PUBLICATION

Biphasic inflammation control by fibroblasts enables spinal cord regeneration in zebrafish

Authors
John, N., Fleming, T., Kolb, J., Lyraki, O., Vásquez-Sepúlveda, S., Parmar, A., Kim, K., Tarczewska, M., Gupta, P., Singh, K., Marini, F., Singh, S.P., Tsata, V., Falk, S., Franze, K., Guck, J., Wehner, D.
ID
ZDB-PUB-251025-5
Date
2025
Source
Cell Reports   44: 116469116469 (Journal)
Registered Authors
Kolb, Julia, Singh, Sumeet Pal, Wehner, Daniel
Keywords
AFM-based nanoindentation, Brillouin microscopy, CP: Immunology, CP: Neuroscience, axon regeneration, fibroblasts, inflammation, neutrophils, spinal cord injury, tissue mechanics, zebrafish
Datasets
GEO:GSE281751, GEO:GSE281255
MeSH Terms
  • Animals
  • Fibroblasts*/metabolism
  • Fibroblasts*/pathology
  • Inflammation*/metabolism
  • Inflammation*/pathology
  • Neutrophils/metabolism
  • Spinal Cord*/physiology
  • Spinal Cord Injuries/pathology
  • Spinal Cord Regeneration*/physiology
  • Zebrafish*/physiology
  • Zebrafish Proteins/metabolism
PubMed
41134665 Full text @ Cell Rep.
Abstract
Fibrosis and persistent inflammation are interconnected processes that inhibit axon regeneration in the mammalian central nervous system (CNS). Here, we show that in zebrafish, the interplay between fibroblasts and immune cells is required for regeneration. Using temporally resolved single-cell transcriptomics, we identify a transient, injury-induced cthrc1a+ fibroblast state with an inflammation-associated, less differentiated, non-fibrotic profile. Ca2+-dependent induction of this fibroblast state precedes and is critical for the initiation of the inflammatory response. Subsequently, cthrc1a+ fibroblasts coordinate the resolution of the neutrophil-driven inflammation. Disruption of these inflammation dynamics alters the mechano-structural properties of the lesion microenvironment and inhibits regeneration and recovery of locomotor function. Thus, zebrafish fibroblasts possess a dual role in inducing and resolving inflammation, which are both required for regeneration. This establishes the biphasic inflammation control by fibroblasts as an important mechanism directing CNS regeneration.
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