abnormal motor neuron axon guidance disrupted, abnormal motor neuron mislocalised, abnormal
	Fig. 7		vu17Tg; vu234Tg + MO4-nkx2.2a (AB)	myelinating Schwann cell mislocalised, abnormal Schwann cell development disrupted, abnormal
	Fig. S2		vu17Tg; vu19Tg + MO4-nkx2.2a (AB)	glial cell bicellular tight junction absent, abnormal glial cell mislocalised, abnormal myelin assembly disrupted, abnormal
Pauls et al., 2007	Fig. 6		WT + MO3-nkx2.2a	endocrine pancreas physical object quality, abnormal
	Fig. 7		WT + MO3-nkx2.2a ,   WT + MO2-nkx2.2a	pancreatic duct aplastic, abnormal
	Fig. S3		WT + MO3-nkx2.2a ,   WT + MO2-nkx2.2a ,   WT + MO2-nkx2.2a + MO3-nkx2.2a	fourth ventricle increased size, abnormal
	text only		WT + MO2-nkx2.2a + MO3-nkx2.2a ,   WT + MO2-nkx2.2a ,   WT + MO3-nkx2.2a	heart edematous, abnormal mandibular arch skeleton decreased size, abnormal whole organism decreased length, abnormal
Schafer et al., 2007	Fig. 7		WT + MO1-nkx2.2a	floor plate formation disrupted, abnormal lateral floor plate composition, abnormal